IFN-β mediates coordinate expression of antigen-processing genes in RSV-infected pulmonary epithelial cells
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Allan R. Brasier | Mohammad Jamaluddin | S. Baron | R. Garofalo | M. Jamaluddin | A. Brasier | A. Casola | Shaofei Wang | Roberto P. Garofalo | Antonella Casola | Todd F. Elliott | Samuel Baron | T. Elliott | Shao-Kai Wang
[1] S. Beck,et al. Sequences encoded in the class II region of the MHC related to the 'ABC' superfamily of transporters , 1990, Nature.
[2] J. Monaco,et al. Transport protein genes in the murine MHC: possible implications for antigen processing. , 1990, Science.
[3] P. Cresswell,et al. Proteasome subunits encoded in the MHC are not generally required for the processing of peptides bound by MHC class I molecules , 1992, Nature.
[4] R. Garofalo,et al. The Major Component of IκBα Proteolysis Occurs Independently of the Proteasome Pathway in Respiratory Syncytial Virus-Infected Pulmonary Epithelial Cells , 1998, Journal of Virology.
[5] O. Ruuskanen,et al. Respiratory syncytial virus , 1993, Current Problems in Pediatrics.
[6] R. Garofalo,et al. Inducible translational regulation of the NF-IL6 transcription factor by respiratory syncytial virus infection in pulmonary epithelial cells , 1996, Journal of virology.
[7] Barry H. Smith,et al. A continuous tumor‐cell line from a human lung carcinoma with properties of type II alveolar epithelial cells , 1976, International journal of cancer.
[8] T. Noah,et al. Respiratory syncytial virus-induced cytokine production by a human bronchial epithelial cell line. , 1993, The American journal of physiology.
[9] W. König,et al. Interleukin-8, interleukin-6, and soluble tumour necrosis factor receptor type I release from a human pulmonary epithelial cell line (A549) exposed to respiratory syncytial virus. , 1994, Immunology.
[10] D. Wong,et al. The development of respiratory syncytial virus-specific IgE and the release of histamine in nasopharyngeal secretions after infection. , 1981, The New England journal of medicine.
[11] G. Hutchins,et al. Fatal respiratory syncytial virus infection in severe combined immunodeficiency syndrome. , 1985, American journal of diseases of children.
[12] A. Goldberg,et al. Inhibitors of the proteasome block the degradation of most cell proteins and the generation of peptides presented on MHC class I molecules , 1994, Cell.
[13] R. Zinkernagel,et al. Antiviral protection by vesicular stomatitis virus-specific antibodies in alpha/beta interferon receptor-deficient mice , 1995, Journal of virology.
[14] P. Ogra,et al. Mechanisms of Mucosal Immunopathology in Respiratory Syncytial Virus Infection , 1996 .
[15] R. Garofalo,et al. A promoter recruitment mechanism for tumor necrosis factor-alpha-induced interleukin-8 transcription in type II pulmonary epithelial cells. Dependence on nuclear abundance of Rel A, NF-kappaB1, and c-Rel transcription factors. , 1998, The Journal of biological chemistry.
[16] P. Cresswell,et al. Evidence that transporters associated with antigen processing translocate a major histocompatibility complex class I-binding peptide into the endoplasmic reticulum in an ATP-dependent manner. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[17] P. Ogra,et al. Cell-mediated immune response to respiratory syncytial virus infection: relationship to the development of reactive airway disease. , 1979, The Journal of pediatrics.
[18] B A Askonas,et al. Cytotoxic T cells clear virus but augment lung pathology in mice infected with respiratory syncytial virus , 1988, The Journal of experimental medicine.
[19] G. Widera,et al. Simplified northern blot hybridization using 5% sodium dodecyl sulfate. , 1990, BioTechniques.
[20] R. Chanock,et al. Respiratory syncytial virus disease in infants despite prior administration of antigenic inactivated vaccine. , 1969, American journal of epidemiology.
[21] H. Ploegh,et al. Generation, translocation, and presentation of MHC class I-restricted peptides. , 1995, Annual review of biochemistry.
[22] D. Finley,et al. MHC-linked LMP gene products specifically alter peptidase activities of the proteasome , 1993, Nature.
[23] J. Neefjes,et al. Selective and ATP-dependent translocation of peptides by the MHC-encoded transporter. , 1993, Science.
[24] S. Baron,et al. Interleukin-1 alpha mediates the enhanced expression of intercellular adhesion molecule-1 in pulmonary epithelial cells infected with respiratory syncytial virus. , 1995, American journal of respiratory cell and molecular biology.
[25] H. Rammensee,et al. Cell lines transfected with the TAP inhibitor ICP47 allow testing peptide binding to a variety of HLA class I molecules. , 1998, International immunology.
[26] J. Monaco,et al. MHC class II region encoding proteins related to the muKidrug resistance family of transmembrane transporters , 1990, Nature.
[27] S. Baron,et al. Respiratory syncytial virus infection of human respiratory epithelial cells up-regulates class I MHC expression through the induction of IFN-beta and IL-1 alpha. , 1996, Journal of immunology.
[28] A. Brasier,et al. Nuclear factor-kappaB-dependent induction of interleukin-8 gene expression by tumor necrosis factor alpha: evidence for an antioxidant sensitive activating pathway distinct from nuclear translocation. , 1999, Blood.
[29] D. Andrews,et al. Stable binding of the herpes simplex virus ICP47 protein to the peptide binding site of TAP. , 1996, The EMBO journal.
[30] R. Demars,et al. A gene in the human major histocompatibility complex class II region controlling the class I antigen presentation pathway , 1990, Nature.
[31] S. Tonegawa,et al. Altered peptidase and viral-specific T cell response in LMP2 mutant mice. , 1994, Immunity.
[32] S. Tonegawa,et al. TAP1 mutant mice are deficient in antigen presentation, surface class I molecules, and CD4−8+ T cells , 1992, Cell.
[33] Allan R. Brasier,et al. Tumor Necrosis Factor-α-inducible IκBα Proteolysis Mediated by Cytosolic m-Calpain , 1999, The Journal of Biological Chemistry.
[34] T. Elliott,et al. Transporter associated with antigen processing. , 1997, Advances in immunology.
[35] P. A. Peterson,et al. Molecular mechanism and species specificity of TAP inhibition by herpes simplex virus ICP47. , 1996, The EMBO journal.
[36] J. Trowsdale,et al. DNA sequence analysis of 66 kb of the human MHC class II region encoding a cluster of genes for antigen processing. , 1992, Journal of molecular biology.
[37] A. Goldberg,et al. Peptidase activities of proteasomes are differentially regulated by the major histocompatibility complex-encoded genes for LMP2 and LMP7. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[38] R F Standaert,et al. Inhibition of proteasome activities and subunit-specific amino-terminal threonine modification by lactacystin , 1995, Science.
[39] P. Cresswell,et al. Assembly and transport of class I MHC-peptide complexes. , 1994, Ciba Foundation symposium.
[40] S. Beck,et al. Coordinate regulation of the human TAP1 and LMP2 genes from a shared bidirectional promoter , 1995, The Journal of experimental medicine.
[41] D. Andrews,et al. A cytosolic herpes simplex virus protein inhibits antigen presentation to CD8+ T lymphocytes , 1994, Cell.
[42] R. Garofalo,et al. Transcriptional activation of the interleukin-8 gene by respiratory syncytial virus infection in alveolar epithelial cells: nuclear translocation of the RelA transcription factor as a mechanism producing airway mucosal inflammation , 1996, Journal of virology.
[43] K. Rajewsky,et al. MHC class I expression in mice lacking the proteasome subunit LMP-7. , 1994, Science.
[44] A. Goldberg,et al. Gamma-interferon and expression of MHC genes regulate peptide hydrolysis by proteasomes. , 1993, Nature.
[45] B. Graham,et al. Role of T lymphocyte subsets in the pathogenesis of primary infection and rechallenge with respiratory syncytial virus in mice. , 1991, The Journal of clinical investigation.
[46] J. Neefjes,et al. Proteasome subunits encoded by the major histocompatibility complex are not essential for antigen presentation , 1992, Nature.
[47] J. Sinnott,et al. Respiratory Syncytial Virus , 1988, Infection Control & Hospital Epidemiology.
[48] A. Goldberg,et al. Proteasome Subunits X and Y Alter Peptidase Activities in Opposite Ways to the Interferon-γ-induced Subunits LMP2 and LMP7* , 1996, The Journal of Biological Chemistry.
[49] W. Newell,et al. Evolutionary dynamics of non-coding sequences within the class II region of the human MHC. , 1996, Journal of molecular biology.
[50] J. Yewdell,et al. Herpes simplex virus turns off the TAP to evade host immunity , 1995, Nature.