Expression of intercellular adhesion molecules in human saphenous veins: effects of inflammatory cytokines and neointima formation in culture.

[1]  M. Yacoub,et al.  Expression of vascular adhesion molecules in saphenous vein coronary bypass grafts. , 1998, The Annals of thoracic surgery.

[2]  G. Angelini,et al.  Adenovirus-mediated gene transfer of the human TIMP-1 gene inhibits smooth muscle cell migration and neointimal formation in human saphenous vein. , 1998, Human gene therapy.

[3]  M Janier,et al.  Mitral subvalvular apparatus: different functions of primary and secondary chordae. , 1997, Circulation.

[4]  G. Baumann,et al.  Regulation of Tumor Necrosis Factor-α and Interleukin-1β Induced Adhesion Molecule Expression in Human Vascular Smooth Muscle Cells by cAMP , 1997 .

[5]  G. Angelini,et al.  Short-term exposure to thapsigargin inhibits neointima formation in human saphenous vein. , 1997, Arteriosclerosis, thrombosis, and vascular biology.

[6]  G. Schett,et al.  Co-expression of ICAM-1, VCAM-1, ELAM-1 and Hsp60 in human arterial and venous endothelial cells in response to cytokines and oxidized low-density lipoproteins. , 1997, Cell stress & chaperones.

[7]  J. Powell,et al.  Circumferential deformation and shear stress induce differential responses in saphenous vein endothelium exposed to arterial flow. , 1997, The Journal of clinical investigation.

[8]  F. Liao,et al.  Soluble Domain 1 of Platelet–Endothelial Cell Adhesion Molecule (PECAM) Is Sufficient to Block Transendothelial Migration In Vitro and In Vivo , 1997, The Journal of experimental medicine.

[9]  M. Morimatsu,et al.  Inhibition of intimal hyperplasia after balloon injury by antibodies to intercellular adhesion molecule-1 and lymphocyte function-associated antigen-1. , 1997, Circulation.

[10]  Post Coronary Artery Bypass Graft Trial Investigators The effect of aggressive lowering of low-density lipoprotein cholesterol levels and low-dose anticoagulation on obstructive changes in saphenous-vein coronary-artery bypass grafts. , 1997, The New England journal of medicine.

[11]  A. Robert,et al.  [Hyaluronic acid (hyaluronan) levels in pathological human saphenous veins. Effects of procyanidol oligomers]. , 1997, Pathologie-biologie.

[12]  N. Morisaki,et al.  Specific inhibition of vascular cell adhesion molecule-1 expression by type IV collagen in endothelial cells. , 1995, Biochemical and biophysical research communications.

[13]  J. Gamble,et al.  TGF-β and Endothelial Cells Inhibit VCAM-1 Expression on Human Vascular Smooth Muscle Cells , 1995 .

[14]  T. Schall,et al.  IL‐1 activation of endothelium supports VLA‐4 (CD49d/CD29)‐mediated monocyte transendothelial migration to C5a, MIP‐1α, RANTES, and PAF but inhibits migration to MCP‐1: a regulatory role for endothelium‐derived MCP‐1 , 1995, Journal of leukocyte biology.

[15]  M. Furie,et al.  The adhesion molecules used by monocytes for migration across endothelium include CD11a/CD18, CD11b/CD18, and VLA-4 on monocytes and ICAM-1, VCAM-1, and other ligands on endothelium. , 1995, Journal of immunology.

[16]  G. Angelini,et al.  The biology of saphenous vein graft occlusion: etiology and strategies for prevention , 1994, Current opinion in cardiology.

[17]  C. Marwick Coronary bypass grafting economics, including rehabilitation. , 1994, Current opinion in cardiology.

[18]  M. Furie,et al.  Monocytes use either CD11/CD18 or VLA-4 to migrate across human endothelium in vitro. , 1994, Journal of immunology.

[19]  J. Bonnet,et al.  Regulation of vascular cell adhesion molecule-1 and intercellular adhesion molecule-1 in human vascular smooth muscle cells. , 1994, Circulation research.

[20]  T. Springer Traffic signals for lymphocyte recirculation and leukocyte emigration: The multistep paradigm , 1994, Cell.

[21]  G. Angelini,et al.  Surgical preparation induces injury and promotes smooth muscle cell proliferation in a culture of human saphenous vein. , 1993, Cardiovascular research.

[22]  D. Phillips,et al.  PECAM-1 is required for transendothelial migration of leukocytes , 1993, The Journal of experimental medicine.

[23]  A. Gown,et al.  Various cell types in human atherosclerotic lesions express ICAM-1. Further immunocytochemical and immunochemical studies employing monoclonal antibody 10F3. , 1992, The American journal of pathology.

[24]  J. Coucher,et al.  Expression of intercellular adhesion molecule-1 in atherosclerotic plaques. , 1992, The American journal of pathology.

[25]  G. Angelini,et al.  Intimal proliferation in an organ culture of human saphenous vein. , 1990, The American journal of pathology.

[26]  J. Matloff,et al.  Thrombosed, ruptured atheromatous plaques in saphenous vein coronary artery bypass grafts: ten years' experience. , 1987, American heart journal.

[27]  G. Angelini,et al.  A surgical preparative technique for coronary bypass grafts of human saphenous vein which preserves medial and endothelial functional integrity. , 1987, The Journal of thoracic and cardiovascular surgery.

[28]  J. Barboriak,et al.  Atherosclerosis in aortocoronary bypass grafts. Morphologic study and risk factor analysis 6 to 12 years after surgery. , 1986, Arteriosclerosis.

[29]  D. Bowyer,et al.  Endothelial injury and healing in vitro. Studies using an organ culture system. , 1985, The American journal of pathology.

[30]  G. Hutchins,et al.  Accelerated “Atherosclerosis” A Morphologic Study of 97 Saphenous Vein Coronary Artery Bypass Grafts: A Morphologic Study of 97 Saphenous Vein Coronary Artery Bypass Grafts , 1977, Circulation.

[31]  E. Jaffe,et al.  Culture of human endothelial cells derived from umbilical veins. Identification by morphologic and immunologic criteria. , 1973, The Journal of clinical investigation.