Expression of intercellular adhesion molecules in human saphenous veins: effects of inflammatory cytokines and neointima formation in culture.
暂无分享,去创建一个
[1] M. Yacoub,et al. Expression of vascular adhesion molecules in saphenous vein coronary bypass grafts. , 1998, The Annals of thoracic surgery.
[2] G. Angelini,et al. Adenovirus-mediated gene transfer of the human TIMP-1 gene inhibits smooth muscle cell migration and neointimal formation in human saphenous vein. , 1998, Human gene therapy.
[3] M Janier,et al. Mitral subvalvular apparatus: different functions of primary and secondary chordae. , 1997, Circulation.
[4] G. Baumann,et al. Regulation of Tumor Necrosis Factor-α and Interleukin-1β Induced Adhesion Molecule Expression in Human Vascular Smooth Muscle Cells by cAMP , 1997 .
[5] G. Angelini,et al. Short-term exposure to thapsigargin inhibits neointima formation in human saphenous vein. , 1997, Arteriosclerosis, thrombosis, and vascular biology.
[6] G. Schett,et al. Co-expression of ICAM-1, VCAM-1, ELAM-1 and Hsp60 in human arterial and venous endothelial cells in response to cytokines and oxidized low-density lipoproteins. , 1997, Cell stress & chaperones.
[7] J. Powell,et al. Circumferential deformation and shear stress induce differential responses in saphenous vein endothelium exposed to arterial flow. , 1997, The Journal of clinical investigation.
[8] F. Liao,et al. Soluble Domain 1 of Platelet–Endothelial Cell Adhesion Molecule (PECAM) Is Sufficient to Block Transendothelial Migration In Vitro and In Vivo , 1997, The Journal of experimental medicine.
[9] M. Morimatsu,et al. Inhibition of intimal hyperplasia after balloon injury by antibodies to intercellular adhesion molecule-1 and lymphocyte function-associated antigen-1. , 1997, Circulation.
[10] Post Coronary Artery Bypass Graft Trial Investigators. The effect of aggressive lowering of low-density lipoprotein cholesterol levels and low-dose anticoagulation on obstructive changes in saphenous-vein coronary-artery bypass grafts. , 1997, The New England journal of medicine.
[11] A. Robert,et al. [Hyaluronic acid (hyaluronan) levels in pathological human saphenous veins. Effects of procyanidol oligomers]. , 1997, Pathologie-biologie.
[12] N. Morisaki,et al. Specific inhibition of vascular cell adhesion molecule-1 expression by type IV collagen in endothelial cells. , 1995, Biochemical and biophysical research communications.
[13] J. Gamble,et al. TGF-β and Endothelial Cells Inhibit VCAM-1 Expression on Human Vascular Smooth Muscle Cells , 1995 .
[14] T. Schall,et al. IL‐1 activation of endothelium supports VLA‐4 (CD49d/CD29)‐mediated monocyte transendothelial migration to C5a, MIP‐1α, RANTES, and PAF but inhibits migration to MCP‐1: a regulatory role for endothelium‐derived MCP‐1 , 1995, Journal of leukocyte biology.
[15] M. Furie,et al. The adhesion molecules used by monocytes for migration across endothelium include CD11a/CD18, CD11b/CD18, and VLA-4 on monocytes and ICAM-1, VCAM-1, and other ligands on endothelium. , 1995, Journal of immunology.
[16] G. Angelini,et al. The biology of saphenous vein graft occlusion: etiology and strategies for prevention , 1994, Current opinion in cardiology.
[17] C. Marwick. Coronary bypass grafting economics, including rehabilitation. , 1994, Current opinion in cardiology.
[18] M. Furie,et al. Monocytes use either CD11/CD18 or VLA-4 to migrate across human endothelium in vitro. , 1994, Journal of immunology.
[19] J. Bonnet,et al. Regulation of vascular cell adhesion molecule-1 and intercellular adhesion molecule-1 in human vascular smooth muscle cells. , 1994, Circulation research.
[20] T. Springer. Traffic signals for lymphocyte recirculation and leukocyte emigration: The multistep paradigm , 1994, Cell.
[21] G. Angelini,et al. Surgical preparation induces injury and promotes smooth muscle cell proliferation in a culture of human saphenous vein. , 1993, Cardiovascular research.
[22] D. Phillips,et al. PECAM-1 is required for transendothelial migration of leukocytes , 1993, The Journal of experimental medicine.
[23] A. Gown,et al. Various cell types in human atherosclerotic lesions express ICAM-1. Further immunocytochemical and immunochemical studies employing monoclonal antibody 10F3. , 1992, The American journal of pathology.
[24] J. Coucher,et al. Expression of intercellular adhesion molecule-1 in atherosclerotic plaques. , 1992, The American journal of pathology.
[25] G. Angelini,et al. Intimal proliferation in an organ culture of human saphenous vein. , 1990, The American journal of pathology.
[26] J. Matloff,et al. Thrombosed, ruptured atheromatous plaques in saphenous vein coronary artery bypass grafts: ten years' experience. , 1987, American heart journal.
[27] G. Angelini,et al. A surgical preparative technique for coronary bypass grafts of human saphenous vein which preserves medial and endothelial functional integrity. , 1987, The Journal of thoracic and cardiovascular surgery.
[28] J. Barboriak,et al. Atherosclerosis in aortocoronary bypass grafts. Morphologic study and risk factor analysis 6 to 12 years after surgery. , 1986, Arteriosclerosis.
[29] D. Bowyer,et al. Endothelial injury and healing in vitro. Studies using an organ culture system. , 1985, The American journal of pathology.
[30] G. Hutchins,et al. Accelerated “Atherosclerosis” A Morphologic Study of 97 Saphenous Vein Coronary Artery Bypass Grafts: A Morphologic Study of 97 Saphenous Vein Coronary Artery Bypass Grafts , 1977, Circulation.
[31] E. Jaffe,et al. Culture of human endothelial cells derived from umbilical veins. Identification by morphologic and immunologic criteria. , 1973, The Journal of clinical investigation.