Both RyRs and TPCs are required for NAADP-induced intracellular Ca2+ release
暂无分享,去创建一个
K. Mikoshiba | O. Gerasimenko | O. Petersen | J. Gerasimenko | Mark W Sherwood | J. Parrington | P. Ferdek | Richard M. Charlesworth
[1] A. Galione. A primer of NAADP-mediated Ca(2+) signalling: From sea urchin eggs to mammalian cells. , 2015, Cell calcium.
[2] Yigong Shi,et al. Structure of the rabbit ryanodine receptor RyR1 at near-atomic resolution , 2014, Nature.
[3] J. Frank,et al. Structure of a mammalian ryanodine receptor , 2014, Nature.
[4] S. Muallem,et al. Convergent regulation of the lysosomal two‐pore channel‐2 by Mg2+, NAADP, PI(3,5)P2 and multiple protein kinases , 2014, The EMBO journal.
[5] O. Petersen,et al. The role of Ca2+ in the pathophysiology of pancreatitis , 2013, The Journal of physiology.
[6] David E. Clapham,et al. mTOR Regulates Lysosomal ATP-Sensitive Two-Pore Na+ Channels to Adapt to Metabolic State , 2013, Cell.
[7] Michael X. Zhu,et al. TPC Proteins Are Phosphoinositide- Activated Sodium-Selective Ion Channels in Endosomes and Lysosomes , 2012, Cell.
[8] A. Guse. Linking NAADP to Ion Channel Activity: A Unifying Hypothesis , 2012, Science Signaling.
[9] O. Gerasimenko,et al. Aberrant Ca(2+) signalling through acidic calcium stores in pancreatic acinar cells. , 2011, Cell calcium.
[10] Alexander M. Lewis,et al. Analogues of the Nicotinic Acid Adenine Dinucleotide Phosphate (NAADP) Antagonist Ned-19 Indicate Two Binding Sites on the NAADP Receptor* , 2009, The Journal of Biological Chemistry.
[11] N. Dun,et al. Essential requirement for two-pore channel 1 in NAADP-mediated calcium signaling , 2009, The Journal of cell biology.
[12] M. Hohenegger,et al. NAADP-mediated Ca2+ signaling via type 1 ryanodine receptor in T cells revealed by a synthetic NAADP antagonist , 2009, Proceedings of the National Academy of Sciences.
[13] O. Petersen,et al. Fatty acids, alcohol and fatty acid ethyl esters: toxic Ca2+ signal generation and pancreatitis. , 2009, Cell calcium.
[14] A. Galione,et al. NAADP mobilizes calcium from acidic organelles through two-pore channels , 2009, Nature.
[15] Alexander M. Lewis,et al. Identification of a chemical probe for NAADP by virtual screening , 2009, Nature chemical biology.
[16] O. Petersen,et al. Polarized calcium signaling in exocrine gland cells. , 2008, Annual review of physiology.
[17] R. Billington,et al. PPADS is a reversible competitive antagonist of the NAADP receptor. , 2007, Cell calcium.
[18] I. Prior,et al. Activation of trypsinogen in large endocytic vacuoles of pancreatic acinar cells , 2007, Proceedings of the National Academy of Sciences.
[19] J. Deitmer,et al. Calcium mobilization by nicotinic acid adenine dinucleotide phosphate (NAADP) in rat astrocytes. , 2006, Cell calcium.
[20] O. Petersen,et al. NAADP, cADPR and IP3 all release Ca2+ from the endoplasmic reticulum and an acidic store in the secretory granule area , 2006, Journal of Cell Science.
[21] Andreas H. Guse,et al. Functional Ryanodine Receptor Expression Is Required for NAADP-mediated Local Ca2+ Signaling in T-lymphocytes* , 2005, Journal of Biological Chemistry.
[22] Alexander M. Lewis,et al. Role of NAADP and cADPR in the Induction and Maintenance of Agonist-Evoked Ca2+ Spiking in Mouse Pancreatic Acinar Cells , 2005, Current Biology.
[23] Ole H Petersen,et al. The NAADP receptor: new receptors or new regulation? , 2005, Molecular interventions.
[24] A. Galione,et al. Organelle Selection Determines Agonist-specific Ca2+ Signals in Pancreatic Acinar and β Cells* , 2004, Journal of Biological Chemistry.
[25] A. Tepikin,et al. The endoplasmic reticulum as one continuous Ca2+ pool: visualization of rapid Ca2+ movements and equilibration , 2000, The EMBO journal.
[26] O. Gerasimenko,et al. Two different but converging messenger pathways to intracellular Ca2+ release: the roles of nicotinic acid adenine dinucleotide phosphate, cyclic ADP‐ribose and inositol trisphosphate , 2000, The EMBO journal.
[27] K. Mikoshiba,et al. Facilitation of NMDAR-Independent LTP and Spatial Learning in Mutant Mice Lacking Ryanodine Receptor Type 3 , 1999, Neuron.
[28] O. Gerasimenko,et al. Calcium uptake via endocytosis with rapid release from acidifying endosomes , 1998, Current Biology.
[29] W. Chin,et al. Role of Ca2+/K+ ion exchange in intracellular storage and release of Ca2+ , 1998, Nature.
[30] Min Goo Lee,et al. Polarized Expression of Ca2+ Channels in Pancreatic and Salivary Gland Cells , 1997, The Journal of Biological Chemistry.
[31] R. Wojcikiewicz,et al. Evidence That Zymogen Granules Are Not a Physiologically Relevant Calcium Pool , 1997, The Journal of Biological Chemistry.
[32] A. Tepikin,et al. Ca2+ Flow via Tunnels in Polarized Cells: Recharging of Apical Ca2+ Stores by Focal Ca2+ Entry through Basal Membrane Patch , 1997, Cell.
[33] P. Pacaud,et al. The effect of PPADS as an antagonist of inositol (1,4,5)trisphosphate induced intracellular calcium mobilization , 1996, British journal of pharmacology.
[34] O. Gerasimenko,et al. Inositol Trisphosphate and Cyclic ADP-Ribose–Mediated Release of Ca2+ from Single Isolated Pancreatic Zymogen Granules , 1996, Cell.
[35] M. Fallon,et al. Localization of the type 3 inositol 1,4,5-trisphosphate receptor in the Ca2+ wave trigger zone of pancreatic acinar cells. , 1994, The Journal of biological chemistry.
[36] G. Burnstock,et al. PPADS selectively antagonizes P2X‐purinoceptor‐mediated responses in the rabbit urinary bladder , 1993, British journal of pharmacology.
[37] O. Petersen,et al. Local and global cytosolic Ca2+ oscillations in exocrine cells evoked by agonists and inositol trisphosphate , 1993, Cell.
[38] Y. Miyashita,et al. Subcellular distribution of Ca2+ release channels underlying Ca2+ waves and oscillations in exocrine pancreas , 1993, Cell.
[39] H. Bäumert,et al. PPADS, a novel functionally selective antagonist of P2 purinoceptor-mediated responses. , 1992, European journal of pharmacology.
[40] O. Petersen,et al. Receptor-activated cytoplasmic Ca2+ spiking mediated by inositol trisphosphate is due to Ca2+-induced Ca2+ release , 1990, Cell.
[41] S. Yoo,et al. Inositol 1,4,5-trisphosphate-triggered Ca2+ release from bovine adrenal medullary secretory vesicles. , 1990, The Journal of biological chemistry.
[42] F. Thévenod,et al. Modulation of intracellular free Ca2+ concentration by IP3-sensitive and IP3-insensitive nonmitochondrial Ca2+ pools. , 1989, Cell calcium.
[43] M. J. Berridge,et al. Release of Ca2+ from a nonmitochondrial intracellular store in pancreatic acinar cells by inositol-1,4,5-trisphosphate , 1983, Nature.
[44] O. Petersen,et al. Transport of calcium in the perfused submandibular gland of the cat , 1972, The Journal of physiology.
[45] Z. Selinger,et al. ATP-dependent calcium uptake by microsomal preparations from rat parotid and submaxillary glands. , 1970, Biochimica et biophysica acta.
[46] E. Bayerdörffer,et al. Effect of inositol-1,4,5-trisphosphate on isolated subcellular fractions of rat pancreas , 2005, The Journal of Membrane Biology.