Transplantation of endothelial progenitor cells attenuated paraquat-induced acute lung injury via miR-141-3p-Notch-Nrf2 axis
暂无分享,去创建一个
[1] J. Shao,et al. Potential mechanisms of microRNA-141-3p to alleviate chronic inflammatory pain by downregulation of downstream target gene HMGB1: in vitro and in vivo studies , 2017, Gene Therapy.
[2] Yingchun He,et al. MicroRNA-34a Suppresses Autophagy in Alveolar Type II Epithelial Cells in Acute Lung Injury by Inhibiting FoxO3 Expression , 2017, Inflammation.
[3] J. Hao,et al. microRNA-1246 mediates lipopolysaccharide-induced pulmonary endothelial cell apoptosis and acute lung injury by targeting angiotensin-converting enzyme 2. , 2017, American journal of translational research.
[4] Yulin Li,et al. Macrophage-Derived mir-155-Containing Exosomes Suppress Fibroblast Proliferation and Promote Fibroblast Inflammation during Cardiac Injury. , 2017, Molecular therapy : the journal of the American Society of Gene Therapy.
[5] Jun Liu,et al. Downregulation of miR-181a protects mice from LPS-induced acute lung injury by targeting Bcl-2. , 2016, Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie.
[6] Jidong Zhang,et al. MicroRNA-7 Deficiency Ameliorates the Pathologies of Acute Lung Injury through Elevating KLF4 , 2016, Front. Immunol..
[7] S. Bray. Notch signalling in context , 2016, Nature Reviews Molecular Cell Biology.
[8] Y. Tseng,et al. Autologous endothelial progenitor cells improve allograft survival in porcine lung transplantation with prolonged ischemia. , 2016, Annals of translational medicine.
[9] Rurong Tang,et al. Down-regulation of microRNA-126-5p contributes to overexpression of VEGFA in lipopolysaccharide-induced acute lung injury , 2016, Biotechnology Letters.
[10] Chunjin Li,et al. Differential Expression of microRNAs in the Ovaries from Letrozole-Induced Rat Model of Polycystic Ovary Syndrome. , 2016, DNA and cell biology.
[11] R. Ain,et al. MicroRNA-141-3p and miR-200a-3p regulate insulin-like growth factor 2 during mouse placental development , 2015, Molecular and Cellular Endocrinology.
[12] A. Uezumi,et al. Evidence of Notch-Hesr-Nrf2 Axis in Muscle Stem Cells, but Absence of Nrf2 Has No Effect on Their Quiescent and Undifferentiated State , 2015, PloS one.
[13] Z. Xia,et al. Smoke inhalation injury repaired by a bone marrow-derived mesenchymal stem cell paracrine mechanism: Angiogenesis involving the Notch signaling pathway , 2015, The journal of trauma and acute care surgery.
[14] Xiao-rong Zhan,et al. MicroRNA-185 regulates expression of lipid metabolism genes and improves insulin sensitivity in mice with non-alcoholic fatty liver disease. , 2014, World journal of gastroenterology.
[15] Ho-Yeon Song,et al. The dose of cyclophosphamide for treating paraquat-induced rat lung injury , 2013, The Korean journal of internal medicine.
[16] Yu Du,et al. [Predictive value of 3 methods in severity evaluation and prognosis of acute paraquat poisoning]. , 2013, Zhong nan da xue xue bao. Yi xue ban = Journal of Central South University. Medical sciences.
[17] M. Majesky,et al. Patterning the artery wall by lateral induction of Notch signaling. , 2012, Circulation.
[18] Y. Tsai,et al. Transplantation of Endothelial Progenitor Cells Improves Pulmonary Endothelial Function and Gas Exchange in Rabbits with Endotoxin-Induced Acute Lung Injury , 2011, Anesthesia and analgesia.
[19] M. Nogueira-Barbosa,et al. Severe paraquat poisoning: clinical and radiological findings in a survivor. , 2010, Jornal brasileiro de pneumologia : publicacao oficial da Sociedade Brasileira de Pneumologia e Tisilogia.
[20] M. Harmsen,et al. Endothelial progenitor cell-based neovascularization: implications for therapy. , 2009, Trends in molecular medicine.
[21] Q. Gong,et al. Fructose-1,6-diphosphate attenuates acute lung injury induced by lipopolysaccharide in mice. , 2008, International immunopharmacology.
[22] Ming-Wei Lin,et al. Autologous Transplantation of Endothelial Progenitor Cells Attenuates Acute Lung Injury in Rabbits , 2008, Anesthesiology.
[23] A. Fischer,et al. Delta–Notch—and then? Protein interactions and proposed modes of repression by Hes and Hey bHLH factors , 2007, Nucleic acids research.
[24] A. Avogaro,et al. Circulating Progenitor Cells Are Reduced in Patients with Severe Lung Disease , 2006, Stem cells.
[25] S. Raisuddin,et al. Effects of Paraquat on the Freshwater Fish Channa punctata (Bloch): Non-Enzymatic Antioxidants as Biomarkers of Exposure , 2006, Archives of environmental contamination and toxicology.
[26] V. Ambros. The functions of animal microRNAs , 2004, Nature.
[27] S. Dimmeler,et al. Endothelial Progenitor Cells: Characterization and Role in Vascular Biology , 2004, Circulation research.
[28] K. Orito,et al. Chymase is activated in the pulmonary inflammation and fibrosis induced by paraquat in hamsters. , 2004, The Tohoku journal of experimental medicine.
[29] Z. Suntres. Role of antioxidants in paraquat toxicity. , 2002, Toxicology.
[30] Thomas D. Schmittgen,et al. Analysis of relative gene expression data using real-time quantitative PCR and the 2(-Delta Delta C(T)) Method. , 2001, Methods.
[31] J. Matsumoto,et al. Further studies of lipid peroxidation in human paraquat poisoning. , 1986, Archives of internal medicine.