Magnetic resonance detects changes in phosphocholine associated with Ras activation and inhibition in NIH 3T3 cells
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M. Leach | S. Ronen | L. Jackson | M. Beloueche | SM Ronen | LE Jackson | MO Leach
[1] C. Bowden,et al. Phase I and pharmacokinetic study of farnesyl protein transferase inhibitor R115777 in advanced cancer. , 2000, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[2] F. Podo. Tumour phospholipid metabolism , 1999, NMR in biomedicine.
[3] J. Downward,et al. Multiple Ras Effector Pathways Contribute to G1Cell Cycle Progression* , 1999, The Journal of Biological Chemistry.
[4] J. Griffiths,et al. Magnetic resonance detects metabolic changes associated with chemotherapy-induced apoptosis , 1999, British Journal of Cancer.
[5] Z. Bhujwalla,et al. Malignant transformation alters membrane choline phospholipid metabolism of human mammary epithelial cells. , 1999, Cancer research.
[6] L. Neckers,et al. The benzoquinone ansamycin 17-allylamino-17-demethoxygeldanamycin binds to HSP90 and shares important biologic activities with geldanamycin , 1998, Cancer Chemotherapy and Pharmacology.
[7] K. Skorecki,et al. Induction of Cytosolic Phospholipase A2 by Oncogenic Ras in Human Non-small Cell Lung Cancer* , 1997, The Journal of Biological Chemistry.
[8] S. Williams,et al. Immortalization and transformation are associated with specific alterations in choline metabolism. , 1996, Cancer research.
[9] H. Degani,et al. Variations in energy and phospholipid metabolism in normal and cancer human mammary epithelial cells. , 1996, Anticancer research.
[10] F. Podo,et al. Detection of phosphatidylcholine-specific phospholipase C in NIH-3T3 fibroblasts and their H-ras transformants: NMR and immunochemical studies. , 1996, Anticancer research.
[11] T. Wieder,et al. Evidence for Phosphorylation of CTP:Phosphocholine Cytidylyltransferase by Multiple Proline-directed Protein Kinases (*) , 1996, The Journal of Biological Chemistry.
[12] H. Degani,et al. Simultaneous extraction of cellular lipids and water‐soluble metabolites: Evaluation by NMR spectroscopy , 1996, Magnetic resonance in medicine.
[13] C. Kent,et al. Early increase in choline kinase activity upon induction of the H-ras oncogene in mouse fibroblast cell lines. , 1995, Archives of biochemistry and biophysics.
[14] M. Diaz-Meco,et al. Evidence for a Bifurcation of the Mitogenic Signaling Pathway Activated by Ras and Phosphatidylcholine-hydrolyzing Phospholipase C (*) , 1995, The Journal of Biological Chemistry.
[15] D. Spandidos,et al. Mutations of ras genes in human tumors (review). , 1995, International journal of oncology.
[16] S. Ng,et al. Ras transformation results in an elevated level of cyclin D1 and acceleration of G1 progression in NIH 3T3 cells , 1995, Molecular and cellular biology.
[17] A. Cuadrado,et al. Activation of type D phospholipase by serum stimulation and ras-induced transformation in NIH3T3 cells. , 1994, Oncogene.
[18] Roger J. Davis,et al. cPLA2 is phosphorylated and activated by MAP kinase , 1993, Cell.
[19] W. Negendank,et al. Studies of human tumors by MRS: A review , 1992, NMR in biomedicine.
[20] H Degani,et al. Lipid metabolism in large T47D human breast cancer spheroids: 31P- and 13C-NMR studies of choline and ethanolamine uptake. , 1992, Biochimica et biophysica acta.
[21] M. Leach,et al. The phosphocholine and glycerophosphocholine content of an oestrogen-sensitive rat mammary tumour correlates strongly with growth rate. , 1991, British Journal of Cancer.
[22] M. Diaz-Meco,et al. Kinetic evidence of a rapid activation of phosphatidylcholine hydrolysis by Ki-ras oncogene. Possible involvement in late steps of the mitogenic cascade. , 1990, The Journal of biological chemistry.
[23] C. Kent,et al. Altered phosphatidylcholine metabolism in C3H10T1/2 cells transfected with the Harvey-ras oncogene. , 1990, The Journal of biological chemistry.
[24] L. Beck,et al. Inhibition of isoprenoid biosynthesis and the post-translational modification of pro-p21. , 1990, Journal of Biological Chemistry.
[25] J. Lacal. Diacylglycerol production in Xenopus laevis oocytes after microinjection of p21ras proteins is a consequence of activation of phosphatidylcholine metabolism , 1990, Molecular and cellular biology.
[26] C. Marshall,et al. Stimulation of phosphatidylcholine hydrolysis, diacylglycerol release, and arachidonic acid production by oncogenic ras is a consequence of protein kinase C activation. , 1989, The Journal of biological chemistry.
[27] S. Aaronson,et al. Novel source of 1,2-diacylglycerol elevated in cells transformed by Ha-ras oncogene , 1987, Nature.