Immunochemotherapy of malignant glioma: synergistic activity of CD95 ligand and chemotherapeutics
暂无分享,去创建一个
[1] L. French,et al. Fas and Fas ligand in embryos and adult mice: ligand expression in several immune-privileged tissues and coexpression in adult tissues characterized by apoptotic cell turnover , 1996, The Journal of cell biology.
[2] M. Weller,et al. T cell apoptosis induced by interleukin-2 deprivation or transforming growth factor-beta 2: modulation by the phosphatase inhibitors okadaic acid and calyculin A. , 1995, Experimental cell research.
[3] D. Green,et al. Fas Ligand-Induced Apoptosis as a Mechanism of Immune Privilege , 1995, Science.
[4] P. Kleihues,et al. The p53 gene and its role in human brain tumors , 1995, Glia.
[5] D. Bellgrau,et al. A role for CD95 ligand in preventing graft rejection , 1995, Nature.
[6] Adriano Fontana,et al. The failure of current immunotherapy for malignant glioma. Tumor-derived TGF-β, T-cell apoptosis, and the immune privilege of the brain , 1995, Brain Research Reviews.
[7] K. Scotto,et al. Ceramide synthase mediates daunorubicin-induced apoptosis: An alternative mechanism for generating death signals , 1995, Cell.
[8] M. Weller,et al. Local Fas/APO‐1 (CD95) ligand‐mediated tumor cell killing in vivo , 1995, European journal of immunology.
[9] M. Weller,et al. Fas/APO-1 gene transfer for human malignant glioma. , 1995, Cancer research.
[10] M. Weller,et al. Protooncogene bcl-2 gene transfer abrogates Fas/APO-1 antibody-mediated apoptosis of human malignant glioma cells and confers resistance to chemotherapeutic drugs and therapeutic irradiation. , 1995, The Journal of clinical investigation.
[11] D. Green,et al. FAS-induced apoptosis is mediated via a ceramide-initiated RAS signaling pathway. , 1995, Immunity.
[12] John Calvin Reed. Bcl-2: prevention of apoptosis as a mechanism of drug resistance. , 1995, Hematology/oncology clinics of North America.
[13] C. Thompson,et al. Apoptosis in the pathogenesis and treatment of disease , 1995, Science.
[14] S. Nagata,et al. The Fas death factor , 1995, Science.
[15] D. Louis,et al. Human gliomas with wild-type p53 express bcl-2. , 1995, Cancer research.
[16] D. Green,et al. Apoptosis and cancer: the failure of controls on cell death and cell survival. , 1995, Critical reviews in oncology/hematology.
[17] R. Testi,et al. Apoptotic signaling through CD95 (Fas/Apo-1) activates an acidic sphingomyelinase , 1994, The Journal of experimental medicine.
[18] M. Weller,et al. Anti-Fas/APO-1 antibody-mediated apoptosis of cultured human glioma cells. Induction and modulation of sensitivity by cytokines. , 1994, The Journal of clinical investigation.
[19] D. Fisher. Apoptosis in cancer therapy: Crossing the threshold , 1994, Cell.
[20] D. Constam,et al. Transforming growth factor‐β2 induces apoptosis of murine T cell clones without down‐regulating bcl‐2 mRNA expression , 1994, European journal of immunology.
[21] C. Winterford,et al. Apoptosis. Its significance in cancer and cancer Therapy , 1994, Cancer.
[22] G. Lesser,et al. The chemotherapy of high-grade astrocytomas. , 1994, Seminars in oncology.
[23] Erwin G. Van Meir,et al. Analysis of the p53 gene and its expression in human glioblastoma cells. , 1994, Cancer research.
[24] John Calvin Reed. Bcl-2 and the regulation of programmed cell death , 1994, The Journal of cell biology.
[25] Takashi Suda,et al. Molecular cloning and expression of the fas ligand, a novel member of the tumor necrosis factor family , 1993, Cell.
[26] P. Graham. Meta‐analysis of radiation therapy with and without adjuvant chemotherapy for malignant gliomas in adults , 1993, Cancer.
[27] S. Nagata,et al. Lethal effect of the anti-Fas antibody in mice , 1993, Nature.
[28] S. Yonehara,et al. Overcoming tumor necrosis factor and drug resistance of human tumor cell lines by combination treatment with anti-Fas antibody and drugs or toxins. , 1993, Cancer research.
[29] G. Deng,et al. Suppression of apoptosis in a cytotoxic T-cell line by interleukin 2-mediated gene transcription and deregulated expression of the protooncogene bcl-2. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[30] D. Louis,et al. Comparative Study of p53 Gene and Protein Alterations in Human Astrocytic Tumors , 1993, Journal of neuropathology and experimental neurology.
[31] J. Safrit,et al. Sensitivity of resistant human tumor cell lines to tumor necrosis factor and adriamycin used in combination: correlation between down-regulation of tumor necrosis factor-messenger RNA induction and overcoming resistance. , 1992, Cancer research.
[32] E. Appella,et al. Human wild-type p53 adopts a unique conformational and phosphorylation state in vivo during growth arrest of glioblastoma cells. , 1992, Oncogene.
[33] S. Rosenberg,et al. Augmentation of antitumor efficacy by the combination of recombinant tumor necrosis factor and chemotherapeutic agents in vivo. , 1989, Cancer research.
[34] S. Yonehara,et al. A cell-killing monoclonal antibody (anti-Fas) to a cell surface antigen co-downregulated with the receptor of tumor necrosis factor , 1989, The Journal of experimental medicine.
[35] H. Karasuyama,et al. Establishment of mouse cell lines which constitutively secrete large quantities of interleukin 2, 3, 4 or 5, using modified cDNA expression vectors , 1988, European journal of immunology.
[36] D. S. Coffey,et al. Synergistic enhancement by tumor necrosis factor of in vitro cytotoxicity from chemotherapeutic drugs targeted at DNA topoisomerase II. , 1987, Cancer research.
[37] A. Matter,et al. Anti‐tumor effects of tumor necrosis factor in combination with chemotherapeutic agents , 1987, International journal of cancer.
[38] R. Milroy,et al. Potentiation of Tumour Necrosis Factor-mediated Cell Killing by VP16 on Human Ovarian Cancer Cell Lines. In vitro Results and Clinical Implications , 1993 .
[39] P. Kleihues,et al. Astrocytomas Expression of Fas/apo-1 during the Progression of Updated Version Citing Articles E-mail Alerts Expression of Fas/apo-1 during the Progression of Astrocytomas , 2022 .