Neuroprotection by Caffeine and A2A Adenosine Receptor Inactivation in a Model of Parkinson's Disease
暂无分享,去创建一个
M. Schwarzschild | P. Sonsalla | J. P. Petzer | K. Castagnoli | N. Castagnoli | M A Schwarzschild | K Castagnoli | N Castagnoli | J F Chen | K Xu | J P Petzer | R Staal | Y H Xu | M Beilstein | P K Sonsalla | R. Staal | Kui Xu | Yuehang Xu | J. Chen | Mark Beilstein | J. Petzer
[1] E. Ongini,et al. Adenosine A2A Receptors and Neuroprotection , 1997, Annals of the New York Academy of Sciences.
[2] M. Fernstrom,et al. Effects of Caffeine on Monoamine Neurotransmitters in the Central and Peripheral Nervous System , 1984 .
[3] K. Jacobson,et al. Activation of the A2A adenosine receptor inhibits nitric oxide production in glial cells , 1998, FEBS letters.
[4] Phillis Jw,et al. Adenosine in the control of the cerebral circulation. , 1989 .
[5] E. Ongini,et al. Blockade of adenosine A2A receptors by SCH 58261 results in neuroprotective effects in cerebral ischaemia in rats , 1998, Neuroreport.
[6] L. Grégoire,et al. Antiparkinsonian effect of a new selective adenosine A2A receptor antagonist in MPTP-treated monkeys , 1999, Neurology.
[7] B. Fredholm,et al. Pharmacology of adenosine A2A receptors. , 1996, Trends in pharmacological sciences.
[8] P. Svenningsson,et al. Distribution, biochemistry and function of striatal adenosine A2A receptors , 1999, Progress in Neurobiology.
[9] S. Schiffmann,et al. Aggressiveness, hypoalgesia and high blood pressure in mice lacking the adenosine A2a receptor , 1997, Nature.
[10] D. Deleu. Smoking, alcohol, and coffee consumption preceding Parkinson’s disease , 2001, Neurology.
[11] B. Fredholm,et al. Actions of caffeine in the brain with special reference to factors that contribute to its widespread use. , 1999, Pharmacological reviews.
[12] P. Jenner,et al. Adenosine A2A Antagonist: A novel antiparkinsonian agent that does not provoke dyskinesia in Parkinsonian monkeys , 1998, Annals of neurology.
[13] J. Phillis. Adenosine in the control of the cerebral circulation. , 1989, Cerebrovascular and brain metabolism reviews.
[14] R. Mayfield,et al. Opposing actions of adenosine A2a and dopamine D2 receptor activation on GABA release in the basal ganglia: Evidence for an A2a/D2 receptor interaction in globus pallidus , 1996, Synapse.
[15] O. Steward,et al. Genetic determinants of susceptibility to excitotoxic cell death: implications for gene targeting approaches. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[16] A. Benabid,et al. Subthalamic Nucleus Lesion in Rats Prevents Dopaminergic Nigral Neuron Degeneration After Striatal 6‐OHDA Injection: Behavioural and Immunohistochemical Studies , 1996, The European journal of neuroscience.
[17] C. Tanner,et al. Association of coffee and caffeine intake with the risk of Parkinson disease. , 2000, JAMA.
[18] P. Jenner,et al. Adenosine A2A receptor antagonists as new agents for the treatment of Parkinson's disease. , 1997, Trends in pharmacological sciences.
[19] T. Stone,et al. Protection against hippocampal kainate excitotoxicity by intracerebral administration of an adenosine A2A receptor antagonist , 1998, Brain Research.
[20] W. Willett,et al. Prospective study of caffeine consumption and risk of Parkinson's disease in men and women , 2001, Annals of neurology.
[21] G. Siegel,et al. Neurotrophic factors in Alzheimer’s and Parkinson’s disease brain , 2000, Brain Research Reviews.
[22] M. Mouradian,et al. Protection Against Acute MPTP‐Induced Dopamine Depletion in Mice by Adenosine A1 Agonist , 1993, Journal of neurochemistry.
[23] P. Popoli,et al. Adenosine A2A receptor stimulation enhances striatal extracellular glutamate levels in rats. , 1995, European journal of pharmacology.
[24] J. Obeso,et al. Subthalamic nucleus‐mediated excitotoxicity in parkinson's disease: A target for neuroprotection , 1998, Annals of neurology.
[25] J. Ribeiro,et al. Adenosine A2 receptor-mediated excitatory actions on the nervous system , 1996, Progress in Neurobiology.
[26] P. Sonsalla,et al. Correlation between the neostriatal content of the 1-methyl-4-phenylpyridinium species and dopaminergic neurotoxicity following 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine administration to several strains of mice. , 1991, The Journal of pharmacology and experimental therapeutics.
[27] A. Kupsch,et al. Pretreatment with nimodipine prevents MPTP-induced neurotoxicity at the nigral, but not at the striatal level in mice. , 1995, Neuroreport.
[28] Protection against methamphetamine-induced neurotoxicity to neostriatal dopaminergic neurons by adenosine receptor activation. , 1994, The Journal of pharmacology and experimental therapeutics.
[29] Hiroshi Kase,et al. Adenosine A2A antagonists with potent anti-cataleptic activity , 1997 .
[30] H. Kase,et al. Photoisomerization of a potent and selective adenosine A2 antagonist, (E)-1,3-Dipropyl-8-(3,4-dimethoxystyryl)-7-methylxanthine. , 1993, Journal of medicinal chemistry.
[31] M. Moskowitz,et al. A2A Adenosine Receptor Deficiency Attenuates Brain Injury Induced by Transient Focal Ischemia in Mice , 1999, The Journal of Neuroscience.