Antisense oligonucleotide therapeutics for iron–sulphur cluster deficiency myopathy
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[1] E. Holme,et al. Clinical manifestation and a new ISCU mutation in iron-sulphur cluster deficiency myopathy. , 2009, Brain : a journal of neurology.
[2] G. van Ommen,et al. In vivo comparison of 2′‐O‐methyl phosphorothioate and morpholino antisense oligonucleotides for Duchenne muscular dystrophy exon skipping , 2009, The journal of gene medicine.
[3] C. Lázaro,et al. Antisense therapeutics for neurofibromatosis type 1 caused by deep intronic mutations , 2009, Human mutation.
[4] T. Rouault,et al. Iron-sulfur cluster biogenesis and human disease. , 2008, Trends in genetics : TIG.
[5] M. Holmberg,et al. Myopathy with lactic acidosis is linked to chromosome 12q23.3-24.11 and caused by an intron mutation in the ISCU gene resulting in a splicing defect. , 2008, Human molecular genetics.
[6] D. Hernandez,et al. Splice mutation in the iron-sulfur cluster scaffold protein ISCU causes myopathy with exercise intolerance. , 2008, American journal of human genetics.
[7] Johan T den Dunnen,et al. Local dystrophin restoration with antisense oligonucleotide PRO051. , 2007, The New England journal of medicine.
[8] M. Ugarte,et al. Propionic and methylmalonic acidemia: antisense therapeutics for intronic variations causing aberrantly spliced messenger RNA. , 2007, American journal of human genetics.
[9] G. van Ommen,et al. Antisense-mediated exon skipping: a versatile tool with therapeutic and research applications. , 2007, RNA.
[10] E. Buratti,et al. RNA structure is a key regulatory element in pathological ATM and CFTR pseudoexon inclusion events , 2007, Nucleic acids research.
[11] H. Elsässer,et al. Mechanisms of iron-sulfur protein maturation in mitochondria, cytosol and nucleus of eukaryotes. , 2006, Biochimica et biophysica acta.
[12] Francisco E. Baralle,et al. Genomic variants in exons and introns: identifying the splicing spoilers , 2004, Nature Reviews Genetics.
[13] Michael Zuker,et al. Mfold web server for nucleic acid folding and hybridization prediction , 2003, Nucleic Acids Res..
[14] L. Chasin,et al. Multiple Splicing Defects in an Intronic False Exon , 2000, Molecular and Cellular Biology.
[15] C. Liew,et al. A modular domain of NifU, a nitrogen fixation cluster protein, is highly conserved in evolution , 1996, Journal of Molecular Evolution.
[16] E. Hoffman,et al. A renaissance for antisense oligonucleotide drugs in neurology: exon skipping breaks new ground. , 2009, Archives of neurology.