Cryptogenic Stroke in Relation to Genetic Variation in Clotting Factors and Other Genetic Polymorphisms Among Young Men and Women
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L. Wechsler | R. Wityk | H. Austin | W. Hooper | B. Evatt | M. Chimowitz | S. Chaturvedi | C. Sila | B. Coull | J. Wilterdink | H. Hill | P. Mitsias | E. Walz
[1] R. Hegele. SNP judgments and freedom of association. , 2002, Arteriosclerosis, thrombosis, and vascular biology.
[2] Vilmundur Gudnason,et al. Localization of a susceptibility gene for common forms of stroke to 5q12. , 2002, American journal of human genetics.
[3] J. Lalouel,et al. Power and replication in case-control studies. , 2002, American journal of hypertension.
[4] R. Junker,et al. Symptomatic Ischemic Stroke in Full-Term Neonates: Role of Acquired and Genetic Prothrombotic Risk Factors , 2000, Stroke.
[5] C. Mannhalter,et al. The 4G/4G genotype at nucleotide position −675 in the promotor region of the plasminogen activator inhibitor 1 (PAI‐1) gene is less frequent in young patients with minor stroke than in controls , 2000, British journal of haematology.
[6] N. Wenger,et al. The role of the t-PA I/D and PAI-1 4G/5G polymorphisms in African-American adults with a diagnosis of myocardial infarction or venous thromboembolism. , 2000, Thrombosis research.
[7] B. Damasceno,et al. Inherited Thrombophilia as a Risk Factor for the Development of Ischemic Stroke in Young Adults , 2000, Thrombosis and Haemostasis.
[8] E. Ringelstein,et al. Tissue plasminogen activator and plasminogen activator inhibitor in patients with acute ischemic stroke: relation to stroke etiology. , 1999, Neurological research.
[9] A. von Eckardstein,et al. Lipoprotein (a) and genetic polymorphisms of clotting factor V, prothrombin, and methylenetetrahydrofolate reductase are risk factors of spontaneous ischemic stroke in childhood. , 1999, Blood.
[10] N. Wenger,et al. The relationship between polymorphisms in the endothelial cell nitric oxide synthase gene and the platelet GPIIIa gene with myocardial infarction and venous thromboembolism in African Americans. , 1999, Chest.
[11] I. Perry. Homocysteine and Risk of Stroke , 1999, Journal of cardiovascular risk.
[12] O. Benavente,et al. Stroke: part I. A clinical update on prevention. , 1999, American Family Physician.
[13] D. Wilcken,et al. Common methylenetetrahydrofolate reductase gene mutation leads to hyperhomocysteinemia but not to vascular disease: the result of a meta-analysis. , 1998, Circulation.
[14] M. Margaglione,et al. Increased Risk for Venous Thrombosis in Carriers of the Prothrombin GA20210 Gene Variant , 1998, Annals of Internal Medicine.
[15] T. Raghunathan,et al. Risk of stroke in young women and two prothrombotic mutations: factor V Leiden and prothrombin gene variant (G20210A) , 1998, Stroke.
[16] A. Rumley,et al. Hemostatic factors as predictors of ischemic heart disease and stroke in the Edinburgh Artery Study. , 1997, Arteriosclerosis, thrombosis, and vascular biology.
[17] A. Hofman,et al. Tissue plasminogen activator and risk of myocardial infarction. The Rotterdam Study. , 1997, Circulation.
[18] P. Reitsma,et al. A common genetic variation in the 3'-untranslated region of the prothrombin gene is associated with elevated plasma prothrombin levels and an increase in venous thrombosis. , 1996, Blood.
[19] G. Murray,et al. Lack of association between angiotensin converting enzyme gene insertion/deletion polymorphism and stroke , 1995, Journal of hypertension.
[20] H. Markus,et al. Angiotensin-converting enzyme gene deletion polymorphism. A new risk factor for lacunar stroke but not carotid atheroma. , 1995, Stroke.
[21] P. Ridker,et al. Mutation in the gene coding for coagulation factor V and the risk of myocardial infarction, stroke, and venous thrombosis in apparently healthy men. , 1995, The New England journal of medicine.
[22] P. Reitsma,et al. High risk of thrombosis in patients homozygous for factor V Leiden (activated protein C resistance) , 1995, Blood.
[23] N. Carter,et al. Molecular approach to assessing the genetic risk of cerebral infarction: deletion polymorphism in the gene encoding angiotensin 1-converting enzyme. , 1994, Journal of human hypertension.
[24] Pieter H. Reitsma,et al. Mutation in blood coagulation factor V associated with resistance to activated protein C , 1994, Nature.
[25] J. Manson,et al. Prospective study of endogenous tissue plasminogen activator and risk of stroke , 1994, The Lancet.
[26] R. Poole,et al. Left Atrial Spontaneous Echo Contrast Is Highly Associated With Previous Stroke in Patients With Atrial Fibrillation or Mitral Stenosis , 1993, Stroke.
[27] Philippe Amouyel,et al. Deletion polymorphism in the gene for angiotensin-converting enzyme is a potent risk factor for myocardial infarction , 1992, Nature.
[28] J. Bogousslavsky,et al. Ischemic stroke in patients under age 45. , 1992, Neurologic clinics.
[29] D. Hosmer,et al. Applied Logistic Regression , 1991 .
[30] G. Beck,et al. Etiology of motor or sensory stroke: A prospective study of the predictive value of clinical and radiological features , 1991, Annals of neurology.
[31] S. Humphries,et al. Variation in the Promoter Region of the β Fibrinogen Gene Is Associated with Plasma Fibrinogen Levels in Smokers and Non-Smokers , 1991, Thrombosis and Haemostasis.
[32] R. Hart,et al. Hematologic disorders and ischemic stroke. A selective review. , 1990, Stroke.
[33] Daniel B Hier,et al. The Stroke Data Bank: design, methods, and baseline characteristics. , 1988, Stroke.
[34] Tx Station. Stata Statistical Software: Release 7. , 2001 .
[35] G. Hallmans,et al. Tissue plasminogen activator, plasminogen activator inhibitor-1, and tissue plasminogen activator/plasminogen activator inhibitor-1 complex as risk factors for the development of a first stroke. , 2000, Stroke.
[36] E. Taioli,et al. The G20210A mutation of the prothrombin gene in patients with previous first episodes of deep-vein thrombosis: prevalence and association with factor V G1691A, methylenetetrahydrofolate reductase C677T and plasma prothrombin levels. , 1999, Thrombosis research.
[37] R. Badenhop,et al. A smoking–dependent risk of coronary artery disease associated with a polymorphism of the endothelial nitric oxide synthase gene , 1996, Nature Medicine.
[38] R. Matthews,et al. A candidate genetic risk factor for vascular disease: a common mutation in methylenetetrahydrofolate reductase , 1995, Nature Genetics.
[39] David W. Hosmer,et al. Best subsets logistic regression , 1989 .