The E3 ubiquitin ligase MIB1 suppresses breast cancer cell migration through regulating CTNND1 protein level.

[1]  Min Fu,et al.  Circular RNA EIF4G3 suppresses gastric cancer progression through inhibition of β-catenin by promoting δ-catenin ubiquitin degradation and upregulating SIK1 , 2022, Molecular cancer.

[2]  D. Pei,et al.  The E3 Ligase MIB1 Promotes Proteasomal Degradation of NRF2 and Sensitizes Lung Cancer Cells to Ferroptosis , 2021, Molecular Cancer Research.

[3]  A. Jemal,et al.  Global Cancer Statistics 2020: GLOBOCAN Estimates of Incidence and Mortality Worldwide for 36 Cancers in 185 Countries , 2021, CA: a cancer journal for clinicians.

[4]  Xiang Cheng,et al.  MIB1 upregulates IQGAP1 and promotes pancreatic cancer progression by inducing ST7 degradation , 2020, Molecular oncology.

[5]  G. Silvani,et al.  Biomechanical Characterization of Endothelial Cells Exposed to Shear Stress Using Acoustic Force Spectroscopy , 2020, bioRxiv.

[6]  G. Nehmetallah,et al.  Keratin 19 maintains E-cadherin localization at the cell surface and stabilizes cell-cell adhesion of MCF7 cells , 2020, bioRxiv.

[7]  B. Raught,et al.  Proximity interactions of the ubiquitin ligase Mind bomb 1 reveal a role in regulation of epithelial polarity complex proteins , 2019, Scientific Reports.

[8]  Farid Zayeri,et al.  Global Trend of Breast Cancer Mortality Rate: A 25-Year Study , 2019, Asian Pacific journal of cancer prevention : APJCP.

[9]  Cheng-Ta Yang,et al.  Downregulation of lumican accelerates lung cancer cell invasion through p120 catenin , 2018, Cell Death & Disease.

[10]  Soumen Kumar Das,et al.  Bioimpedimetric analysis in conjunction with growth dynamics to differentiate aggressiveness of cancer cells , 2018, Scientific Reports.

[11]  M. Itoh,et al.  Mib1 contributes to persistent directional cell migration by regulating the Ctnnd1-Rac1 pathway , 2017, Proceedings of the National Academy of Sciences.

[12]  Xiaofeng Dai,et al.  Breast Cancer Cell Line Classification and Its Relevance with Breast Tumor Subtyping , 2017, Journal of Cancer.

[13]  Chad J. Creighton,et al.  UALCAN: A Portal for Facilitating Tumor Subgroup Gene Expression and Survival Analyses , 2017, Neoplasia.

[14]  K. Hozumi,et al.  Identification of Domains for Efficient Notch Signaling Activity in Immobilized Notch Ligand Proteins , 2017, Journal of cellular biochemistry.

[15]  A. Jemal,et al.  Cancer statistics, 2017 , 2017, CA: a cancer journal for clinicians.

[16]  W. Birchmeier,et al.  RNF4-Dependent Oncogene Activation by Protein Stabilization , 2016, Cell reports.

[17]  M. Okano,et al.  Mib1 modulates dynamin 2 recruitment via Snx18 to promote Dll1 endocytosis for efficient Notch signaling , 2016, Genes to cells : devoted to molecular & cellular mechanisms.

[18]  Huamin Wang,et al.  Transforming Growth Factor-β Limits Secretion of Lumican by Activated Stellate Cells within Primary Pancreatic Adenocarcinoma Tumors , 2016, Clinical Cancer Research.

[19]  Andrew J. Ewald,et al.  A collective route to metastasis: Seeding by tumor cell clusters , 2016, Science.

[20]  E. Nigg,et al.  The E3 ubiquitin ligase Mib1 regulates Plk4 and centriole biogenesis , 2015, Journal of Cell Science.

[21]  Ji-Hoon Cho,et al.  Sequential Elution Interactome Analysis of the Mind Bomb 1 Ubiquitin Ligase Reveals a Novel Role in Dendritic Spine Outgrowth* , 2015, Molecular & Cellular Proteomics.

[22]  Chirayu Pankaj Goswami,et al.  PROGgeneV2: enhancements on the existing database , 2014, BMC Cancer.

[23]  M. Gack,et al.  The Ubiquitin-Specific Protease USP15 Promotes RIG-I–Mediated Antiviral Signaling by Deubiquitylating TRIM25 , 2014, Science Signaling.

[24]  F. van Roy,et al.  Functions of p120ctn isoforms in cell-cell adhesion and intracellular signaling. , 2012, Frontiers in bioscience.

[25]  R. Moon,et al.  Mindbomb 1, an E3 ubiquitin ligase, forms a complex with RYK to activate Wnt/β-catenin signaling , 2011, The Journal of cell biology.

[26]  Kyucheol Cho,et al.  Shared molecular mechanisms regulate multiple catenin proteins: canonical Wnt signals and components modulate p120-catenin isoform-1 and additional p120 subfamily members , 2010, Journal of Cell Science.

[27]  Alan Wells,et al.  Breast carcinoma cells re-express E-cadherin during mesenchymal to epithelial reverting transition , 2010, Molecular Cancer.

[28]  Zhiwei Wang,et al.  The role of Notch signaling pathway in epithelial-mesenchymal transition (EMT) during development and tumor aggressiveness. , 2010, Current drug targets.

[29]  M. Ikura,et al.  Dynamic and Static Interactions between p120 Catenin and E-Cadherin Regulate the Stability of Cell-Cell Adhesion , 2010, Cell.

[30]  Leonardo Morsut,et al.  FAM/USP9x, a Deubiquitinating Enzyme Essential for TGFβ Signaling, Controls Smad4 Monoubiquitination , 2009, Cell.

[31]  C. D’Souza-Schorey,et al.  Lysosomal Targeting of E-Cadherin: a Unique Mechanism for the Down-Regulation of Cell-Cell Adhesion during Epithelial to Mesenchymal Transitions , 2005, Molecular and Cellular Biology.

[32]  J. Martín-Pérez,et al.  Cytoplasmic localization of p120ctn and E-cadherin loss characterize lobular breast carcinoma from preinvasive to metastatic lesions , 2004, Oncogene.

[33]  Cheol‐Hee Kim,et al.  Mind bomb is a ubiquitin ligase that is essential for efficient activation of Notch signaling by Delta. , 2003, Developmental cell.

[34]  Albert B. Reynolds,et al.  Selective Uncoupling of P120ctn from E-Cadherin Disrupts Strong Adhesion , 2000, The Journal of cell biology.

[35]  B. Hogan,et al.  A mouse homologue of FAST-1 transduces TGFβ superfamily signals and is expressed during early embryogenesis , 1998, Mechanisms of Development.