Enhanced nephrotoxicity of acetaminophen in fructose-induced hypertriglyceridemic rats: contribution of oxidation and deacetylation of acetaminophen to an enhancement of nephrotoxicity.
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[1] K. Doi,et al. Enhanced nephrotoxicity of acetaminophen in fructose-induced hypertriglyceridemic rats: effect of partial hepatectomy. , 1997, Experimental and toxicologic pathology : official journal of the Gesellschaft fur Toxikologische Pathologie.
[2] K. Doi,et al. Effects of fructose-induced hypertriglyceridemia on hepatorenal toxicity of acetaminophen in rats: role of pharmacokinetics and metabolism of acetaminophen. , 1997, Experimental and toxicologic pathology : official journal of the Gesellschaft fur Toxikologische Pathologie.
[3] J. Tarloff,et al. Contribution of oxidation and deacetylation to the bioactivation of acetaminophen in vitro in liver and kidney from male and female Sprague-Dawley rats. , 1995, Drug metabolism and disposition: the biological fate of chemicals.
[4] A. Mclean,et al. Prevention of paracetamol-induced liver injury by fructose. , 1991, Biochemical pharmacology.
[5] J. Newton,et al. The role of p-aminophenol in acetaminophen-induced nephrotoxicity: effect of bis(p-nitrophenyl) phosphate on acetaminophen and p-aminophenol nephrotoxicity and metabolism in Fischer 344 rats. , 1985, Toxicology and applied pharmacology.
[6] I. Chaudry,et al. Accelerated functional recovery of isolated rat kidney with ATP-MgCl2 after warm ischemia. , 1984, The American journal of physiology.
[7] R. Schrier,et al. Mitochondrial calcium accumulation and respiration in ischemic acute renal failure in the rat. , 1984, Kidney international.
[8] J. Hook,et al. Potentiation of acute chloroform nephrotoxicity by the glutathione depletor diethyl maleate and protection by the microsomal enzyme inhibitor piperonyl butoxide. , 1981, Toxicology and applied pharmacology.
[9] J. Mitchell,et al. Renal necrosis, glutathione depletion, and covalent binding after acetaminophen. , 1978, Toxicology and applied pharmacology.
[10] N. Whittaker,et al. Phosgene: a metabolite of chloroform. , 1977, Biochemical and biophysical research communications.
[11] B B Brodie,et al. Acetaminophen-induced hepatic necrosis. IV. Protective role of glutathione. , 1973, The Journal of pharmacology and experimental therapeutics.
[12] T. Boyer,et al. Acetaminophen-induced hepatic necrosis and renal failure. , 1971, JAMA.
[13] O. H. Lowry,et al. Effect of fructose, dihydroxyacetone, glycerol, and glucose on metabolites and related compounds in liver and kidney. , 1970, The Journal of biological chemistry.
[14] K. Doi,et al. Effects of fructose-induced hypertriglyceridemia on hepatorenal toxicity of acetaminophen in rats. , 1995, Experimental and toxicologic pathology : official journal of the Gesellschaft fur Toxikologische Pathologie.
[15] Ernest Hodgson,et al. Introduction to Biochemical Toxicology , 1994 .
[16] S. Emeighhart. Evidence against deacetylation and for cytochrome P450-mediated activation in acetaminophen-induced nephrotoxicity in the CD-1 mouse. , 1991 .
[17] E. Jeffery. 5 – Biochemical Basis of Toxicity , 1991 .
[18] J. Hook,et al. Acetaminophen and p-aminophenol nephrotoxicity in aging male Sprague-Dawley and Fischer 344 rats. , 1989, Fundamental and applied toxicology : official journal of the Society of Toxicology.
[19] R. Schrier,et al. Effect of adenosine triphosphate depletion in vivo on renal function in the rat with and without ischemia. , 1989, Mineral and electrolyte metabolism.
[20] R. Schrier,et al. Effects of adenosine triphosphate depletion in the isolated perfused rat kidney. , 1987, Mineral and electrolyte metabolism.
[21] D. Pessayre,et al. In situ formation of the acetaminophen metabolite covalently bound in kidney and lung. Supportive evidence provided by total hepatectomy. , 1982, Biochemical pharmacology.