ATM Functions at the Peroxisome to Induce Pexophagy in Response to ROS
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T. Pandita | M. Kastan | R. Dere | C. Walker | R. Pandita | T. Paull | D. Tripathi | Jinhe Kim | Ji-Hoon Lee | Reid T. Powell | Reid T Powell | Jiangwei Zhang | Ji Jing | V. Charaka | A. Alexander | J. Tait-Mulder | Angela Alexander | Ji‐Hoon Lee | Jacqueline Tait-Mulder
[1] N. Braverman,et al. Peroxisome biogenesis disorders , 2016, Translational science of rare diseases.
[2] T. Ludwig,et al. MOF phosphorylation by ATM regulates 53BP1-mediated double-strand break repair pathway choice. , 2014, Cell reports.
[3] A. Ernst,et al. Cargo recognition and trafficking in selective autophagy , 2014, Nature Cell Biology.
[4] T. Finkel,et al. Cellular mechanisms and physiological consequences of redox-dependent signalling , 2014, Nature Reviews Molecular Cell Biology.
[5] D. Green,et al. To Be or Not to Be? How Selective Autophagy and Cell Death Govern Cell Fate , 2014, Cell.
[6] S. Subramani,et al. Peroxisomal Atg37 binds Atg30 or palmitoyl-CoA to regulate phagophore formation during pexophagy , 2014, The Journal of cell biology.
[7] Andrew D. Rutenberg,et al. PEX5 and Ubiquitin Dynamics on Mammalian Peroxisome Membranes , 2014, PLoS Comput. Biol..
[8] Jennifer J. Smith,et al. Peroxisomes take shape , 2013, Nature Reviews Molecular Cell Biology.
[9] C. Brees,et al. PEX5, the Shuttling Import Receptor for Peroxisomal Matrix Proteins, Is a Redox‐Sensitive Protein , 2013, Traffic : the International Journal of Intracellular Transport.
[10] S. Akira,et al. Recruitment of the autophagic machinery to endosomes during infection is mediated by ubiquitin , 2013, The Journal of cell biology.
[11] D. Benjamin,et al. TSC on the peroxisome controls mTORC1 , 2013, Nature Cell Biology.
[12] Soumitra Polley,et al. Redox-regulated Cargo Binding and Release by the Peroxisomal Targeting Signal Receptor, Pex5* , 2013, The Journal of Biological Chemistry.
[13] C. Walker,et al. Reactive nitrogen species regulate autophagy through ATM-AMPK-TSC2–mediated suppression of mTORC1 , 2013, Proceedings of the National Academy of Sciences.
[14] G. Dorn,et al. PINK1-Phosphorylated Mitofusin 2 Is a Parkin Receptor for Culling Damaged Mitochondria , 2013, Science.
[15] J. Lippincott-Schwartz,et al. NBR1 acts as an autophagy receptor for peroxisomes , 2013, Journal of Cell Science.
[16] Nobutaka Hattori,et al. PINK1-mediated phosphorylation of the Parkin ubiquitin-like domain primes mitochondrial translocation of Parkin and regulates mitophagy , 2012, Scientific Reports.
[17] M. Overholtzer,et al. Interaction Between FIP200 and ATG16L1 Distinguishes ULK1 Complex-Dependent and -Independent Autophagy , 2012, Nature Structural &Molecular Biology.
[18] M. Fransen,et al. Role of peroxisomes in ROS/RNS-metabolism: implications for human disease. , 2012, Biochimica et biophysica acta.
[19] Robert Clarke,et al. Guidelines for the use and interpretation of assays for monitoring autophagy , 2012 .
[20] S. Subramani,et al. Pexophagy: The Selective Degradation of Peroxisomes , 2012, International journal of cell biology.
[21] D. Green,et al. Mitochondrial dysfunction in ataxia-telangiectasia. , 2012, Blood.
[22] Sebastian A. Wagner,et al. A Proteome-wide, Quantitative Survey of In Vivo Ubiquitylation Sites Reveals Widespread Regulatory Roles* , 2011, Molecular & Cellular Proteomics.
[23] S. Subramani,et al. Peroxisome assembly: matrix and membrane protein biogenesis , 2011, The Journal of cell biology.
[24] B. Viollet,et al. AMPK and mTOR regulate autophagy through direct phosphorylation of Ulk1 , 2011, Nature Cell Biology.
[25] M. Lavin,et al. ATM Activation by Oxidative Stress , 2010, Science.
[26] G. Mills,et al. ATM signals to TSC2 in the cytoplasm to regulate mTORC1 in response to ROS , 2010, Proceedings of the National Academy of Sciences.
[27] Fabienne C. Fiesel,et al. PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 , 2010, Nature Cell Biology.
[28] Harald W. Platta,et al. Pex2 and Pex12 Function as Protein-Ubiquitin Ligases in Peroxisomal Protein Import , 2009, Molecular and Cellular Biology.
[29] I. Singh,et al. Peroxisomal Dysfunction in Inflammatory Childhood White Matter Disorders: An Unexpected Contributor to Neuropathology , 2009, Journal of child neurology.
[30] Yongqiang Chen,et al. Superoxide is the major reactive oxygen species regulating autophagy , 2009, Cell Death and Differentiation.
[31] Ivan Dikic,et al. A role for ubiquitin in selective autophagy. , 2009, Molecular cell.
[32] Jennifer Lippincott-Schwartz,et al. Ubiquitin signals autophagic degradation of cytosolic proteins and peroxisomes , 2008, Proceedings of the National Academy of Sciences.
[33] Y. Fujiki,et al. The peroxin Pex14p is involved in LC3-dependent degradation of mammalian peroxisomes. , 2008, Experimental cell research.
[34] B. Warscheid,et al. Members of the E2D (UbcH5) Family Mediate the Ubiquitination of the Conserved Cysteine of Pex5p, the Peroxisomal Import Receptor* , 2008, Journal of Biological Chemistry.
[35] F. Gonzalez,et al. PPARalpha: mechanism of species differences and hepatocarcinogenesis of peroxisome proliferators. , 2008, Toxicology.
[36] T. Hunter. The age of crosstalk: phosphorylation, ubiquitination, and beyond. , 2007, Molecular cell.
[37] M. Fransen,et al. Ubiquitination of Mammalian Pex5p, the Peroxisomal Import Receptor* , 2007, Journal of Biological Chemistry.
[38] Z. Elazar,et al. Reactive oxygen species are essential for autophagy and specifically regulate the activity of Atg4 , 2007, The EMBO journal.
[39] M. Löbrich,et al. Hyperthermia activates a subset of ataxia-telangiectasia mutated effectors independent of DNA strand breaks and heat shock protein 70 status. , 2007, Cancer research.
[40] S. Thoms,et al. Peroxisomal matrix protein receptor ubiquitination and recycling. , 2006, Biochimica et biophysica acta.
[41] Michael Schrader,et al. Peroxisomes and oxidative stress. , 2006, Biochimica et biophysica acta.
[42] C. Walker,et al. Activity of TSC2 is inhibited by AKT-mediated phosphorylation and membrane partitioning , 2006, The Journal of cell biology.
[43] T. Pandita,et al. Mammalian Rad9 Plays a Role in Telomere Stability, S- and G2-Phase-Specific Cell Survival, and Homologous Recombinational Repair , 2006, Molecular and Cellular Biology.
[44] Keiji Tanaka,et al. Excess Peroxisomes Are Degraded by Autophagic Machinery in Mammals* , 2006, Journal of Biological Chemistry.
[45] N. Inestrosa,et al. Peroxisomal Proliferation Protects from β-Amyloid Neurodegeneration* , 2005, Journal of Biological Chemistry.
[46] Y. Fujiki,et al. Shuttling Mechanism of Peroxisome Targeting Signal Type 1 Receptor Pex5: ATP-Independent Import and ATP-Dependent Export , 2005, Molecular and Cellular Biology.
[47] J. Cregg,et al. Pexophagy: The Selective Autophagy of Peroxisomes , 2005, Autophagy.
[48] Jiri Bartek,et al. Cell-cycle checkpoints and cancer , 2004, Nature.
[49] S. Subramani,et al. Peroxisome turnover by micropexophagy: an autophagy-related process. , 2004, Trends in cell biology.
[50] Jiri Bartek,et al. Targeting the checkpoint kinases: chemosensitization versus chemoprotection , 2004, Nature Reviews Cancer.
[51] M. Kastan,et al. The many substrates and functions of ATM , 2000, Nature Reviews Molecular Cell Biology.
[52] M. Hande,et al. Inactivation of 14-3-3ς Influences Telomere Behavior and Ionizing Radiation-Induced Chromosomal Instability , 2000, Molecular and Cellular Biology.
[53] T. Zwingman,et al. ATM is a cytoplasmic protein in mouse brain required to prevent lysosomal accumulation. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[54] S. T. Kim,et al. Substrate Specificities and Identification of Putative Substrates of ATM Kinase Family Members* , 1999, The Journal of Biological Chemistry.
[55] M. Gatei,et al. Localization of a Portion of Extranuclear ATM to Peroxisomes* , 1999, The Journal of Biological Chemistry.
[56] R. Darnell,et al. ATM binds to β-adaptin in cytoplasmic vesicles , 1998 .
[57] H. Waterham,et al. Peroxisome biogenesis , 1997, BioEssays : news and reviews in molecular, cellular and developmental biology.
[58] T. Paull,et al. The ATM protein kinase and cellular redox signaling: beyond the DNA damage response. , 2012, TIBS -Trends in Biochemical Sciences. Regular ed.
[59] N. Inestrosa,et al. Peroxisomal proliferation protects from beta-amyloid neurodegeneration. , 2005, The Journal of biological chemistry.
[60] T Hashimoto,et al. Peroxisomal beta-oxidation and peroxisome proliferator-activated receptor alpha: an adaptive metabolic system. , 2001, Annual review of nutrition.
[61] T. Hashimoto,et al. PEROXISOMAL β-OXIDATION AND PEROXISOME PROLIFERATOR–ACTIVATED RECEPTOR α: An Adaptive Metabolic System , 2001 .
[62] R. Darnell,et al. ATM binds to beta-adaptin in cytoplasmic vesicles. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[63] H. Moser,et al. Mutations in PEX1 are the most common cause of peroxisome biogenesis disorders , 1997, Nature Genetics.