Comparison of myeloid neoplasms with nonclassic 3q26.2/MECOM versus classic inv(3)/t(3;3) rearrangements reveals diverse clinicopathologic features, genetic profiles, and molecular mechanisms of MECOM activation
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O. Frankfurt | J. Altman | M. Kocherginsky | Xinyan Lu | P. Ji | L. Jennings | M. Sukhanova | F. Wehbe | S. Gurbuxani | Juehua Gao | Yi-Hua Chen | Q. Chen | Taylor Zak