A novel function for cadherin 11/osteoblast-cadherin in vascular smooth muscle cells: modulation of cell migration and proliferation.

[1]  K. Martin,et al.  Endothelial cell activation of the smooth muscle cell phosphoinositide 3-kinase/Akt pathway promotes differentiation. , 2005, Journal of vascular surgery.

[2]  P. Altevogt,et al.  L1, a novel target of β-catenin signaling, transforms cells and is expressed at the invasive front of colon cancers , 2005, The Journal of cell biology.

[3]  D. Berry,et al.  Changes in the Wnt signalling pathway in gastrointestinal cancers and their prognostic significance. , 2005, European journal of cancer.

[4]  Lisheng Zhang,et al.  Vein Graft Neointimal Hyperplasia Is Exacerbated by Tumor Necrosis Factor Receptor-1 Signaling in Graft-Intrinsic Cells , 2004, Arteriosclerosis, thrombosis, and vascular biology.

[5]  Yumiko Saga,et al.  Cell‐Cell Interaction Mediated by Cadherin‐11 Directly Regulates the Differentiation of Mesenchymal Cells Into the Cells of the Osteo‐Lineage and the Chondro‐Lineage , 2004, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

[6]  P. Erne,et al.  T-cadherin upregulation correlates with cell-cycle progression and promotes proliferation of vascular cells. , 2004, Cardiovascular research.

[7]  W. Quist,et al.  Temporal gene expression following prosthetic arterial grafting. , 2004, The Journal of surgical research.

[8]  W. Quist,et al.  Temporal genomics of vein bypass grafting through oligonucleotide microarray analysis. , 2004, Journal of vascular surgery.

[9]  Jan P. Stegemann,et al.  Phenotype Modulation in Vascular Tissue Engineering Using Biochemical and Mechanical Stimulation , 2003, Annals of Biomedical Engineering.

[10]  Judy Lieberman,et al.  RNA interference targeting Fas protects mice from fulminant hepatitis , 2003, Nature Medicine.

[11]  O. Blaschuk,et al.  An alternatively spliced cadherin-11 enhances human breast cancer cell invasion. , 2002, Cancer research.

[12]  P. Erne,et al.  Expression of adhesion molecule T-cadherin is increased during neointima formation in experimental restenosis , 2002, Histochemistry and Cell Biology.

[13]  S A Bustin,et al.  Quantification of mRNA using real-time reverse transcription PCR (RT-PCR): trends and problems. , 2002, Journal of molecular endocrinology.

[14]  P. Erne,et al.  Expression of cell adhesion molecule T-cadherin in the human vasculature , 2001, Histochemistry and Cell Biology.

[15]  B. Angst,et al.  COMMENTARY The cadherin superfamily: diversity in form and function , 2022 .

[16]  F. Parhami,et al.  Tumor Necrosis Factor-&agr; Promotes In Vitro Calcification of Vascular Cells via the cAMP Pathway , 2000, Circulation.

[17]  L. Moldawer,et al.  Direct Evidence for Cytokine Involvement in Neointimal Hyperplasia , 2000, Circulation.

[18]  J. Schalken,et al.  Cadherin switching in human prostate cancer progression. , 2000, Cancer research.

[19]  H. Hutchinson,et al.  Pressure-mediated oligonucleotide transfection of rat and human cardiovascular tissues. , 1999, Proceedings of the National Academy of Sciences of the United States of America.

[20]  S. Byers,et al.  Cadherin-11 is expressed in invasive breast cancer cell lines. , 1999, Cancer research.

[21]  S. Korsmeyer,et al.  Caspase Cleaved BID Targets Mitochondria and Is Required for Cytochrome c Release, while BCL-XL Prevents This Release but Not Tumor Necrosis Factor-R1/Fas Death* , 1999, The Journal of Biological Chemistry.

[22]  H. Höfler,et al.  Cadherin‐11 is highly expressed in rhabdomyosarcomas and during differentiation of myoblasts in vitro , 1999, The Journal of pathology.

[23]  Andrew O. Brightman,et al.  Application and evaluation of the alamarblue assay for cell growth and survival of fibroblasts , 1998, In Vitro Cellular & Developmental Biology - Animal.

[24]  R. Ross,et al.  Atherosclerosis is an inflammatory disease. , 1998, American heart journal.

[25]  P. Libby,et al.  Interferon-gamma deficiency prevents coronary arteriosclerosis but not myocardial rejection in transplanted mouse hearts. , 1997, The Journal of clinical investigation.

[26]  L. Aiello,et al.  Vascular endothelial growth factor expression in canine peripheral vein bypass grafts. , 1997, Journal of vascular surgery.

[27]  S. Jovinge,et al.  Tumor necrosis factor-alpha activates smooth muscle cell migration in culture and is expressed in the balloon-injured rat aorta. , 1997, Arteriosclerosis, thrombosis, and vascular biology.

[28]  P. Faries,et al.  Immunolocalization and temporal distribution of cytokine expression during the development of vein graft intimal hyperplasia in an experimental model. , 1996, Journal of vascular surgery.

[29]  S M Schwartz,et al.  The intima. Soil for atherosclerosis and restenosis. , 1995, Circulation research.

[30]  R. Kikuno,et al.  Molecular cloning and characterization of OB-cadherin, a new member of cadherin family expressed in osteoblasts. , 1994, The Journal of biological chemistry.

[31]  W. Quist,et al.  Prevention of smooth muscle cell phenotypic modulation in vein grafts: a histomorphometric study. , 1992, Journal of vascular surgery.

[32]  M. Reidy,et al.  Proliferation of smooth muscle cells after vascular injury is inhibited by an antibody against basic fibroblast growth factor. , 1991, Proceedings of the National Academy of Sciences of the United States of America.

[33]  M. Davies,et al.  Pathophysiology of vein graft failure: a review. , 1995, European journal of vascular and endovascular surgery : the official journal of the European Society for Vascular Surgery.

[34]  R. Heimark,et al.  Cloning of five human cadherins clarifies characteristic features of cadherin extracellular domain and provides further evidence for two structurally different types of cadherin. , 1994, Cell adhesion and communication.