NogoA Neutralization Promotes Axonal Restoration After White Matter Injury In Subcortical Stroke

[1]  M. Gutiérrez-Fernández,et al.  White Matter Repair After Extracellular Vesicles Administration in an Experimental Animal Model of Subcortical Stroke , 2017, Scientific Reports.

[2]  S. Carmichael,et al.  Molecular, cellular and functional events in axonal sprouting after stroke , 2017, Experimental Neurology.

[3]  G. Kartje,et al.  Anti-Nogo-A Immunotherapy Does Not Alter Hippocampal Neurogenesis after Stroke in Adult Rats , 2016, Front. Neurosci..

[4]  M. Gutiérrez-Fernández,et al.  Enhanced brain-derived neurotrophic factor delivery by ultrasound and microbubbles promotes white matter repair after stroke. , 2016, Biomaterials.

[5]  J. Kong,et al.  Astrocytes in Oligodendrocyte Lineage Development and White Matter Pathology , 2016, Front. Cell. Neurosci..

[6]  S. Johansson,et al.  How to Properly Measure a Current-Voltage Relation?—Interpolation vs. Ramp Methods Applied to Studies of GABAA Receptors , 2016, Front. Cell. Neurosci..

[7]  I. K. Yazdi,et al.  Antibody-mediated inhibition of Nogo-A signaling promotes neurite growth in PC-12 cells , 2016, Journal of tissue engineering.

[8]  H. Widmer,et al.  Non-canonical actions of Nogo-A and its receptors. , 2016, Biochemical pharmacology.

[9]  B. Ransom,et al.  Ischemic Preconditioning in White Matter: Magnitude and Mechanism , 2015, The Journal of Neuroscience.

[10]  T. Sobrino,et al.  White matter injury restoration after stem cell administration in subcortical ischemic stroke , 2015, Stem cell research & therapeutics.

[11]  Sebastián Cerdán,et al.  Intralesional Patterns of MRI ADC Maps Predict Outcome in Experimental Stroke , 2015, Cerebrovascular Diseases.

[12]  S. Cerdán,et al.  Brain-Derived Neurotrophic Factor Administration Mediated Oligodendrocyte Differentiation and Myelin Formation in Subcortical Ischemic Stroke , 2015, Stroke.

[13]  M. Schwab,et al.  Finding an optimal rehabilitation paradigm after stroke: enhancing fiber growth and training of the brain at the right moment , 2014, Front. Hum. Neurosci..

[14]  Ajmal Zemmar,et al.  Neutralization of Nogo-A Enhances Synaptic Plasticity in the Rodent Motor Cortex and Improves Motor Learning in Vivo , 2014, The Journal of Neuroscience.

[15]  Kelly A. Tennant Thinking outside the brain: Structural plasticity in the spinal cord promotes recovery from cortical stroke , 2014, Experimental Neurology.

[16]  Martin E Schwab,et al.  Rewiring of the corticospinal tract in the adult rat after unilateral stroke and anti-Nogo-A therapy. , 2014, Brain : a journal of neurology.

[17]  Carlos Matute,et al.  Protecting White Matter From Stroke Injury , 2013, Stroke.

[18]  John H. Zhang,et al.  RIGOR Guidelines: Escalating STAIR and STEPS for Effective Translational Research , 2012, Translational Stroke Research.

[19]  M. Schwab,et al.  The role of Nogo-A in axonal plasticity, regrowth and repair , 2012, Cell and Tissue Research.

[20]  M. Schwab,et al.  Delayed Anti-Nogo-A Therapy Improves Function After Chronic Stroke in Adult Rats , 2011, Stroke.

[21]  D. Wallace,et al.  Cognitive recovery in the aged rat after stroke and anti-Nogo-A immunotherapy , 2010, Behavioural Brain Research.

[22]  Leif A. Havton,et al.  A white matter stroke model in the mouse: Axonal damage, progenitor responses and MRI correlates , 2009, Journal of Neuroscience Methods.

[23]  Marc Fisher,et al.  Update of the Stroke Therapy Academic Industry Roundtable Preclinical Recommendations , 2009, Stroke.

[24]  M. Fehlings,et al.  A quantitative ELISA for bioactive anti-Nogo-A, a promising regenerative molecule for spinal cord injury repair. , 2009, Methods.

[25]  S. Strittmatter,et al.  Axon regeneration in the peripheral and central nervous systems. , 2009, Results and problems in cell differentiation.

[26]  H. Thompson,et al.  Cognitive outcome following brain injury and treatment with an inhibitor of Nogo-A in association with an attenuated downregulation of hippocampal growth-associated protein-43 expression. , 2007, Journal of neurosurgery.

[27]  M. Schwab,et al.  Intrathecal treatment with anti-Nogo-A antibody improves functional recovery in adult rats after stroke , 2007, Experimental Brain Research.

[28]  S. Lacroix,et al.  Expression profile of receptors for myelin-associated inhibitors of axonal regeneration in the intact and injured mouse central nervous system , 2007, Molecular and Cellular Neuroscience.

[29]  B. Kolb,et al.  Cerebral Cortex doi:10.1093/cercor/bhi132 Dendritic Plasticity in the Adult Rat Following Middle Cerebral Artery , 2005 .

[30]  M. Rausch,et al.  Recovery and brain reorganization after stroke in adult and aged rats , 2005, Annals of neurology.

[31]  M. Schwab,et al.  Delayed Treatment with Monoclonal Antibody IN-1 1 Week after Stroke Results in Recovery of Function and Corticorubral Plasticity in Adult Rats , 2005, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[32]  M. Schwab Nogo and axon regeneration , 2004, Current Opinion in Neurobiology.

[33]  M. Schwab,et al.  Rapid induction of autoantibodies against Nogo‐A and MOG in the absence of an encephalitogenic T cell response: implication for immunotherapeutic approaches in neurological diseases , 2003, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[34]  A. B. Huber,et al.  Nogo-A Inhibits Neurite Outgrowth and Cell Spreading with Three Discrete Regions , 2003, The Journal of Neuroscience.

[35]  M. Schwab,et al.  Anti—Nogo-A Antibody Infusion 24 Hours after Experimental Stroke Improved Behavioral Outcome and Corticospinal Plasticity in Normotensive and Spontaneously Hypertensive Rats , 2003, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[36]  S. Strittmatter,et al.  Localization of Nogo-A and Nogo-66 Receptor Proteins at Sites of Axon–Myelin and Synaptic Contact , 2002, The Journal of Neuroscience.

[37]  M. Schwab,et al.  Functional recovery and neuroanatomical plasticity following middle cerebral artery occlusion and IN‐1 antibody treatment in the adult rat , 2002, Annals of neurology.

[38]  S. Carmichael,et al.  New Patterns of Intracortical Projections after Focal Cortical Stroke , 2001, Neurobiology of Disease.

[39]  M. Cipolla,et al.  Mechanical signaling through connective tissue: a mechanism for the therapeutic effect of acupuncture , 2001, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[40]  M. Chopp,et al.  Adult Bone Marrow Transplantation after Stroke in Adult Rats , 2001, Cell transplantation.

[41]  K. Fouad,et al.  Compensatory Sprouting and Impulse Rerouting after Unilateral Pyramidal Tract Lesion in Neonatal Rats , 2000, The Journal of Neuroscience.

[42]  Martin E. Schwab,et al.  Nogo-A is a myelin-associated neurite outgrowth inhibitor and an antigen for monoclonal antibody IN-1 , 2000, Nature.

[43]  R. Prinjha,et al.  Neurobiology: Inhibitor of neurite outgrowth in humans , 2000, Nature.

[44]  Fumio Nakamura,et al.  Identification of the Nogo inhibitor of axon regeneration as a Reticulon protein , 2000, Nature.

[45]  Stroke Therapy Academic Industry Roundtable Recommendations for standards regarding preclinical neuroprotective and restorative drug development. , 1999, Stroke.