Regional differences in the morphological and functional effects of aging on cerebral basement membranes and perivascular drainage of amyloid‐β from the mouse brain
暂无分享,去创建一个
R. Carare | A. Dorr | R. Kalaria | R. Weller | H. Yuen | C. Hawkes | M. Gatherer | M. Sharp
[1] Marwan N. Sabbagh,et al. Morphological and Pathological Evolution of the Brain Microcirculation in Aging and Alzheimer’s Disease , 2012, PloS one.
[2] J. McLaurin,et al. Region-specific distribution of β-amyloid peptide and cytokine expression in TgCRND8 mice , 2011, Neuroscience Letters.
[3] K. Jellinger,et al. Review: Sporadic cerebral amyloid angiopathy , 2011, Neuropathology and applied neurobiology.
[4] Reinhard Schliebs,et al. Perivascular drainage of solutes is impaired in the ageing mouse brain and in the presence of cerebral amyloid angiopathy , 2011, Acta Neuropathologica.
[5] J. Morris,et al. Decreased Clearance of CNS β-Amyloid in Alzheimer’s Disease , 2010, Science.
[6] G. Dienel,et al. Trafficking of Glucose, Lactate, and Amyloid-β from the Inferior Colliculus through Perivascular Routes , 2010, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[7] V. Perry,et al. Degenerating synaptic boutons in prion disease: microglia activation without synaptic stripping. , 2009, The American journal of pathology.
[8] Berislav V. Zlokovic,et al. Neurovascular mechanisms and blood–brain barrier disorder in Alzheimer’s disease , 2009, Acta Neuropathologica.
[9] M. Ghazizadeh,et al. Interaction between β-amyloid protein and heparan sulfate proteoglycans from the cerebral capillary basement membrane in Alzheimer’s disease , 2009, Journal of Clinical Neuroscience.
[10] J. McLaurin,et al. Selective targeting of perivascular macrophages for clearance of β-amyloid in cerebral amyloid angiopathy , 2009, Proceedings of the National Academy of Sciences.
[11] P. Grammas,et al. Structural changes in Alzheimer's disease brain microvessels. , 2008, Current Alzheimer research.
[12] R O Weller,et al. Solutes, but not cells, drain from the brain parenchyma along basement membranes of capillaries and arteries: significance for cerebral amyloid angiopathy and neuroimmunology , 2008, Neuropathology and applied neurobiology.
[13] R. Carare,et al. Perivascular drainage of amyloid-beta peptides from the brain and its failure in cerebral amyloid angiopathy and Alzheimer's disease. , 2008, Brain pathology.
[14] S. Love,et al. Abeta-degrading enzymes in Alzheimer's disease. , 2008, Brain pathology.
[15] R. Carare,et al. SYMPOSIUM: Clearance of Aβ from the Brain in Alzheimer's Disease: Perivascular Drainage of Amyloid‐β Peptides from the Brain and Its Failure in Cerebral Amyloid Angiopathy and Alzheimer's Disease , 2007 .
[16] Phillip B. Jones,et al. Age-dependent cerebrovascular dysfunction in a transgenic mouse model of cerebral amyloid angiopathy. , 2007, Brain : a journal of neurology.
[17] M. Staufenbiel,et al. Occurrence and co-localization of amyloid β-protein and apolipoprotein E in perivascular drainage channels of wild-type and APP-transgenic mice , 2007, Neurobiology of Aging.
[18] John G. Sled,et al. Three-dimensional cerebral vasculature of the CBA mouse brain: A magnetic resonance imaging and micro computed tomography study , 2007, NeuroImage.
[19] I. Bechmann,et al. T cells traffic from brain to cervical lymph nodes via the cribroid plate and the nasal mucosa , 2006, Journal of leukocyte biology.
[20] V. Perry,et al. Mechanisms to explain the reverse perivascular transport of solutes out of the brain. , 2006, Journal of theoretical biology.
[21] S. DeKosky,et al. Simplified quantification of Pittsburgh Compound B amyloid imaging PET studies: a comparative analysis. , 2005, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.
[22] N. Patel,et al. Impaired angiogenesis in a transgenic mouse model of cerebral amyloidosis , 2004, Neuroscience Letters.
[23] R. Deane,et al. Early-onset and Robust Cerebral Microvascular Accumulation of Amyloid β-Protein in Transgenic Mice Expressing Low Levels of a Vasculotropic Dutch/Iowa Mutant Form of Amyloid β-Protein Precursor* , 2004, Journal of Biological Chemistry.
[24] Thomas Krucker,et al. Age-Dependent Cerebrovascular Abnormalities and Blood Flow Disturbances in APP23 Mice Modeling Alzheimer's Disease , 2003, The Journal of Neuroscience.
[25] B. Lamb,et al. Alterations in β-amyloid production and deposition in brain regions of two transgenic models , 2003, Neurobiology of Aging.
[26] J. Schmahmann. Vascular syndromes of the thalamus. , 2003, Stroke.
[27] G. Plant,et al. Cerebral Amyloid Angiopathies: A Pathologic, Biochemical, and Genetic View , 2003, Journal of neuropathology and experimental neurology.
[28] L. Launer,et al. Cerebral amyloid angiopathy and cognitive function: The HAAS autopsy study , 2002, Neurology.
[29] Y. Kiuchi,et al. Disassembly of amyloid β-protein fibril by basement membrane components , 2002 .
[30] R. Kalaria. Small Vessel Disease and Alzheimer’s Dementia: Pathological Considerations , 2002, Cerebrovascular Diseases.
[31] P. Mcgeer,et al. Relationship between beta amyloid peptide generating molecules and neprilysin in Alzheimer disease and normal brain , 2001, Brain Research.
[32] U. Dirnagl,et al. Immune surveillance of mouse brain perivascular spaces by blood‐borne macrophages , 2001, The European journal of neuroscience.
[33] M. Rafii,et al. Agrin and microvascular damage in Alzheimer's disease , 2000, Neurobiology of Aging.
[34] Judianne Davis,et al. Cerebrovascular Smooth Muscle Cell Surface Fibrillar Aβ: Alteration of the Proteolytic Environment in the Cerebral Vessel Wall , 2000, Annals of the New York Academy of Sciences.
[35] W. Halfter,et al. Agrin Binds to β-Amyloid (Aβ), Accelerates Aβ Fibril Formation, and Is Localized to Aβ Deposits in Alzheimer's Disease Brain , 2000, Molecular and Cellular Neuroscience.
[36] C. Masters,et al. Soluble pool of Aβ amyloid as a determinant of severity of neurodegeneration in Alzheimer's disease , 1999, Annals of neurology.
[37] T. Wight,et al. Perlecan Binds to the β‐Amyloid Proteins (Aβ) of Alzheimer's Disease, Accelerates Aβ Fibril Formation, and Maintains Aβ Fibril Stability , 1997 .
[38] B. Sommer,et al. Two amyloid precursor protein transgenic mouse models with Alzheimer disease-like pathology. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[39] R O Weller,et al. Perivascular spaces in the basal ganglia of the human brain: their relationship to lacunes , 1997, Journal of anatomy.
[40] H. Chui,et al. Vascular Basement Membrane Pathology and Alzheimer's Disease , 1997, Annals of the New York Academy of Sciences.
[41] R. Timpl. Macromolecular organization of basement membranes. , 1996, Current opinion in cell biology.
[42] R N Kalaria,et al. Apolipoprotein E-epsilon4 alleles in cerebral amyloid angiopathy and cerebrovascular pathology associated with Alzheimer's disease. , 1996, The American journal of pathology.
[43] C. Cotman,et al. Amyloid β‐Protein Induces Its Own Production in Cultured Degenerating Cerebrovascular Smooth Muscle Cells , 1995 .
[44] L. S. Perlmutter. Microvascular pathology and vascular basement membrane components in Alzheimer's disease , 1994, Molecular Neurobiology.
[45] H. Vinters,et al. Microvasculature in brain biopsy specimens from patients with Alzheimer's disease: an immunohistochemical and ultrastructural study. , 1994, Ultrastructural pathology.
[46] M. Yașargil,et al. Microsurgical anatomy of the hippocampal arteries. , 1993, Journal of neurosurgery.
[47] R O Weller,et al. Interrelationships of the pia mater and the perivascular (Virchow-Robin) spaces in the human cerebrum. , 1990, Journal of anatomy.
[48] H. Vinters,et al. Cerebral amyloid angiopathy: incidence and complications in the aging brain. II. The distribution of amyloid vascular changes. , 1983, Stroke.
[49] H F Cserr,et al. Bulk flow of interstitial fluid after intracranial injection of blue dextran 2000. , 1974, Experimental neurology.
[50] H. Vinters,et al. Effect of cerebral amyloid angiopathy on brain iron, copper, and zinc in Alzheimer's disease. , 2011, Journal of Alzheimer's disease : JAD.
[51] J. Wegiel,et al. Ultrastructural studies of the cells forming amyloid in the cortical vessel wall in Alzheimer's disease , 2004, Acta Neuropathologica.
[52] Y. Kiuchi,et al. Disassembly of amyloid beta-protein fibril by basement membrane components. , 2002, Life Science.
[53] L. Mucke,et al. Chronic overproduction of transforming growth factor-beta1 by astrocytes promotes Alzheimer's disease-like microvascular degeneration in transgenic mice. , 2000, The American journal of pathology.
[54] W. Halfter,et al. Agrin binds to beta-amyloid (Abeta), accelerates abeta fibril formation, and is localized to Abeta deposits in Alzheimer's disease brain. , 2000, Molecular and cellular neurosciences.
[55] N. Inestrosa,et al. Laminin blocks the assembly of wild-type A beta and the Dutch variant peptide into Alzheimer's fibrils. , 1998, Amyloid : the international journal of experimental and clinical investigation : the official journal of the International Society of Amyloidosis.
[56] T. Wight,et al. Perlecan binds to the beta-amyloid proteins (A beta) of Alzheimer's disease, accelerates A beta fibril formation, and maintains A beta fibril stability. , 1997, Journal of neurochemistry.
[57] C. Cotman,et al. Amyloid beta-protein induces its own production in cultured degenerating cerebrovascular smooth muscle cells. , 1995, Journal of neurochemistry.