Proto-oncogenes/ transcription factors

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[16]  C. Marshall,et al.  Activation of MAP kinase kinase is necessary and sufficient for PC12 differentiation and for transformation of NIH 3T3 cells , 1994, Cell.

[17]  J. Woodgett,et al.  The stress-activated protein kinase subfamily of c-Jun kinases , 1994, Nature.

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[19]  Donald M Miller,et al.  The G‐C Specific DNA Binding Drug, Mithramycin, Selectively Inhibits Transcription of the C‐MYC and C‐HA-RAS Genes in Regenerating Liver , 1994, The American journal of the medical sciences.

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[21]  P. Godowski,et al.  In vivo response of hepatocytes to growth factors requires an initial priming stimulus , 1994, Hepatology.

[22]  P. Dent,et al.  Inhibition of the EGF-activated MAP kinase signaling pathway by adenosine 3',5'-monophosphate. , 1993, Science.

[23]  S. Cook,et al.  Inhibition by cAMP of Ras-dependent activation of Raf. , 1993, Science.

[24]  M. Karin,et al.  Identification of an oncoprotein- and UV-responsive protein kinase that binds and potentiates the c-Jun activation domain. , 1993, Genes & development.

[25]  A. Baldwin,et al.  Distinct mechanisms for regulation of the interleukin-8 gene involve synergism and cooperativity between C/EBP and NF-kappa B , 1993, Molecular and cellular biology.

[26]  W. Greene,et al.  Cross‐coupling of the NF‐kappa B p65 and Fos/Jun transcription factors produces potentiated biological function. , 1993, The EMBO journal.

[27]  E. Wagner,et al.  c-Jun is essential for normal mouse development and hepatogenesis , 1993, Nature.

[28]  C. Barlow,et al.  Differential patterns of expression of three C/EBP isoforms, HNF-1, and HNF-4 after partial hepatectomy in rats. , 1993, Experimental cell research.

[29]  A. Baldwin,et al.  Functional and physical associations between NF-kappa B and C/EBP family members: a Rel domain-bZIP interaction. , 1993, Molecular and cellular biology.

[30]  A. Baldwin,et al.  Tumor necrosis factor and interleukin-1 lead to phosphorylation and loss of I kappa B alpha: a mechanism for NF-kappa B activation , 1993, Molecular and cellular biology.

[31]  Richard Treisman,et al.  Functional analysis of a growth factor-responsive transcription factor complex , 1993, Cell.

[32]  R. Treisman,et al.  The SRF accessory protein Elk-1 contains a growth factor-regulated transcriptional activation domain , 1993, Cell.

[33]  R Taub,et al.  Interactions among LRF-1, JunB, c-Jun, and c-Fos define a regulatory program in the G1 phase of liver regeneration , 1992, Molecular and cellular biology.

[34]  C. McClain,et al.  Antibodies to tumor necrosis factor-alpha inhibit liver regeneration after partial hepatectomy. , 1992, The American journal of physiology.

[35]  P. Dobrzanski,et al.  Rapid induction in regenerating liver of RL/IF-1 (an I kappa B that inhibits NF-kappa B, RelB-p50, and c-Rel-p50) and PHF, a novel kappa B site-binding complex , 1992, Molecular and cellular biology.

[36]  D. Mischoulon,et al.  Growth-dependent inhibition of CCAAT enhancer-binding protein (C/EBP alpha) gene expression during hepatocyte proliferation in the regenerating liver and in culture , 1992, Molecular and cellular biology.

[37]  M. Karin,et al.  The role of Jun, Fos and the AP-1 complex in cell-proliferation and transformation. , 1991, Biochimica et biophysica acta.

[38]  J. Azizkhan,et al.  Induction of NF-kappa B DNA-binding activity during the G0-to-G1 transition in mouse fibroblasts , 1991, Molecular and cellular biology.

[39]  R. Bravo,et al.  The jun and fos protein families are both required for cell cycle progression in fibroblasts , 1991, Molecular and cellular biology.

[40]  S. Haskill,et al.  Characterization of an immediate-early gene induced in adherent monocytes that encodes IκB-like activity , 1991, Cell.

[41]  C. Tanaka,et al.  Expression of the CRE-BP1 transcriptional regulator binding to the cyclic AMP response element in central nervous system, regenerating liver, and human tumors. , 1991, Oncogene.

[42]  Y. Yamaguchi-Iwai,et al.  The nuclear protooncogenes c-jun and c-fos as regulators of DNA replication. , 1991, Proceedings of the National Academy of Sciences of the United States of America.

[43]  Tsonwin Hai,et al.  Cross-family dimerization of transcription factors Fos/Jun and ATF/CREB alters DNA binding specificity. , 1991, Proceedings of the National Academy of Sciences of the United States of America.

[44]  J. Hsu,et al.  Identification of LRF-1, a leucine-zipper protein that is rapidly and highly induced in regenerating liver. , 1991, Proceedings of the National Academy of Sciences of the United States of America.

[45]  D. Soprano,et al.  A potential role for c-jun in cell cycle progression through late G1 and S. , 1991, Oncogene.

[46]  J. Hsu,et al.  The immediate-early growth response in regenerating liver and insulin-stimulated H-35 cells: comparison with serum-stimulated 3T3 cells and identification of 41 novel immediate-early genes , 1991, Molecular and cellular biology.

[47]  R. Taub,et al.  Immediate-early gene expression differs between regenerating liver, insulin-stimulated H-35 cells, and mitogen-stimulated Balb/c 3T3 cells. Liver-specific induction patterns of gene 33, phosphoenolpyruvate carboxykinase, and the jun, fos, and egr families. , 1990, The Journal of biological chemistry.

[48]  C. Babinet,et al.  c-myc, c-fos, and c-jun regulation in the regenerating livers of normal and H-2K/c-myc transgenic mice , 1990, Molecular and cellular biology.

[49]  D. Brenner,et al.  Transient induction of C‐jun during hepatic regeneration , 1990, Hepatology.

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[51]  S. McKnight,et al.  The DNA binding domain of the rat liver nuclear protein C/EBP is bipartite. , 1989, Science.

[52]  D. Nathans,et al.  jun-D: a third member of the jun gene family. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[53]  Peter Angel,et al.  The jun proto-oncogene is positively autoregulated by its product, Jun/AP-1 , 1988, Cell.

[54]  P. Fort,et al.  Regulation of c-fos gene expression in hamster fibroblasts: initiation and elongation of transcription and mRNA degradation. , 1987, Nucleic acids research.

[55]  I. Verma,et al.  Proto-oncogene expression in regenerating liver is simulated in cultures of primary adult rat hepatocytes. , 1986, The Journal of biological chemistry.

[56]  N. Thompson,et al.  Sequential protooncogene expression during rat liver regeneration. , 1986, Cancer research.

[57]  C. Guguen-Guillouzo,et al.  Expression of c-fos oncogene during hepatocarcinogenesis, liver regeneration and in synchronized HTC cells. , 1985, Experimental cell research.

[58]  T. Sugimura,et al.  C-myc transcript is induced in rat liver at a very early stage of regeneration or by cycloheximide treatment , 1984, Nature.

[59]  C. Petropoulos,et al.  Expression of a cellular oncogene during liver regeneration. , 1983, Science.

[60]  H. Leffert,et al.  Hormonal control of rat liver regeneration. , 1979, Gastroenterology.

[61]  B. Errede,et al.  The proliferation of MAP kinase signaling pathways in yeast. , 1995, Current opinion in cell biology.

[62]  R. Baserga,et al.  Cell cycle and growth control. , 1993, Critical reviews in eukaryotic gene expression.

[63]  M. Karin.  The AP-1 complex and its role in transcriptional control by protein kinase C , 1991 .

[64]  H. Herschman.  Primary response genes induced by growth factors and tumor promoters. , 1991, Annual review of biochemistry.

[65]  P. Vogt,et al.  Interaction of cellular factors related to the Jun oncoprotein with the promoter of a replication-dependent hamster histone H3.2 gene. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

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