Arsenite suppresses IL-2-dependent tumoricidal activities of natural killer cells
暂无分享,去创建一个
[1] Md Imam Hossain,et al. Association between chronic arsenic exposure and the characteristic features of asthma. , 2019, Chemosphere.
[2] D. Sumi,et al. Chronic exposure to submicromolar arsenite promotes the migration of human esophageal Het1A cells induced by heparin-binding EGF-like growth factor , 2019, Archives of Toxicology.
[3] C. Watzl,et al. Mechanisms of natural killer cell‐mediated cellular cytotoxicity , 2019, Journal of leukocyte biology.
[4] N. Bernard,et al. Expression of ligands for activating natural killer cell receptors on cell lines commonly used to assess natural killer cell function , 2019, BMC Immunology.
[5] Alok Srivastava,et al. Arsenic Trioxide Enhances the NK Cell Cytotoxicity Against Acute Promyelocytic Leukemia While Simultaneously Inhibiting Its Bio-Genesis , 2018, Front. Immunol..
[6] A. Sharma,et al. Arsenic exposure impels CD4 commitment in thymus and suppress T cell cytokine secretion by increasing regulatory T cells , 2017, Scientific Reports.
[7] V. Varbanova,et al. Killer-cell immunoglobulin-like receptor genes and ligands and their role in hematologic malignancies , 2016, Cancer Immunology, Immunotherapy.
[8] Lewis L. Lanier,et al. NK cells and cancer: you can teach innate cells new tricks , 2015, Nature Reviews Cancer.
[9] L. Lanier. NKG2D Receptor and Its Ligands in Host Defense , 2015, Cancer Immunology Research.
[10] Md Imam Hossain,et al. Associations of total arsenic in drinking water, hair and nails with serum vascular endothelial growth factor in arsenic-endemic individuals in Bangladesh. , 2015, Chemosphere.
[11] J. Baron,et al. Arsenic and Lung Disease Mortality in Bangladeshi Adults , 2014, Epidemiology.
[12] Abdullah Al Mamun,et al. Increases in oxidized low-density lipoprotein and other inflammatory and adhesion molecules with a concomitant decrease in high-density lipoprotein in the individuals exposed to arsenic in Bangladesh. , 2013, Toxicological sciences : an official journal of the Society of Toxicology.
[13] P. Haris,et al. Elevated levels of plasma Big endothelin-1 and its relation to hypertension and skin lesions in individuals exposed to arsenic. , 2012, Toxicology and applied pharmacology.
[14] C. Ferreccio,et al. Evidence from Chile that arsenic in drinking water may increase mortality from pulmonary tuberculosis. , 2011, American journal of epidemiology.
[15] M. Smyth,et al. Activating and inhibitory receptors of natural killer cells , 2011, Immunology and cell biology.
[16] F. Parvez,et al. A prospective study of the synergistic effects of arsenic exposure and smoking, sun exposure, fertilizer use, and pesticide use on risk of premalignant skin lesions in Bangladeshi men. , 2011, American journal of epidemiology.
[17] N. Banerjee,et al. Chronic Arsenic Exposure Impairs Macrophage Functions in the Exposed Individuals , 2009, Journal of Clinical Immunology.
[18] L. Vega,et al. Arsenic interferes with the signaling transduction pathway of T cell receptor activation by increasing basal and induced phosphorylation of Lck and Fyn in spleen cells. , 2008, Toxicology and applied pharmacology.
[19] A. Banerjee,et al. Analysis of T-cell proliferation and cytokine secretion in the individuals exposed to arsenic , 2008, Human & experimental toxicology.
[20] Eric Vivier,et al. Functions of natural killer cells , 2008, Nature Immunology.
[21] Y. Hsueh,et al. Arsenic exposure, urinary arsenic speciation, and the incidence of urothelial carcinoma: a twelve-year follow-up study , 2008, Cancer Causes & Control.
[22] D. Jewell,et al. Drinking-Water Arsenic Exposure Modulates Gene Expression in Human Lymphocytes from a U.S. Population , 2008, Environmental health perspectives.
[23] T. Wang,et al. Mechanisms of arsenic-induced cell transformation , 1989, Biological Trace Element Research.
[24] M. Cebrián,et al. Assessment of lymphocyte subpopulations and cytokine secretion in children exposed to arsenic , 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[25] V. Groh,et al. Immunobiology of human NKG2D and its ligands. , 2006, Current topics in microbiology and immunology.
[26] S. Negrini,et al. Tumor-Induced Apoptosis of Human IL-2-Activated NK Cells: Role of Natural Cytotoxicity Receptors1 , 2005, The Journal of Immunology.
[27] Xianglin Shi,et al. Molecular mechanisms of arsenic carcinogenesis , 2004, Molecular and Cellular Biochemistry.
[28] T. Inaoka,et al. Skin manifestations of arsenicosis in two villages in Bangladesh , 2002, International Journal of Dermatology.
[29] A. Diefenbach,et al. Strategies for target cell recognition by natural killer cells , 2001, Immunological reviews.
[30] C. Chen,et al. Incidence of transitional cell carcinoma and arsenic in drinking water: a follow-up study of 8,102 residents in an arseniasis-endemic area in northeastern Taiwan. , 2001, American journal of epidemiology.
[31] R. Biassoni,et al. Human NK-cell receptors. , 2000, Immunology today.
[32] D. Johnson. Differential expression of human major histocompatibility class I loci: HLA-A, -B, and -C. , 2000, Human immunology.
[33] R. Kastelein,et al. IL‐10 enhances NK cell proliferation, cytotoxicity and production of IFN‐γ when combined with IL‐18 , 1999, European journal of immunology.
[34] D. Ito,et al. Mice with a targeted mutation in lymphotoxin-alpha exhibit enhanced tumor growth and metastasis: impaired NK cell development and recruitment. , 1999, Journal of immunology.
[35] D. Chaplin,et al. Requirement for membrane lymphotoxin in natural killer cell development. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[36] C. Figdor,et al. Killer cell inhibitory receptors for MHC class I molecules regulate lysis of melanoma cells mediated by NK cells, gamma delta T cells, and antigen-specific CTL. , 1998, Journal of immunology.
[37] T. Ley,et al. Natural killer and lymphokine-activated killer cells require granzyme B for the rapid induction of apoptosis in susceptible target cells. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[38] M. Gonsebatt,et al. Lymphocyte replicating ability in individuals exposed to arsenic via drinking water. , 1994, Mutation research.
[39] A. Smith,et al. Vascular effects of chronic arsenic exposure: a review. , 1994, Epidemiologic reviews.
[40] G. Berke. The binding and lysis of target cells by cytotoxic lymphocytes: molecular and cellular aspects. , 1994, Annual review of immunology.
[41] M. Gonsebatt,et al. Lymphocyte proliferation kinetics and genotoxic findings in a pilot study on individuals chronically exposed to arsenic in Mexico. , 1991, Mutation research.
[42] F. Dautry,et al. Induction of tumor necrosis factor-alpha and -beta and interferon-gamma mRNA by interleukin 2 in murine lymphocytic cell lines. , 1988, Oncogene research.
[43] D. Weigent,et al. Interleukin 2 enhances natural killer cell activity through induction of gamma interferon , 1983, Infection and immunity.
[44] C. Henney,et al. Murine NK cell cultures: effects of interleukin-2 and interferon on cell growth and cytotoxic reactivity. , 1981, Journal of immunology.