Effects of Exenatide Alone and in Combination With Daclizumab on β-Cell Function in Long-Standing Type 1 Diabetes
暂无分享,去创建一个
D. Harlan | L. Spain | K. Rother | A. Baron | H. Dosch | Patric Nelson | J. Palmer | R. Wesley | B. Brooks-Worrell | M. Ring | B. Digon | Kim Chen | B. J. Digon
[1] K. Herold,et al. Type 1 diabetes as a relapsing–remitting disease? , 2007, Nature Reviews Immunology.
[2] D. Harlan,et al. Pancreatic beta cell function persists in many patients with chronic type 1 diabetes, but is not dramatically improved by prolonged immunosuppression and euglycaemia from a beta cell allograft , 2009, Diabetologia.
[3] M. Atkinson,et al. Exendin‐4 Therapy in NOD Mice with New‐Onset Diabetes Increases Regulatory T Cell Frequency , 2008, Annals of the New York Academy of Sciences.
[4] A. Michael,et al. Twin-to-twin pancreas transplantation: reversal and reenactment of the pathogenesis of type I diabetes. , 1984, Transactions of the Association of American Physicians.
[5] T. Waldmann,et al. Humanized anti-CD25 (daclizumab) inhibits disease activity in multiple sclerosis patients failing to respond to interferon beta. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[6] S. Bonner-Weir,et al. Exendin-4 stimulates both beta-cell replication and neogenesis, resulting in increased beta-cell mass and improved glucose tolerance in diabetic rats. , 1999, Diabetes.
[7] Douglas A. Melton,et al. In vivo reprogramming of adult pancreatic exocrine cells to β-cells , 2008, Nature.
[8] Seung K. Kim,et al. Glucose Infusion in Mice , 2007, Diabetes.
[9] J. Bluestone,et al. Exendin-4 improves reversal of diabetes in NOD mice treated with anti-CD3 monoclonal antibody by enhancing recovery of beta-cells. , 2007, Endocrinology.
[10] D. Stoffers,et al. Exendin-4 does not promote Beta-cell proliferation or survival during the early post-islet transplant period in mice. , 2008, Transplantation proceedings.
[11] T. McDonald,et al. Exendin-4 normalized postcibal glycemic excursions in type 1 diabetes. , 2004, The Journal of clinical endocrinology and metabolism.
[12] Lidia Ruiz,et al. Alternation of Antiretroviral Drug Regimens for HIV Infection , 2003, Annals of Internal Medicine.
[13] D. Hafler,et al. Expanded T cells from pancreatic lymph nodes of type 1 diabetic subjects recognize an insulin epitope , 2005, Nature.
[14] R. Nussenblatt,et al. Initial evaluation of subcutaneous daclizumab treatments for noninfectious uveitis: a multicenter noncomparative interventional case series. , 2005, Ophthalmology.
[15] Stephen Senn,et al. Cross‐over trials in Statistics in Medicine: the first ‘25’ years , 2006, Statistics in medicine.
[16] Yuval Dor,et al. Adult pancreatic beta-cells are formed by self-duplication rather than stem-cell differentiation. , 2004, Nature.
[17] R. Rizza,et al. β-Cell Replication Is the Primary Mechanism Subserving the Postnatal Expansion of β-Cell Mass in Humans , 2008, Diabetes.
[18] D. Harlan,et al. Assessment of islet function following islet and pancreas transplantation , 2006, Current diabetes reports.
[19] D. Harlan,et al. A Single Course of Anti-CD3 Monoclonal Antibody hOKT3γ1(Ala-Ala) Results in Improvement in C-Peptide Responses and Clinical Parameters for at Least 2 Years after Onset of Type 1 Diabetes , 2005, Diabetes.
[20] Y. Yamasaki,et al. Beta-cell regeneration in a patient with type 1 diabetes mellitus who was receiving immunosuppressive therapy. , 2003, Annals of internal medicine.
[21] R. Rizza,et al. Sustained beta cell apoptosis in patients with long-standing type 1 diabetes: indirect evidence for islet regeneration? , 2005, Diabetologia.
[22] J. Bluestone,et al. Analysis of T-Cell Assays to Measure Autoimmune Responses in Subjects With Type 1 Diabetes , 2006, Diabetes.
[23] Johnny Ludvigsson,et al. GAD treatment and insulin secretion in recent-onset type 1 diabetes. , 2008, The New England journal of medicine.
[24] J. Todd,et al. HLA DR-DQ Haplotypes and Genotypes and Type 1 Diabetes Risk , 2008, Diabetes.
[25] J. Kushner,et al. Growth and regeneration of adult beta cells does not involve specialized progenitors. , 2007, Developmental cell.
[26] J. Gaglia,et al. Benefits and risks of solitary islet transplantation for type 1 diabetes using steroid-sparing immunosuppression: the National Institutes of Health experience. , 2003, Diabetes care.