Key role of the cyclin-dependent kinase inhibitor p27kip1 for embryonal carcinoma cell survival and differentiation

[1]  J. Sambrook,et al.  Molecular Cloning: A Laboratory Manual , 2001 .

[2]  A. Berns,et al.  A link between cell cycle and cell death: Bax and Bcl‐2 modulate Cdk2 activation during thymocyte apoptosis , 1998, The EMBO journal.

[3]  L. Meijer,et al.  The cyclin-dependent kinase inhibitors olomoucine and roscovitine arrest human fibroblasts in G1 phase by specific inhibition of CDK2 kinase activity. , 1998, Experimental cell research.

[4]  F. Di Cunto,et al.  Inhibitory function of p21Cip1/WAF1 in differentiation of primary mouse keratinocytes independent of cell cycle control. , 1998, Science.

[5]  James M. Roberts,et al.  Cleavage of p21Cip1/Waf1 and p27Kip1 mediates apoptosis in endothelial cells through activation of Cdk2: role of a caspase cascade. , 1998, Molecular cell.

[6]  M. Gorospe,et al.  p27Kip1 overexpression causes apoptotic death of mammalian cells , 1997, Oncogene.

[7]  L. Meijer,et al.  The effect of the cyclin-dependent kinase inhibitor olomoucine on cell cycle kinetics. , 1997, Experimental cell research.

[8]  H. Kiyokawa,et al.  Oligodendrocyte precursor differentiation is perturbed in the absence of the cyclin-dependent kinase inhibitor p27Kip1. , 1997, Genes & development.

[9]  Stefania Staibano,et al.  Expression of teratocarcinoma-derived growth factor-1 (TDGF-1) in testis germ cell tumors and its effects on growth and differentiation of embryonal carcinoma cell line NTERA2/D1 , 1997, Oncogene.

[10]  Kenneth Cowan,et al.  A recombinant adenovirus expressing p27Kip1 induces cell cycle arrest and loss of cyclin-Cdk activity in human breast cancer cells , 1997, Oncogene.

[11]  A. Strasser,et al.  The cell death inhibitor Bcl‐2 and its homologues influence control of cell cycle entry. , 1996, The EMBO journal.

[12]  F. Di Cunto,et al.  The absence of p21Cip1/WAF1 alters keratinocyte growth and differentiation and promotes ras-tumor progression. , 1996, Genes & development.

[13]  K. Walsh,et al.  Resistance to Apoptosis Conferred by Cdk Inhibitors During Myocyte Differentiation , 1996, Science.

[14]  R A Rifkind,et al.  Second generation hybrid polar compounds are potent inducers of transformed cell differentiation. , 1996, Proceedings of the National Academy of Sciences of the United States of America.

[15]  K. Manova-Todorova,et al.  Enhanced Growth of Mice Lacking the Cyclin-Dependent Kinase Inhibitor Function of p27 Kip1 , 1996, Cell.

[16]  James M. Roberts,et al.  Requirement of p27Kip1 for Restriction Point Control of the Fibroblast Cell Cycle , 1996, Science.

[17]  A. Ross,et al.  The cyclin-dependent kinase inhibitor p21 (WAF1) is required for survival of differentiating neuroblastoma cells , 1996, Molecular and cellular biology.

[18]  L. Freedman,et al.  Transcriptional activation of the Cdk inhibitor p21 by vitamin D3 leads to the induced differentiation of the myelomonocytic cell line U937. , 1996, Genes & development.

[19]  T. Akiyama,et al.  Constitutive Overexpression of CDK2 Inhibits Neuronal Differentiation of Rat Pheochromocytoma PC12 Cells (*) , 1995, The Journal of Biological Chemistry.

[20]  P. Beer-Romero,et al.  Role of the ubiquitin-proteasome pathway in regulating abundance of the cyclin-dependent kinase inhibitor p27. , 1995, Science.

[21]  K. Walsh,et al.  MyoD-induced expression of p21 inhibits cyclin-dependent kinase activity upon myocyte terminal differentiation , 1995, Molecular and cellular biology.

[22]  A. Zantema,et al.  Cyclin‐dependent kinases and pRb: regulators of the proliferation‐differentiation switch , 1995, FEBS letters.

[23]  R. Schlegel,et al.  Apoptosis and the cell cycle , 1995, Journal of cellular biochemistry.

[24]  James M. Roberts,et al.  Inhibitors of mammalian G1 cyclin-dependent kinases. , 1995, Genes & development.

[25]  R. Weinberg,et al.  The retinoblastoma protein and cell cycle control , 1995, Cell.

[26]  T. Hunter,et al.  Cyclins and cancer II: Cyclin D and CDK inhibitors come of age , 1994, Cell.

[27]  C. Sherr G1 phase progression: Cycling on cue , 1994, Cell.

[28]  P. Marks,et al.  Suppression of cyclin-dependent kinase 4 during induced differentiation of erythroleukemia cells , 1994, Molecular and cellular biology.

[29]  James M. Roberts,et al.  Cloning of p27 Kip1 , a cyclin-dependent kinase inhibitor and a potential mediator of extracellular antimitogenic signals , 1994, Cell.

[30]  J. Roberts,et al.  Inactivation of a Cdk2 inhibitor during interleukin 2-induced proliferation of human T lymphocytes , 1994, Molecular and cellular biology.

[31]  M. Chalfie,et al.  Green fluorescent protein as a marker for gene expression. , 1994, Science.

[32]  S. van den Heuvel,et al.  Distinct roles for cyclin-dependent kinases in cell cycle control. , 1993, Science.

[33]  J. Trent,et al.  WAF1, a potential mediator of p53 tumor suppression , 1993, Cell.

[34]  J. Michaeli,et al.  Hexamethylene bisacetamide in myelodysplastic syndrome and acute myelogenous leukemia: a phase II clinical trial with a differentiation-inducing agent. , 1992, Blood.

[35]  S. Korsmeyer,et al.  bcl-2 inhibits multiple forms of apoptosis but not negative selection in thymocytes , 1991, Cell.

[36]  P. Andrews,et al.  Different patterns of glycolipid antigens are expressed following differentiation of TERA-2 human embryonal carcinoma cells induced by retinoic acid, hexamethylene bisacetamide (HMBA) or bromodeoxyuridine (BUdR). , 1990, Differentiation; research in biological diversity.

[37]  P. Marks,et al.  Induction of transformed cells to terminal differentiation and the modulation of gene expression. , 1987, Cancer research.

[38]  P. Andrews Retinoic acid induces neuronal differentiation of a cloned human embryonal carcinoma cell line in vitro. , 1984, Developmental biology.

[39]  D. Solter,et al.  Monoclonal antibody to murine embryos defines a stage-specific embryonic antigen expressed on mouse embryos and human teratocarcinoma cells , 1982, Cell.

[40]  E. White,et al.  Life, death, and the pursuit of apoptosis. , 1996, Genes & development.