Half the Genetic Variance in Vitamin D Concentration is Shared with Skin Colour and Sun Exposure Genes

[1]  Y. Rhee,et al.  Familial clustering of vitamin D deficiency via shared environment: The Korean National Health and Nutrition Examination Survey 2008–2012 , 2018, European Journal of Clinical Nutrition.

[2]  P. O’Reilly,et al.  Genome-wide association study in 79,366 European-ancestry individuals informs the genetic architecture of 25-hydroxyvitamin D levels , 2018, Nature Communications.

[3]  J. Prins,et al.  Association of Sun Exposure, Skin Colour and Body Mass Index with Vitamin D Status in Individuals Who Are Morbidly Obese , 2017, Nutrients.

[4]  T. Spector,et al.  Genome-Wide Association Shows that Pigmentation Genes Play a Role in Skin Aging. , 2017, The Journal of investigative dermatology.

[5]  M. Ramírez-Zea,et al.  Correlates and family aggregation of vitamin D concentrations in school-aged children and their parents in nine Mesoamerican countries , 2017, Public Health Nutrition.

[6]  H. Wulf,et al.  The half-life of 25(OH)D after UVB exposure depends on gender and vitamin D receptor polymorphism but mainly on the start level , 2017, Photochemical & photobiological sciences : Official journal of the European Photochemistry Association and the European Society for Photobiology.

[7]  H. Campbell,et al.  The impact of vitamin D pathway genetic variation and circulating 25-hydroxyvitamin D on cancer outcome: systematic review and meta-analysis , 2017, British Journal of Cancer.

[8]  M. C. Savanelli,et al.  Low vitamin D status and obesity: Role of nutritionist , 2017, Reviews in Endocrine and Metabolic Disorders.

[9]  B. Altieri,et al.  Shedding new light on female fertility: The role of vitamin D , 2017, Reviews in Endocrine and Metabolic Disorders.

[10]  A. Young,et al.  The impact of skin colour on human photobiological responses , 2016, Pigment cell & melanoma research.

[11]  N. Wray,et al.  Heritability of Transforming Growth Factor-β1 and Tumor Necrosis Factor-Receptor Type 1 Expression and Vitamin D Levels in Healthy Adolescent Twins , 2014, Twin Research and Human Genetics.

[12]  K. H. Madsen,et al.  Vitamin D status and its determinants in children and adults among families in late summer in Denmark. , 2014, The British journal of nutrition.

[13]  R. Lucas,et al.  The contributions of solar ultraviolet radiation exposure and other determinants to serum 25-hydroxyvitamin D concentrations in Australian adults: the AusD Study. , 2014, American journal of epidemiology.

[14]  J. McGrath,et al.  Vitamin D and health in pregnancy, infants, children and adolescents in Australia and New Zealand: a position statement , 2013, The Medical journal of Australia.

[15]  J. McGrath,et al.  Vitamin D, effects on brain development, adult brain function and the links between low levels of vitamin D and neuropsychiatric disease , 2013, Frontiers in Neuroendocrinology.

[16]  A. Webb,et al.  Recommended summer sunlight exposure amounts fail to produce sufficient vitamin D status in UK adults of South Asian origin. , 2011, The American journal of clinical nutrition.

[17]  V. Vaccarino,et al.  Heritability and seasonal variability of vitamin D concentrations in male twins. , 2010, The American journal of clinical nutrition.

[18]  JoAnn E. Manson,et al.  The 2011 Report on Dietary Reference Intakes for Calcium and Vitamin D from the Institute of Medicine: What Clinicians Need to Know , 2010, The Journal of clinical endocrinology and metabolism.

[19]  D. Eyles.  Vitamin D and autism: does skin colour modify risk? , 2010, Acta paediatrica.

[20]  A. Wolk,et al.  Seasonal Genetic Influence on Serum 25-Hydroxyvitamin D Levels: A Twin Study , 2009, PloS one.

[21]  Xiping Xu,et al.  Heritability and environmental factors affecting vitamin D status in rural Chinese adolescent twins. , 2009, The Journal of clinical endocrinology and metabolism.

[22]  P. Mortensen,et al.  A sensitive LC/MS/MS assay of 25OH vitamin D3 and 25OH vitamin D2 in dried blood spots. , 2009, Clinica chimica acta; international journal of clinical chemistry.

[23]  R. D'Agostino,et al.  Genetic and non-genetic correlates of vitamins K and D , 2009, European Journal of Clinical Nutrition.

[24]  J. Norris,et al.  Genetic and environmental determinants of 25-hydroxyvitamin D and 1,25-dihydroxyvitamin D levels in Hispanic and African Americans. , 2008, The Journal of clinical endocrinology and metabolism.

[25]  A. Morris,et al.  Evidence for genetic regulation of vitamin D status in twins with multiple sclerosis , 2008, The American journal of clinical nutrition.

[26]  L. Bouter,et al.  Fatty fish and supplements are the greatest modifiable contributors to the serum 25‐hydroxyvitamin D concentration in a multiethnic population , 2008, Clinical endocrinology.

[27]  A. Wolk,et al.  Associations of diet, supplement use, and ultraviolet B radiation exposure with vitamin D status in Swedish women during winter. , 2007, The American journal of clinical nutrition.

[28]  M. Holick.  Vitamin D deficiency. , 2007, The New England journal of medicine.

[29]  M. Wjst,et al.  A genome-wide linkage scan for 25-OH-D3 and 1,25-(OH)2-D3 serum levels in asthma families , 2007, The Journal of Steroid Biochemistry and Molecular Biology.

[30]  Ann R Webb,et al.  Who, what, where and when-influences on cutaneous vitamin D synthesis. , 2006, Progress in biophysics and molecular biology.

[31]  J. Rockell,et al.  Serum 25-hydroxyvitamin D concentrations of New Zealanders aged 15 years and older , 2006, Osteoporosis International.

[32]  B. Hollis.  Circulating 25-hydroxyvitamin D levels indicative of vitamin D sufficiency: implications for establishing a new effective dietary intake recommendation for vitamin D. , 2005, The Journal of nutrition.

[33]  J. McGrath,et al.  Distribution of the Vitamin D receptor and 1α-hydroxylase in human brain , 2005, Journal of Chemical Neuroanatomy.

[34]  J. Rees,et al.  The genetics of sun sensitivity in humans. , 2004, American journal of human genetics.

[35]  Margaret J. Wright,et al.  Brisbane Adolescent Twin Study: Outline of study methods and research projects , 2004 .

[36]  Wei Chen,et al.  Interactive effects of MC1R and OCA2 on melanoma risk phenotypes. , 2003, Human molecular genetics.

[37]  N. Martin,et al.  Zygosity Diagnosis in the Absence of Genotypic Data: An Approach Using Latent Class Analysis , 2003, Twin Research.

[38]  B. Dawson-Hughes,et al.  Serum 25-hydroxyvitamin D status of adolescents and adults in two seasonal subpopulations from NHANES III. , 2002, Bone.

[39]  I. Jackson,et al.  Functional variation of MC1R alleles from red-haired individuals. , 2001, Human molecular genetics.

[40]  T. Spector,et al.  Genetic Contribution to Bone Metabolism, Calcium Excretion, and Vitamin D and Parathyroid Hormone Regulation , 2001, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

[41]  D. Battistutta,et al.  Incidence and determinants of skin cancer in a high‐risk australian population , 1990, International journal of cancer.

[42]  N. Martin,et al.  The inheritance of scholastic abilities in a sample of twins I. Ascertainment of the sample and diagnosis of zygosity , 1975, Annals of human genetics.

[43]  C. Annweiler,et al.  Vitamin D and chronic diseases: the current state of the art , 2016, Archives of Toxicology.

[44]  J. Trent,et al.  A genome-wide scan for naevus count: linkage to CDKN2A and to other chromosome regions , 2007, European Journal of Human Genetics.

[45]  Victor Siskind,et al.  Sun exposure and interaction with family history in risk of melanoma, Queensland, Australia , 2002, International journal of cancer.