The ketogenic diet: metabolic influences on brain excitability and epilepsy
暂无分享,去创建一个
[1] I. Nissim,et al. Brain amino acid metabolism and ketosis , 2001, Journal of neuroscience research.
[2] R. Paul,et al. Vascular smooth muscle: aerobic glycolysis linked to sodium and potassium transport processes. , 1979, Science.
[3] P. Bregestovski,et al. Lactate Effectively Covers Energy Demands during Neuronal Network Activity in Neonatal Hippocampal Slices , 2011, Front. Neuroenerg..
[4] N. Danial,et al. BAD-Dependent Regulation of Fuel Metabolism and KATP Channel Activity Confers Resistance to Epileptic Seizures , 2012, Neuron.
[5] S. Cunnane,et al. Lipid and fatty acid profiles in rats consuming different high-fat ketogenic diets , 2001, Lipids.
[6] F. Proverbio,et al. Membrane compartmentalized ATP and its preferential use by the Na, K-ATPase of human red cell ghosts , 1977, The Journal of general physiology.
[7] J. Rho,et al. Ketones inhibit mitochondrial production of reactive oxygen species production following glutamate excitotoxicity by increasing NADH oxidation , 2007, Neuroscience.
[8] W. Burnham,et al. Dietary Fat, Ketosis, and Seizure Resistance in Rats on the Ketogenic Diet , 2000, Epilepsia.
[9] D. Richter,et al. Dynamic activation of KATP channels in rhythmically active neurons , 2001, The Journal of physiology.
[10] F. Ashcroft,et al. Overlapping distribution of KATP channel‐forming Kir6.2 subunit and the sulfonylurea receptor SUR1 in rodent brain , 1997, FEBS letters.
[11] Robert Brenner,et al. BK channel β4 subunit reduces dentate gyrus excitability and protects against temporal lobe seizures , 2005, Nature Neuroscience.
[12] D. Devivo,et al. Chronic ketosis and cerebral metabolism , 1978 .
[13] A. Konnerth,et al. Cell-type specific expression of ATP-sensitive potassium channels in the rat hippocampus. , 1999, The Journal of physiology.
[14] Rita Zemankovics,et al. Maintaining network activity in submerged hippocampal slices: importance of oxygen supply , 2009, The European journal of neuroscience.
[15] Thomas Rülicke,et al. Astrogliosis in epilepsy leads to overexpression of adenosine kinase, resulting in seizure aggravation. , 2005, Brain : a journal of neurology.
[16] Gary Yellen,et al. Ketone bodies, glycolysis, and KATP channels in the mechanism of the ketogenic diet , 2008, Epilepsia.
[17] P. Huttenlocher. Ketonemia and Seizures: Metabolic and Anticonvulsant Effects of Two Ketogenic Diets in Childhood Epilepsy , 1976, Pediatric Research.
[18] S. Masino,et al. Metabolic Autocrine Regulation of Neurons Involves Cooperation among Pannexin Hemichannels, Adenosine Receptors, and KATP Channels , 2010, The Journal of Neuroscience.
[19] Avital Schurr,et al. Lactate: The Ultimate Cerebral Oxidative Energy Substrate? , 2006, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[20] L. Raeymaekers,et al. Preferential support of Ca2+ uptake in smooth muscle plasma membrane vesicles by an endogenous glycolytic cascade , 1989, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[21] J. Phillis,et al. The role of adenosine and its nucleotides in central synaptic transmission , 1981, Progress in Neurobiology.
[22] S. Jarrett,et al. The ketogenic diet increases mitochondrial glutathione levels , 2008, Journal of neurochemistry.
[23] E. Thiele,et al. Low-glycemic-index treatment: A liberalized ketogenic diet for treatment of intractable epilepsy , 2005, Neurology.
[24] D. Richter,et al. Intracellular signalling pathways modulate KATP channels in inspiratory brainstem neurones and their hypoxic activation: involvement of metabotropic receptors, G-proteins and cytoskeleton , 2000, Brain Research.
[25] K. Borges,et al. Anticonvulsant profile of a balanced ketogenic diet in acute mouse seizure models , 2008, Epilepsy Research.
[26] J. Rho,et al. Ketone bodies are protective against oxidative stress in neocortical neurons , 2007, Journal of neurochemistry.
[27] D. Attwell,et al. Updated Energy Budgets for Neural Computation in the Neocortex and Cerebellum , 2012, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[28] E. Thiele,et al. Efficacy, safety, and tolerability of the low glycemic index treatment in pediatric epilepsy , 2009, Epilepsia.
[29] J. B. Wolfe,et al. Localization of the primary metabolic block produced by 2-deoxyglucose. , 1957, The Journal of biological chemistry.
[30] Qingchun Tong,et al. Glucose stimulation of hypothalamic MCH neurons involves K(ATP) channels, is modulated by UCP2, and regulates peripheral glucose homeostasis. , 2010, Cell metabolism.
[31] J. Hoffman,et al. On the functional use of the membrane compartmentalized pool of ATP by the Na+ and Ca++ pumps in human red blood cell ghosts , 2009, The Journal of general physiology.
[32] N. Danial. BAD: undertaker by night, candyman by day , 2008, Oncogene.
[33] J. McNamara,et al. Conditional Deletion of TrkB but Not BDNF Prevents Epileptogenesis in the Kindling Model , 2004, Neuron.
[34] David Hsu,et al. The dentate gyrus as a filter or gate: a look back and a look ahead. , 2007, Progress in brain research.
[35] N. Secher,et al. Blood Lactate is an Important Energy Source for the Human Brain , 2009, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[36] G F Cahill,et al. Brain metabolism during fasting. , 1967, The Journal of clinical investigation.
[37] E. Kossoff,et al. Efficacy of the Atkins diet as therapy for intractable epilepsy , 2003, Neurology.
[38] I. Nissim,et al. The ketogenic diet and brain metabolism of amino acids: relationship to the anticonvulsant effect. , 2007, Annual review of nutrition.
[39] M. G. Peterman,et al. Ketogenic Diet for Epilepsy , 2008 .
[40] Garnette R Sutherland,et al. The human brain utilizes lactate via the tricarboxylic acid cycle: a 13C-labelled microdialysis and high-resolution nuclear magnetic resonance study. , 2009, Brain : a journal of neurology.
[41] K. Bough,et al. An anticonvulsant profile of the ketogenic diet in the rat , 2002, Epilepsy Research.
[42] M. Cortez,et al. Anticonvulsant properties of acetone, a brain ketone elevated by the ketogenic diet , 2003, Annals of neurology.
[43] A. N. Wick,et al. The effect of 2-deoxyglucose on the metabolism of glucose, fructose, and galactose by rat diaphragm. , 1956, The Journal of biological chemistry.
[44] J. LaManna,et al. Contribution of brain glucose and ketone bodies to oxidative metabolism. , 2013, Advances in experimental medicine and biology.
[45] R. Llinás,et al. Calcium clearance and its energy requirements in cerebellar neurons. , 2010, Cell calcium.
[46] J. L. Stringer,et al. Possible mechanisms for the anticonvulsant activity of fructose‐1,6‐diphosphate , 2008, Epilepsia.
[47] P. Carlen,et al. Chronic in vitro ketosis is neuroprotective but not anti‐convulsant , 2010, Journal of neurochemistry.
[48] Rm. Wilder,et al. The effects of ketonemia on the course of epilepsy , 1921 .
[49] M. Haymond,et al. Effects of ketosis on glucose flux in children and adults. , 1983, The American journal of physiology.
[50] D. Green,et al. The BCL-2 family reunion. , 2010, Molecular cell.
[51] Hong Yang,et al. Glucose transporter type I deficiency syndrome: Epilepsy phenotypes and outcomes , 2012, Epilepsia.
[52] K. Petersen,et al. The Contribution of Blood Lactate to Brain Energy Metabolism in Humans Measured by Dynamic 13C Nuclear Magnetic Resonance Spectroscopy , 2010, The Journal of Neuroscience.
[53] G. S. Wilson,et al. A Temporary Local Energy Pool Coupled to Neuronal Activity: Fluctuations of Extracellular Lactate Levels in Rat Brain Monitored with Rapid‐Response Enzyme‐Based Sensor , 1997, Journal of neurochemistry.
[54] G. Yellen,et al. Ketogenic Diet Metabolites Reduce Firing in Central Neurons by Opening KATP Channels , 2007, The Journal of Neuroscience.
[55] P. Chan,et al. Energy metabolism in hypoxic astrocytes: Protective mechanism of fructose-1,6-bisphosphate , 1995, Neurochemical Research.
[56] S. Masino,et al. Are purines mediators of the anticonvulsant/neuroprotective effects of ketogenic diets? , 2008, Trends in Neurosciences.
[57] B. Fredholm,et al. A ketogenic diet suppresses seizures in mice through adenosine A₁ receptors. , 2011, The Journal of clinical investigation.
[58] N. Ruderman,et al. Regulation of glucose and ketone-body metabolism in brain of anaesthetized rats. , 1974, The Biochemical journal.
[59] M. Guzmán,et al. Is there an astrocyte–neuron ketone body shuttle? , 2001, Trends in Endocrinology & Metabolism.
[60] P. Kwan,et al. Early identification of refractory epilepsy. , 2000, The New England journal of medicine.
[61] E. Thiele,et al. The use of diet in the treatment of epilepsy , 2005, Epilepsy & Behavior.
[62] J. Rho,et al. Ketones prevent synaptic dysfunction induced by mitochondrial respiratory complex inhibitors , 2010, Journal of neurochemistry.
[63] H. M. Keith. FACTORS INFLUENCING EXPERIMENTALLY PRODUCED CONVULSIONS , 1933 .
[64] D. Coulter,et al. Chronic Epileptogenic Cellular Alterations in the Limbic System After Status Epilepticus , 1999, Epilepsia.
[65] Antoine Depaulis,et al. Endogenous control of epilepsy: The nigral inhibitory system , 1994, Progress in Neurobiology.
[66] M. Avoli,et al. Jasper's basic mechanisms of the epilepsies , 2012 .
[67] De-Pei Li,et al. Adenosine inhibits paraventricular pre‐sympathetic neurons through ATP‐dependent potassium channels , 2010, Journal of neurochemistry.
[68] Frances M. Ashcroft,et al. Correlating structure and function in ATP-sensitive K+ channels , 1998, Trends in Neurosciences.
[69] E. Kossoff,et al. A Prospective Study of the Modified Atkins Diet for Intractable Epilepsy in Adults , 2008, Epilepsia.
[70] P. Magistretti,et al. Activity‐dependent regulation of energy metabolism by astrocytes: An update , 2007, Glia.
[71] S. Schultz,et al. Colocalization of glycolytic enzyme activity and KATP channels in basolateral membrane of Necturusenterocytes. , 1998, American journal of physiology. Cell physiology.
[72] G. Yellen,et al. Single KATP Channel Opening in Response to Action Potential Firing in Mouse Dentate Granule Neurons , 2011, The Journal of Neuroscience.
[73] N. Rawson,et al. Distribution and phenotype of neurons containing the ATP-sensitive K+ channel in rat brain , 1998, Brain Research.
[74] K. A. Yamada,et al. Ketone bodies do not directly alter excitatory or inhibitory hippocampal synaptic transmission , 2000, Neurology.
[75] S. Laughlin,et al. An Energy Budget for Signaling in the Grey Matter of the Brain , 2001, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[76] S. Schultz,et al. Colocalization of glycolytic enzyme activity and KATP channels in basolateral membrane of Necturus enterocytes. , 1998, The American journal of physiology.
[77] Kelvin A. Yamada,et al. The ketogenic diet inhibits the mammalian target of rapamycin (mTOR) pathway , 2011, Epilepsia.
[78] Mathew Tantama,et al. Optogenetic reporters: Fluorescent protein-based genetically encoded indicators of signaling and metabolism in the brain. , 2012, Progress in brain research.
[79] B. Lowell,et al. Glucose sensing by POMC neurons regulates glucose homeostasis and is impaired in obesity , 2007, Nature.
[80] P. Schwartzkroin,et al. Calorie Restriction and Ketogenic Diet Diminish Neuronal Excitability in Rat Dentate Gyrus In Vivo , 2003, Epilepsia.
[81] E. Kossoff,et al. A Modified Atkins Diet Is Effective for the Treatment of Intractable Pediatric Epilepsy , 2006, Epilepsia.
[82] H. Scharfman,et al. Temporal lobe epilepsy and the BDNF receptor, TrkB , 2010 .
[83] Avtar Roopra,et al. 2-Deoxy-D-glucose reduces epilepsy progression by NRSF-CtBP–dependent metabolic regulation of chromatin structure , 2006, Nature Neuroscience.
[84] J. Cross,et al. A randomized trial of classical and medium‐chain triglyceride ketogenic diets in the treatment of childhood epilepsy , 2009, Epilepsia.
[85] H. Beck,et al. The dentate gyrus as a regulated gate for the propagation of epileptiform activity. , 1992, Epilepsy research. Supplement.
[86] K. Bough,et al. Higher ketogenic diet ratios confer protection from seizures without neurotoxicity , 1999, Epilepsy Research.
[87] D. Gilbert,et al. The Ketogenic Diet: Seizure Control Correlates Better With Serum β-Hydroxybutyrate Than With Urine Ketones , 2000, Journal of child neurology.
[88] K. Bough,et al. Path Analysis Shows That Increasing Ketogenic Ratio, but Not β-Hydroxybutyrate, Elevates Seizure Threshold in the Rat , 1999, Developmental Neuroscience.
[89] R. Nicoll,et al. Metabolic Control of Vesicular Glutamate Transport and Release , 2010, Neuron.
[90] J. Rho,et al. The ketogenic diet increases mitochondrial uncoupling protein levels and activity , 2004, Annals of neurology.
[91] M. Lu,et al. Interaction between Aldolase and Vacuolar H+-ATPase , 2001, The Journal of Biological Chemistry.
[92] Z. Xiong,et al. Neuron‐restrictive silencer factor is not required for the antiepileptic effect of the ketogenic diet , 2011, Epilepsia.
[93] K. Hussain,et al. Pancreatic β-cell KATP channels: Hypoglycaemia and hyperglycaemia , 2010, Reviews in Endocrine and Metabolic Disorders.
[94] J. Ferrendelli,et al. CHRONIC KETOSIS AND CEREBRAL METABOLISM , 1977, Pediatric Research.
[95] P. Dunham,et al. Membrane-bound ATP fuels the Na/K pump. Studies on membrane-bound glycolytic enzymes on inside-out vesicles from human red cell membranes , 1981, The Journal of general physiology.
[96] J. Rho,et al. Acetoacetate, Acetone, and Dibenzylamine (a Contaminant in l‐(+)‐β‐Hydroxybutyrate) Exhibit Direct Anticonvulsant Actions in Vivo , 2002, Epilepsia.
[97] J. Fischer,et al. Linkage of aerobic glycolysis to sodium-potassium transport in rat skeletal muscle. Implications for increased muscle lactate production in sepsis. , 1996, The Journal of clinical investigation.
[98] J. L. Stringer,et al. Fructose-1,6-Bisphosphate Has Anticonvulsant Activity in Models of Acute Seizures in Adult Rats , 2007, The Journal of Neuroscience.
[99] M. Majoie,et al. Blood beta-hydroxybutyrate correlates better with seizure reduction due to ketogenic diet than do ketones in the urine , 2010, Seizure.