Anisomycin-activated protein kinases p45 and p55 but not mitogen-activated protein kinases ERK-1 and -2 are implicated in the induction of c-fos and c-jun
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[1] J. Sambrook,et al. Molecular Cloning: A Laboratory Manual , 2001 .
[2] M J Barratt,et al. Mitogen-stimulated phosphorylation of histone H3 is targeted to a small hyperacetylation-sensitive fraction. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[3] J. Woodgett,et al. The stress-activated protein kinase subfamily of c-Jun kinases , 1994, Nature.
[4] G. Johnson,et al. Sequential protein kinase reactions controlling cell growth and differentiation. , 1994, Current opinion in cell biology.
[5] M. Karin,et al. JNK1: A protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain , 1994, Cell.
[6] R. Treisman. Ternary complex factors: growth factor regulated transcriptional activators. , 1994, Current opinion in genetics & development.
[7] C. Marshall,et al. MAP kinase kinase kinase, MAP kinase kinase and MAP kinase. , 1994, Current opinion in genetics & development.
[8] L. Mahadevan,et al. Anisomycin and rapamycin define an area upstream of p70/85S6k containing a bifurcation to histone H3-HMG-like protein phosphorylation and c-fos-c-jun induction , 1994 .
[9] Hong Sun,et al. MKP-1 (3CH134), an immediate early gene product, is a dual specificity phosphatase that dephosphorylates MAP kinase in vivo , 1993, Cell.
[10] M. Karin,et al. Identification of an oncoprotein- and UV-responsive protein kinase that binds and potentiates the c-Jun activation domain. , 1993, Genes & development.
[11] A. Brunet,et al. Growth factors induce nuclear translocation of MAP kinases (p42mapk and p44mapk) but not of their activator MAP kinase kinase (p45mapkk) in fibroblasts , 1993, The Journal of cell biology.
[12] G. Pfeifer,et al. In vivo protein-DNA interactions at the c-jun promoter: preformed complexes mediate the UV response , 1993, Molecular and cellular biology.
[13] J. Blenis,et al. Signal transduction via the MAP kinases: proceed at your own RSK. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[14] R. Hipskind,et al. c‐fos transcriptional activation and repression correlate temporally with the phosphorylation status of TCF. , 1993, The EMBO journal.
[15] R. Treisman,et al. The SRF accessory protein Elk-1 contains a growth factor-regulated transcriptional activation domain , 1993, Cell.
[16] E. Nishida,et al. The MAP kinase cascade is essential for diverse signal transduction pathways. , 1993, Trends in biochemical sciences.
[17] S. Winawer,et al. Signal transduction through extracellular signal-regulated kinase-like pp57 blocked in differentiated cells having low protein kinase C beta activity. , 1993, The Journal of biological chemistry.
[18] E. Nishida,et al. cDNA cloning of MAP kinase kinase reveals kinase cascade pathways in yeasts to vertebrates. , 1993, The EMBO journal.
[19] J. Woodgett,et al. Co-purification of mitogen-activated protein kinases with phorbol ester-induced c-Jun kinase activity in U937 leukaemic cells. , 1993, Oncogene.
[20] R. Davis,et al. Mitogen-activated protein kinase stimulation by a tyrosine kinase-negative epidermal growth factor receptor. , 1993, The Journal of biological chemistry.
[21] S. Keyse,et al. Oxidative stress and heat shock induce a human gene encoding a protein-tyrosine phosphatase , 1992, Nature.
[22] D. Meek,et al. Nuclear protein phosphorylation and growth control. , 1992, The Biochemical journal.
[23] S. Ferrari,et al. Activation of p70s6k is associated with phosphorylation of four clustered sites displaying Ser/Thr-Pro motifs. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[24] A. Sharrocks,et al. Phosphorylation of transcription factor p62TCF by MAP kinase stimulates ternary complex formation at c-fos promoter , 1992, Nature.
[25] R. Campos-González,et al. Tyrosine phosphorylation of mitogen-activated protein kinase in cells with tyrosine kinase-negative epidermal growth factor receptors. , 1992, The Journal of biological chemistry.
[26] D. Edwards,et al. Protein synthesis inhibitors differentially superinduce c‐fos and c‐jun by three distinct mechanisms: lack of evidence for labile repressors. , 1992, The EMBO journal.
[27] G. Crabtree,et al. Rapamycin-FKBP specifically blocks growth-dependent activation of and signaling by the 70 kd S6 protein kinases , 1992, Cell.
[28] S. Pelech,et al. Mitogen-activated protein kinases: versatile transducers for cell signaling. , 1992, Trends in biochemical sciences.
[29] L. Heasley,et al. MAP kinase is constitutively activated in gip2 and src transformed rat 1a fibroblasts. , 1992, The Journal of biological chemistry.
[30] J. Blenis,et al. Nuclear localization and regulation of erk- and rsk-encoded protein kinases , 1992, Molecular and cellular biology.
[31] J. Avruch,et al. An array of insulin-activated, proline-directed serine/threonine protein kinases phosphorylate the p70 S6 kinase. , 1992, The Journal of biological chemistry.
[32] G. Thomas. MAP kinase by any other name smells just as sweet , 1992, Cell.
[33] James R. Woodgett,et al. Phosphorylation of c-jun mediated by MAP kinases , 1991, Nature.
[34] H. Kawasaki,et al. Xenopus M phase MAP kinase: isolation of its cDNA and activation by MPF. , 1991, The EMBO journal.
[35] J. Avruch,et al. pp54 microtubule-associated protein-2 kinase requires both tyrosine and serine/threonine phosphorylation for activity. , 1991, The Journal of biological chemistry.
[36] J. Blenis,et al. Distinct mechanisms for the activation of the RSK kinases/MAP2 kinase/pp90rsk and pp70-S6 kinase signaling systems are indicated by inhibition of protein synthesis. , 1991, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.
[37] Anthony C. Willis,et al. Rapid histone H3 phosphorylation in response to growth factors, phorbol esters, okadaic acid, and protein synthesis inhibitors , 1991, Cell.
[38] Nancy Y. Ip,et al. ERKs: A family of protein-serine/threonine kinases that are activated and tyrosine phosphorylated in response to insulin and NGF , 1991, Cell.
[39] D. Burns,et al. Lipid activation of protein kinase C. , 1991, The Journal of biological chemistry.
[40] L. Mahadevan,et al. Signalling and superinduction , 1991, Nature.
[41] L. Cantley,et al. Oncogenes and signal transduction , 1991, Cell.
[42] P. Majerus,et al. Recent insights in phosphatidylinositol signaling , 1990, Cell.
[43] J. Ferrell,et al. Identification of a 42-kilodalton phosphotyrosyl protein as a serine(threonine) protein kinase by renaturation , 1990, Molecular and cellular biology.
[44] A E Nel,et al. Stimulation of MAP-2 kinase activity in T lymphocytes by anti-CD3 or anti-Ti monoclonal antibody is partially dependent on protein kinase C. , 1990, Journal of immunology.
[45] M. Rosenfeld,et al. Hormonally inducible phosphorylation of a nuclear pool of ribosomal protein S6. , 1990, The Journal of biological chemistry.
[46] A. Saltiel,et al. Nerve growth factor stimulates a protein kinase in PC-12 cells that phosphorylates microtubule-associated protein-2. , 1990, The Journal of biological chemistry.
[47] P. Cohen,et al. Okadaic acid: a new probe for the study of cellular regulation. , 1990, Trends in biochemical sciences.
[48] M. Sheng,et al. The inner core of the serum response element mediates both the rapid induction and subsequent repression of c-fos transcription following serum stimulation. , 1990, Genes & development.
[49] F. C. Lucibello,et al. Trans-repression of the mouse c-fos promoter: A novel mechanism of fos-mediated trans-regulation , 1989, Cell.
[50] M. Siegmann,et al. A stimulated S6 kinase from rat liver: identity with the mitogen activated S6 kinase of 3T3 cells. , 1989, The EMBO journal.
[51] I. Kameshita,et al. A sensitive method for detection of calmodulin-dependent protein kinase II activity in sodium dodecyl sulfate-polyacrylamide gel. , 1989, Analytical biochemistry.
[52] Richard Treisman,et al. Removal of poly(A) and consequent degradation of c-fos mRNA facilitated by 3′ AU-rich sequences , 1988, Nature.
[53] M. Yaniv,et al. Transcriptional activation of c-jun during the G0/G1 transition in mouse fibroblasts , 1988, Nature.
[54] R. Bravo,et al. Complexity of the early genetic response to growth factors in mouse fibroblasts , 1988, Molecular and cellular biology.
[55] E. Nishida,et al. Activation of a Ca2+-inhibitable protein kinase that phosphorylates microtubule-associated protein 2 in vitro by growth factors, phorbol esters, and serum in quiescent cultured human fibroblasts. , 1988, The Journal of biological chemistry.
[56] I. Verma,et al. Modification of fos proteins: phosphorylation of c-fos, but not v-fos, is stimulated by 12-tetradecanoyl-phorbol-13-acetate and serum , 1987, Molecular and cellular biology.
[57] P. Chomczyński,et al. Single-step method of RNA isolation by acid guanidinium thiocyanate-phenol-chloroform extraction. , 1987, Analytical biochemistry.
[58] M. Greenberg,et al. Effect of protein synthesis inhibitors on growth factor activation of c-fos, c-myc, and actin gene transcription , 1986, Molecular and cellular biology.
[59] T. Curran,et al. FBJ murine osteosarcoma virus: identification and molecular cloning of biologically active proviral DNA , 1982, Journal of virology.
[60] A. Riggs,et al. Genomic Sequencing , 2010 .
[61] A. Nebreda,et al. Inactivation of MAP kinases. , 1994, Trends in biochemical sciences.
[62] E. Racker. Use of synthetic amino acid polymers for assay of protein-tyrosine and protein-serine kinases. , 1991, Methods in enzymology.
[63] H. Herschman. Primary response genes induced by growth factors and tumor promoters. , 1991, Annual review of biochemistry.
[64] D. Marshak,et al. Synthetic peptide substrates for casein kinase II. , 1991, Methods in enzymology.