Gene dose-dependent control of hematopoiesis and hematologic tumor suppression by CBP.
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D. Livingston | R. Bronson | A. Kung | T. Yao | L. Ch'ng | C. Sieff | D M Livingston | C. Sieff | R T Bronson | V. I. Rebel | A L Kung | V I Rebel | L E Ch'ng | C A Sieff | T P Yao
[1] H. Masuya,et al. Abnormal skeletal patterning in embryos lacking a single Cbp allele: a partial similarity with Rubinstein-Taybi syndrome. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[2] T. Iwama,et al. p300 gene alterations in colorectal and gastric carcinomas. , 1996, Oncogene.
[3] S. Lazo-Kallanian,et al. Essential Role for the P55 Tumor Necrosis Factor Receptor in Regulating Hematopoiesis at a Stem Cell Level , 1999, The Journal of experimental medicine.
[4] R. W. Miller,et al. Tumors in Rubinstein-Taybi syndrome. , 1995, American journal of medical genetics.
[5] Raoul C. M. Hennekam,et al. Rubinstein-Taybi syndrome caused by mutations in the transcriptional co-activator CBP , 1995, Nature.
[6] L. M. Facchini,et al. The molecular role of Myc in growth and transformation: recent discoveries lead to new insights , 1998, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[7] Andrew J. Bannister,et al. The CBP co-activator is a histone acetyltransferase , 1996, Nature.
[8] T. Nabeshima,et al. Truncated CBP protein leads to classical Rubinstein-Taybi syndrome phenotypes in mice: implications for a dominant-negative mechanism. , 1999, Human molecular genetics.
[9] R. Eckner. p300 and CBP as transcriptional regulators and targets of oncogenic events. , 1996, Biological chemistry.
[10] M. Breuning,et al. Conjunction dysfunction: CBP/p300 in human disease. , 1998, Trends in genetics : TIG.
[11] D. Housman,et al. MLL is fused to CBP, a histone acetyltransferase, in therapy-related acute myeloid leukemia with a t(11;16)(q23;p13.3). , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[12] Jeffrey D. Parvin,et al. RNA Helicase A Mediates Association of CBP with RNA Polymerase II , 1997, Cell.
[13] Eric Nyberg,et al. Independent transformation activity by adenovirus-5 E1A-conserved regions 1 or 2 mutants. , 1991, Virology.
[14] D. Livingston,et al. The nuclear hormone receptor coactivator SRC-1 is a specific target of p300. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[15] M. Potter. Experimental plasmacytomagenesis in mice. , 1997, Hematology/oncology clinics of North America.
[16] S. Bacchetti,et al. Definition of adenovirus type 5 functions involved in the induction of chromosomal aberrations in human cells. , 1990, The Journal of general virology.
[17] Johnson M Liu,et al. Chromatin remodeling and leukemia: new therapeutic paradigms. , 1999, Blood.
[18] J. Ward,et al. The Morphology, Immunohistochemistry, and Incidence of Hematopoietic Neoplasms in Mice and Rats , 1993, Toxicologic pathology.
[19] S. Orkin,et al. CREB-binding protein cooperates with transcription factor GATA-1 and is required for erythroid differentiation. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[20] M. Ewen,et al. The adenovirus E1A-associated 300-kD protein exhibits properties of a transcriptional coactivator and belongs to an evolutionarily conserved family. , 1994, Cold Spring Harbor symposia on quantitative biology.
[21] B. Howard,et al. A p300/CBP-associated factor that competes with the adenoviral oncoprotein E1A , 1996, Nature.
[22] C. Disteche,et al. The translocation t(8;16)(p11;p13) of acute myeloid leukaemia fuses a putative acetyltransferase to the CREB–binding protein , 1996, Nature Genetics.
[23] Wei Gu,et al. Activation of p53 Sequence-Specific DNA Binding by Acetylation of the p53 C-Terminal Domain , 1997, Cell.
[24] Mariann Bienz,et al. Drosophila CBP represses the transcription factor TCF to antagonize Wingless signalling , 1998, Nature.
[25] L. Schnipper,et al. Genetic analysis of adenovirus E1A: induction of genetic instability and altered cell morphologic and growth characteristics are segregatable functions. , 1998, Mutation research.
[26] T. Suda,et al. Mice homozygous for a truncated form of CREB-binding protein exhibit defects in hematopoiesis and vasculo-angiogenesis. , 1999, Blood.
[27] B. Howard,et al. The Transcriptional Coactivators p300 and CBP Are Histone Acetyltransferases , 1996, Cell.
[28] James M. Roberts,et al. The murine gene p27Kip1 is haplo-insufficient for tumour suppression , 1998, Nature.
[29] N. Shiama. The p300/CBP family: integrating signals with transcription factors and chromatin. , 1997, Trends in cell biology.
[30] A. Sparks,et al. Identification of c-MYC as a target of the APC pathway. , 1998, Science.
[31] E. Harlow,et al. Antibodies: A Laboratory Manual , 1988 .
[32] David Newsome,et al. Gene Dosage–Dependent Embryonic Development and Proliferation Defects in Mice Lacking the Transcriptional Integrator p300 , 1998, Cell.
[33] E. Harlow,et al. Cellular targets for transformation by the adenovirus E1A proteins , 1989, Cell.