L-tryptophan-kynurenine pathway metabolite 3-hydroxyanthranilic acid induces apoptosis in macrophage-derived cells under pathophysiological conditions.
暂无分享，去创建一个
M. Takemura | M. Seishima | K. Saito | S. Fujigaki | H. Fujii | H. Wada | A. Noma | N. Maekawa | T. Morita | S. Takeuchi
[1] N. Nishiyama,et al. 3‐Hydroxykynurenine, an Endogenous Oxidative Stress Generator, Causes Neuronal Cell Death with Apoptotic Features and Region Selectivity , 1998, Journal of neurochemistry.
[2] C. Reutelingsperger,et al. Expression on B Cells Undergoing Apoptosis Annexin V for Flow Cytometric Detection of Phosphatidylserine , 2022 .
[3] S. Markey,et al. A mechanism for increased quinolinic acid formation following acute systemic immune stimulation. , 1993, The Journal of biological chemistry.
[4] S. Markey,et al. 4-Chloro-3-hydroxyanthranilate, 6-chlorotryptophan and norharmane attenuate quinolinic acid formation by interferon-gamma-stimulated monocytes (THP-1 cells). , 1993, The Biochemical journal.
[5] R. Stocker,et al. Oxidation of 3-hydroxyanthranilic acid to the phenoxazinone cinnabarinic acid by peroxyl radicals and by compound I of peroxidases or catalase. , 1992, Biochemistry.