Transplantation of endothelial progenitor cells attenuated paraquat-induced acute lung injury via miR-141-3p-Notch-Nrf2 axis

[1]  J. Shao,et al.  Potential mechanisms of microRNA-141-3p to alleviate chronic inflammatory pain by downregulation of downstream target gene HMGB1: in vitro and in vivo studies , 2017, Gene Therapy.

[2]  Yingchun He,et al.  MicroRNA-34a Suppresses Autophagy in Alveolar Type II Epithelial Cells in Acute Lung Injury by Inhibiting FoxO3 Expression , 2017, Inflammation.

[3]  J. Hao,et al.  microRNA-1246 mediates lipopolysaccharide-induced pulmonary endothelial cell apoptosis and acute lung injury by targeting angiotensin-converting enzyme 2. , 2017, American journal of translational research.

[4]  Yulin Li,et al.  Macrophage-Derived mir-155-Containing Exosomes Suppress Fibroblast Proliferation and Promote Fibroblast Inflammation during Cardiac Injury. , 2017, Molecular therapy : the journal of the American Society of Gene Therapy.

[5]  Jun Liu,et al.  Downregulation of miR-181a protects mice from LPS-induced acute lung injury by targeting Bcl-2. , 2016, Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie.

[6]  Jidong Zhang,et al.  MicroRNA-7 Deficiency Ameliorates the Pathologies of Acute Lung Injury through Elevating KLF4 , 2016, Front. Immunol..

[7]  S. Bray Notch signalling in context , 2016, Nature Reviews Molecular Cell Biology.

[8]  Y. Tseng,et al.  Autologous endothelial progenitor cells improve allograft survival in porcine lung transplantation with prolonged ischemia. , 2016, Annals of translational medicine.

[9]  Rurong Tang,et al.  Down-regulation of microRNA-126-5p contributes to overexpression of VEGFA in lipopolysaccharide-induced acute lung injury , 2016, Biotechnology Letters.

[10]  Chunjin Li,et al.  Differential Expression of microRNAs in the Ovaries from Letrozole-Induced Rat Model of Polycystic Ovary Syndrome. , 2016, DNA and cell biology.

[11]  R. Ain,et al.  MicroRNA-141-3p and miR-200a-3p regulate insulin-like growth factor 2 during mouse placental development , 2015, Molecular and Cellular Endocrinology.

[12]  A. Uezumi,et al.  Evidence of Notch-Hesr-Nrf2 Axis in Muscle Stem Cells, but Absence of Nrf2 Has No Effect on Their Quiescent and Undifferentiated State , 2015, PloS one.

[13]  Z. Xia,et al.  Smoke inhalation injury repaired by a bone marrow-derived mesenchymal stem cell paracrine mechanism: Angiogenesis involving the Notch signaling pathway , 2015, The journal of trauma and acute care surgery.

[14]  Xiao-rong Zhan,et al.  MicroRNA-185 regulates expression of lipid metabolism genes and improves insulin sensitivity in mice with non-alcoholic fatty liver disease. , 2014, World journal of gastroenterology.

[15]  Ho-Yeon Song,et al.  The dose of cyclophosphamide for treating paraquat-induced rat lung injury , 2013, The Korean journal of internal medicine.

[16]  Yu Du,et al.  [Predictive value of 3 methods in severity evaluation and prognosis of acute paraquat poisoning]. , 2013, Zhong nan da xue xue bao. Yi xue ban = Journal of Central South University. Medical sciences.

[17]  M. Majesky,et al.  Patterning the artery wall by lateral induction of Notch signaling. , 2012, Circulation.

[18]  Y. Tsai,et al.  Transplantation of Endothelial Progenitor Cells Improves Pulmonary Endothelial Function and Gas Exchange in Rabbits with Endotoxin-Induced Acute Lung Injury , 2011, Anesthesia and analgesia.

[19]  M. Nogueira-Barbosa,et al.  Severe paraquat poisoning: clinical and radiological findings in a survivor. , 2010, Jornal brasileiro de pneumologia : publicacao oficial da Sociedade Brasileira de Pneumologia e Tisilogia.

[20]  M. Harmsen,et al.  Endothelial progenitor cell-based neovascularization: implications for therapy. , 2009, Trends in molecular medicine.

[21]  Q. Gong,et al.  Fructose-1,6-diphosphate attenuates acute lung injury induced by lipopolysaccharide in mice. , 2008, International immunopharmacology.

[22]  Ming-Wei Lin,et al.  Autologous Transplantation of Endothelial Progenitor Cells Attenuates Acute Lung Injury in Rabbits , 2008, Anesthesiology.

[23]  A. Fischer,et al.  Delta–Notch—and then? Protein interactions and proposed modes of repression by Hes and Hey bHLH factors , 2007, Nucleic acids research.

[24]  A. Avogaro,et al.  Circulating Progenitor Cells Are Reduced in Patients with Severe Lung Disease , 2006, Stem cells.

[25]  S. Raisuddin,et al.  Effects of Paraquat on the Freshwater Fish Channa punctata (Bloch): Non-Enzymatic Antioxidants as Biomarkers of Exposure , 2006, Archives of environmental contamination and toxicology.

[26]  V. Ambros The functions of animal microRNAs , 2004, Nature.

[27]  S. Dimmeler,et al.  Endothelial Progenitor Cells: Characterization and Role in Vascular Biology , 2004, Circulation research.

[28]  K. Orito,et al.  Chymase is activated in the pulmonary inflammation and fibrosis induced by paraquat in hamsters. , 2004, The Tohoku journal of experimental medicine.

[29]  Z. Suntres Role of antioxidants in paraquat toxicity. , 2002, Toxicology.

[30]  Thomas D. Schmittgen,et al.  Analysis of relative gene expression data using real-time quantitative PCR and the 2(-Delta Delta C(T)) Method. , 2001, Methods.

[31]  J. Matsumoto,et al.  Further studies of lipid peroxidation in human paraquat poisoning. , 1986, Archives of internal medicine.