Acute and Chronic Administration of the Branched-Chain Amino Acids Decreases Nerve Growth Factor in Rat Hippocampus
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M. Bogo | L. W. Kist | G. Scaini | E. Streck | T. C. Pereira | P. F. Schuck | G. C. Ferreira | F. Mina | C. B. Furlanetto | I. Jeremias | Lis Mairá Mello-Santos
[1] Victor A. McKusick,et al. The Metabolic Basis of Inherited Disease , 2015 .
[2] C. Vargas,et al. Protein and lipid damage in maple syrup urine disease patients: l-carnitine effect , 2013, International Journal of Developmental Neuroscience.
[3] J. Quevedo,et al. Antioxidant administration prevents memory impairment in an animal model of maple syrup urine disease , 2012, Behavioural Brain Research.
[4] M. Bogo,et al. Evaluation of Acetylcholinesterase in an Animal Model of Maple Syrup Urine Disease , 2012, Molecular Neurobiology.
[5] C. Dutra-filho,et al. In vivo neuroprotective effect of L-carnitine against oxidative stress in maple syrup urine disease , 2011, Metabolic Brain Disease.
[6] A. Terry,et al. Age-dependent alterations in nerve growth factor (NGF)-related proteins, sortilin, and learning and memory in rats , 2011, Physiology & Behavior.
[7] M. Pérez,et al. The Intra-Hippocampal Leucine Administration Impairs Memory Consolidation and LTP Generation in Rats , 2010, Cellular and Molecular Neurobiology.
[8] G. Leipnitz,et al. α-Ketoisocaproic acid and leucine provoke mitochondrial bioenergetic dysfunction in rat brain , 2010, Brain Research.
[9] T. Sejnowski,et al. NGF Is Essential for Hippocampal Plasticity and Learning , 2009, The Journal of Neuroscience.
[10] Cunxian Song,et al. Recombinant human NGF-loaded microspheres promote survival of basal forebrain cholinergic neurons and improve memory impairments of spatial learning in the rat model of Alzheimer's disease with fimbria-fornix lesion , 2009, Neuroscience Letters.
[11] C. Callaghan,et al. Deficits in LTP and recognition memory in the genetically hypertensive rat are associated with decreased expression of neurotrophic factors and their receptors in the dentate gyrus , 2009, Behavioural Brain Research.
[12] G. Wegener,et al. Reference genes for normalization: A study of rat brain tissue , 2008, Synapse.
[13] F. Dal-Pizzol,et al. Inhibition of brain creatine kinase activity after renal ischemia is attenuated by N-acetylcysteine and deferoxamine administration , 2008, Neuroscience Letters.
[14] F. Dal-Pizzol,et al. Oxidative stress and metabolism in animal model of colitis induced by dextran sulfate sodium , 2007, Journal of gastroenterology and hepatology.
[15] H. Federoff,et al. Proteolytic processing of proNGF is necessary for mature NGF regulated secretion from neurons. , 2007, Biochemical and biophysical research communications.
[16] T. Hökfelt,et al. Changes in brain cholinergic markers and spatial learning in old galanin-overexpressing mice , 2007, Brain Research.
[17] A. Sitta,et al. Evidence that oxidative stress is increased in plasma from patients with maple syrup urine disease , 2006, Metabolic Brain Disease.
[18] C. Vargas,et al. A chemically-induced acute model of maple syrup urine disease in rats for neurochemical studies , 2006, Journal of Neuroscience Methods.
[19] F. Dal-Pizzol,et al. N-acetylcysteine and deferoxamine reduce pulmonary oxidative stress and inflammation in rats after coal dust exposure. , 2005, Environmental research.
[20] H. Çıralık,et al. Beneficial effects of N-acetylcysteine on acetic acid-induced colitis in rats. , 2005, The Tohoku journal of experimental medicine.
[21] L. Maffei,et al. Intranasal administration of nerve growth factor (NGF) rescues recognition memory deficits in AD11 anti-NGF transgenic mice. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[22] M. Wajner,et al. Evaluation of the mechanisms involved in leucine-induced oxidative damage in cerebral cortex of young rats , 2005, Free radical research.
[23] E. Snyder,et al. Differentiation and tropic/trophic effects of exogenous neural precursors in the adult spinal cord , 2004, The Journal of comparative neurology.
[24] S. Schönberger,et al. Dysmyelination in the brain of adolescents and young adults with maple syrup urine disease. , 2004, Molecular genetics and metabolism.
[25] Michael E. Andrades,et al. Treatment with N-acetylcysteine plus deferoxamine protects rats against oxidative stress and improves survival in sepsis* , 2004, Critical care medicine.
[26] M. Wajner,et al. Inhibition of brain energy metabolism by the alpha-keto acids accumulating in maple syrup urine disease. , 2003, Biochimica et biophysica acta.
[27] M. Wajner,et al. Induction of oxidative stress in rat brain by the metabolites accumulating in maple syrup urine disease , 2003, International Journal of Developmental Neuroscience.
[28] M. Wajner,et al. Creatine Kinase Activity from Rat Brain Is Inhibited by Branched-Chain Amino Acids in Vitro , 2003, Neurochemical Research.
[29] Xiaoqing Yuan,et al. Improvement of spatial learning and memory after adenovirus-mediated transfer of the nerve growth factor gene to aged rat brain. , 2002, Human gene therapy.
[30] M. Wajner,et al. Stimulation of Lipid Peroxidation in Vitro in Rat Brain by the Metabolites Accumulating in Maple Syrup Urine Disease , 2002, Metabolic Brain Disease.
[31] C. Vargas,et al. Reduction of large neutral amino acid levels in plasma and brain of hyperleucinemic rats , 2001, Neurochemistry International.
[32] C. Scriver,et al. The Metabolic and Molecular Bases of Inherited Disease, 8th Edition 2001 , 2001, Journal of Inherited Metabolic Disease.
[33] H. Mehmet,et al. Primary Human Fibroblasts from a Maple Syrup Urine Disease Patient Undergo Apoptosis following Exposure to Physiological Concentrations of Branched Chain Amino Acids , 2000, Annals of the New York Academy of Sciences.
[34] M. Wajner,et al. Inhibition of glutamate uptake into synaptic vesicles of rat brain by the metabolites accumulating in maple syrup urine disease , 2000, Journal of the Neurological Sciences.
[35] C. Vargas,et al. Reduction of large neutral amino acid concentrations in plasma and CSF of patients with maple syrup urine disease during crises , 2000, Journal of Inherited Metabolic Disease.
[36] D. Taylor,et al. Branched chain amino acids induce apoptosis in neural cells without mitochondrial membrane depolarization or cytochrome c release: implications for neurological impairment associated with maple syrup urine disease. , 2000, Molecular biology of the cell.
[37] M. Wajner,et al. Chronic early leucine administration induces behavioral deficits in rats. , 1999, Life sciences.
[38] C. Vargas,et al. Reduction of plasma concentrations of large neutral amino acids in patients with maple syrup urine disease during crises , 1999, Archives of disease in childhood.
[39] R. Jope,et al. Oxidative stress differentially modulates phosphorylation of ERK, p38 and CREB induced by NGF or EGF in PC12 cells☆ , 1999, Neurobiology of Aging.
[40] W. Anderson,et al. In Vitro Effects of Oxygen-derived Free Radicals on Type I and Type II cAMP-Dependent Protein Kinases* , 1998, The Journal of Biological Chemistry.
[41] Ted Abel,et al. Positive and negative regulatory mechanisms that mediate long-term memory storage 1 Published on the World Wide Web on 13 January 1998. 1 , 1998, Brain Research Reviews.
[42] J. van Oirschot,et al. Low-dose N-acetylcysteine protects rats against endotoxin-mediated oxidative stress, but high-dose increases mortality. , 1998, American journal of respiratory and critical care medicine.
[43] H. R. Zielke,et al. Effect of α-Ketoisocaproate and Leucine on the in Vivo Oxidation of Glutamate and Glutamine in the Rat Brain , 1997, Neurochemical Research.
[44] E. Alleva,et al. Nerve growth factor affects passive avoidance learning and retention in developing mice , 1996, Brain Research Bulletin.
[45] M. Knipper,et al. Short-term modulation of glutamatergic synapses in adult rat hippocampus by NGF. , 1994, Neuroreport.
[46] D. Chitayat,et al. Maple syrup urine disease: Interrelations between branched-chain amino-, oxo- and hydroxyacids; implications for treatment; associations with CNS dysmyelination , 1992, Journal of Inherited Metabolic Disease.
[47] T. Kameyama,et al. Memory impairment and morphological changes in rats after continuous infusion of active fragment of anti-nerve growth factor-antibody. , 1991, Research communications in chemical pathology and pharmacology.
[48] M. Greenberg,et al. CREB: a Ca(2+)-regulated transcription factor phosphorylated by calmodulin-dependent kinases. , 1991, Science.
[49] D. Kaplan,et al. High-affinity NGF binding requires coexpression of the trk proto-oncogene and the low-affinity NGF receptor , 1991, Nature.
[50] P. Harper,et al. Maple syrup urine disease (branched chain ketoaciduria). , 1990, The American journal of pathology.
[51] R. Levi‐montalcini,et al. The nerve growth factor 35 years later. , 1987, Science.
[52] F. Gage,et al. Amelioration of cholinergic neuron atrophy and spatial memory impairment in aged rats by nerve growth factor , 1987, Nature.
[53] H. Thoenen,et al. Cholinergic denervation of the rat hippocampus by fimbrial transection leads to a transient accumulation of nerve growth factor (NGF) without change in mRNANGF content , 1986, Neuroscience Letters.
[54] A. Ross,et al. Gene transfer and molecular cloning of the human NGF receptor. , 1986, Science.
[55] M. Johnston,et al. Choline acetyltransferase activity in striatum of neonatal rats increased by nerve growth factor. , 1985, Science.
[56] D. Tribble,et al. Myelin proteins: degradation in rat brain initiated by metabolites causative of maple syrup urine disease. , 1983, Biochemical and biophysical research communications.
[57] M. Schwab,et al. NGF-mediated increase of choline acetyltransferase (ChAT) in the neonatal rat forebrain: evidence for a physiological role of NGF in the brain? , 1983, Brain research.
[58] T. Kunishita,et al. Abnormal protein and lipid compositions of the cerebral myelin of a patient with maple syrup urine disease. , 1983, The Japanese journal of experimental medicine.
[59] J. Mowbray,et al. CONTROL OF PYRUVATE AND β‐HYDROXYBUTYRATE UTILIZATION IN RAT BRAIN MITOCHONDRIA AND ITS RELEVANCE TO PHENYLKETONURIA AND MAPLE SYRUP URINE DISEASE , 1976, Journal of neurochemistry.
[60] D. Goldstein,et al. Dopamine‐β‐hydroxylase activity in human cerebrospinal fluid , 1976 .
[61] L. E. Holt,et al. MAPLE SYRUP URINE DISEASE, WITH PARTICULAR REFERENCE TO DIETOTHERAPY. , 1964, Pediatrics.
[62] Melvin Lee,et al. Influence of Alpha-Ketoacids on the Respiration of Brain in vitro. ∗ , 1963, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.
[63] L. Woolf,et al. “Maple Syrup Urine Disease” , 1959, British medical journal.
[64] J. Dancis,et al. “Maple Syrup Urine Disease” , 1959, British medical journal.
[65] Oliver H. Lowry,et al. Protein measurement with the Folin phenol reagent. , 1951, The Journal of biological chemistry.
[66] L. Feksa,et al. Effect of Leucine Administration to Female Rats During Pregnancy and Lactation on Oxidative Stress and Enzymes Activities of Phosphoryltransfer Network in Cerebral Cortex and Hippocampus of the Offspring , 2012, Neurochemical Research.
[67] C. Caltagirone,et al. Brain and serum levels of nerve growth factor in a rat model of Alzheimer's disease. , 2011, Journal of Alzheimer's disease : JAD.
[68] M. Wajner,et al. Inhibition of Brain Energy Metabolism by the Branched-chain Amino Acids Accumulating in Maple Syrup Urine Disease , 2007, Neurochemical Research.
[69] M. Martín,et al. N-acetyl-cysteine abolishes hydrogen peroxide-induced modification of eukaryotic initiation factor 4F activity via distinct signalling pathways. , 2006, Cellular signalling.
[70] M. Sofroniew,et al. Nerve growth factor signaling, neuroprotection, and neural repair. , 2001, Annual review of neuroscience.
[71] W R Markesbery,et al. Oxidative stress hypothesis in Alzheimer's disease. , 1997, Free radical biology & medicine.
[72] H. R. Zielke,et al. Elevation of amino acids in the interstitial space of the rat brain following infusion of large neutral amino and keto acids by microdialysis: alpha-ketoisocaproate infusion. , 1996, Developmental Neuroscience.
[73] H. R. Zielke,et al. Elevation of amino acids in the interstitial space of the rat brain following infusion of large neutral amino and keto acids by microdialysis: leucine infusion. , 1996, Developmental neuroscience.
[74] S. De Flora,et al. Chemopreventive properties and mechanisms of N‐acetylcysteine. The experimental background , 1995, Journal of cellular biochemistry. Supplement.
[75] S. Flora,et al. N‐acetyl‐l‐cysteine , 1993, Journal of cellular biochemistry. Supplement.
[76] U. Wendel. Disorders of Branched-Chain Amino Acid Metabolism , 1990 .
[77] Charles R.scriver,et al. The Metabolic basis of inherited disease , 1989 .