Differential neuropeptide expression after visceral and somatic nerve injury in the cat and rat

[1]  G. Lewin,et al.  Quantitative analysis of peptide levels and neurogenic extravasation following regeneration of afferents to appropriate and inappropriate targets , 1989, Neuroscience.

[2]  T. Hökfelt,et al.  Increase of galanin-like immunoreactivity in rat dorsal root ganglion cells after peripheral axotomy , 1987, Neuroscience Letters.

[3]  P. Anand,et al.  Neuropeptides are selectivemarkers of spinal cord autonomic pathways , 1984, Trends in Neurosciences.

[4]  A. Cameron,et al.  Coexistence of peptide immunoreactivity in sensory neurons of the cat , 1985, Neuroscience.

[5]  T. Hökfelt,et al.  Neuropeptide expression in rat dorsal root ganglion cells and spinal cord after peripheral nerve injury with special reference to galanin , 1989, Neuroscience.

[6]  P. Anand,et al.  Neuropeptides in Leprosy: Evidence of a Role for Vip in the Re-Organisation or Regeneration of the Nervous System , 1983 .

[7]  S. Landis Target regulation of neurotransmitter phenotype , 1990, Trends in Neurosciences.

[8]  P. Anand,et al.  A VIP/PHI-containing pathway links urinary bladder and sacral spinal cord , 1986, Peptides.

[9]  F. Scaravilli,et al.  A new neurological rat mutant "mutilated foot". , 1981, Journal of anatomy.

[10]  M. Atkinson,et al.  Vasoactive intestinal polypeptide (VIP) increases in the spinal cord after peripheral axotomy of the sciatic nerve originate from primary afferent neurons , 1986, Brain Research.

[11]  M. Tohyama,et al.  Vasoactive intestinal polypeptide (VIP)-containing neurons in the spinal cord of the rat and their projections , 1983, Neuroscience Letters.

[12]  P. Anand,et al.  Distribution of galanin immunoreactivity in the central nervous system and the responses of galanin-containing neuronal pathways to injury , 1985, Neuroscience.

[13]  Ichiro Kanazawa,et al.  Substance P: Depletion in the dorsal horn of rat spinal cord after section of the peripheral processes of primary sensory neurons , 1979, Brain Research.

[14]  P. Wall,et al.  Effect of peripheral nerve section and nerve crush on spinal cord neuropeptides in the rat; increased VIP and PHI in the dorsal horn , 1984, Neuroscience.

[15]  P. Anand,et al.  Increase of substance P-like immunoreactivity in the peripheral nerve of the axolotl after injury , 1987, Neuroscience Letters.

[16]  H. Thoenen,et al.  A comparison of the effects of chemical sympathectomy by 6‐hydroxydopamine in newborn and adult rats , 1973, British journal of pharmacology.

[17]  P. Anand,et al.  Depletion of neuropeptides during wound healing in rat skin , 1986, Neuroscience Letters.

[18]  P. Anand,et al.  A VIP-containing system concentrated in the lumbosacral region of human spinal cord , 1983, Nature.

[19]  S. Hunt,et al.  Biochemical and anatomical observations on the degeneration of peptide-containing primary afferent neurons after neonatal capsaicin , 1981, Neuroscience.

[20]  P. Anand,et al.  Marked depletion of dorsal spinal cord substance P and calcitonin gene-related peptide with intact skin flare responses in multiple system atrophy. , 1988, Journal of neurology, neurosurgery, and psychiatry.

[21]  F. Scaravilli,et al.  Studies of vasoactive intestinal polypeptide expression in injured peripheral neurons using capsaicin, sympathectomy and mf mutant rats , 1990, Neuroscience Letters.

[22]  P. Anand,et al.  The distribution and origin of VIP in the spinal cord of six mammalian species , 1984, Peptides.

[23]  P. Anand,et al.  Peripheral neuronal systems show marked increases in VIP/PHI and CCK content following injury , 1983, Regulatory Peptides.

[24]  P. Anand,et al.  Water content, vasoactive intestinal polypeptide and substance P in intact and crushed sciatic nerves of normal and streptozotocin-diabetic rats , 1988, Journal of the Neurological Sciences.