Systematic review: antibodies and anti‐TNF‐α levels in inflammatory bowel disease
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J. Gisbert | M. Chaparro | J. Gisbert | I. Guerra | P. Muñoz-Linares | María Chaparro | J. P. Gisbert
[1] G. Paintaud,et al. P334 Concentration effect relationship of infliximab in Crohn's disease: Results of a cohort study , 2012 .
[2] M. Lukáš,et al. P360 Infliximab trough levels may predict sustained response to infliximab in patients with Crohn's disease: A single cohort study , 2012 .
[3] Ann Gils,et al. OP11 Individualised infliximab treatment using therapeutic drug monitoring: A prospective controlled Trough level Adapted infliXImab Treatment (TAXIT) trial , 2012 .
[4] P. Rutgeerts,et al. OP10 Importance of trough levels and antibodies on the long-term efficacy of infliximab therapy in ulcerative colitis , 2012 .
[5] G. D'Haens,et al. Adalimumab induces and maintains clinical remission in patients with moderate-to-severe ulcerative colitis. , 2012, Gastroenterology.
[6] A. Nesbitt,et al. A PEGylated Fab’ Fragment against Tumor Necrosis Factor for the Treatment of Crohn Disease , 2012, BioDrugs.
[7] P. Rutgeerts,et al. Infliximab for Severe IV Steroid-Refractory Ulcerative Colitis: Can Infliximab Trough Levels Guide Our Management? , 2011 .
[8] K. Bendtzen,et al. Can Measurements of Anti-Infliximab Antibodies Predict Acute Severe Infusion Reactions to Infliximab? , 2011 .
[9] Yasuo Suzuki,et al. Monitoring functional serum antitumor necrosis factor antibody level in Crohn's disease patients who maintained and those who lost response to anti‐TNF , 2010, Inflammatory bowel diseases.
[10] M. Gazouli,et al. Report of the ECCO workshop on anti-TNF therapy failures in inflammatory bowel diseases: biological roles and effects of TNF and TNF antagonists. , 2010, Journal of Crohn's & colitis.
[11] R. Fedorak,et al. Tumor necrosis factor-α monoclonal antibodies in the treatment of inflammatory bowel disease: clinical practice pharmacology. , 2010, Gastroenterology clinics of North America.
[12] S. Brand,et al. Disease Activity, ANCA, and IL23R Genotype Status Determine Early Response to Infliximab in Patients With Ulcerative Colitis , 2010, The American Journal of Gastroenterology.
[13] U. Kopylov,et al. The immunogenic part of infliximab is the F(ab′)2, but measuring antibodies to the intact infliximab molecule is more clinically useful , 2010, Gut.
[14] W. Sandborn,et al. Clinical Utility of Measuring Infliximab and Human Anti-Chimeric Antibody Concentrations in Patients With Inflammatory Bowel Disease , 2010, The American Journal of Gastroenterology.
[15] A. Cohen,et al. S1051 Methotrexate for the Prevention of Antibodies to Infliximab in Patients With Crohn's Disease , 2010 .
[16] P. Rutgeerts,et al. 405 High Infliximab Trough Levels are Associated With Mucosal Healing in Crohn's Disease , 2010 .
[17] Y. Chiu,et al. 741 Evaluation of Potential Correlations Between Serum Adalimumab Concentration and Remission in Patients With Crohn's Disease in Classic I and II , 2010 .
[18] P. Rutgeerts,et al. Infliximab, azathioprine, or combination therapy for Crohn's disease. , 2010, The New England journal of medicine.
[19] E. Bonfá,et al. Immunogenicity of Anti-TNF-α Agents in Autoimmune Diseases , 2010, Clinical reviews in allergy & immunology.
[20] B. Dijkmans,et al. Relationship between the clinical response to adalimumab treatment and serum levels of adalimumab and anti-adalimumab antibodies in patients with psoriatic arthritis , 2010, Annals of the rheumatic diseases.
[21] Takahiko Horiuchi,et al. Transmembrane TNF-α: structure, function and interaction with anti-TNF agents , 2010, Rheumatology.
[22] B. Dijkmans,et al. Anti-infliximab and anti-adalimumab antibodies in relation to response to adalimumab in infliximab switchers and anti-tumour necrosis factor naive patients: a cohort study , 2009, Annals of the rheumatic diseases.
[23] P. Rutgeerts,et al. The effect of dose escalation on trough levels in patients who lost response to infliximab , 2010 .
[24] P. Rutgeerts,et al. Influence of trough serum levels and immunogenicity on long-term outcome of adalimumab therapy in Crohn's disease. , 2009, Gastroenterology.
[25] M. Silverberg,et al. Trough serum infliximab: a predictive factor of clinical outcome for infliximab treatment in acute ulcerative colitis , 2009, Gut.
[26] S. Travis,et al. Incidence and clinical significance of immunogenicity to infliximab in Crohn's disease: A critical systematic review , 2009, Inflammatory bowel diseases.
[27] W. Sandborn,et al. Clinical trial: benefits and risks of immunomodulators and maintenance infliximab for IBD‐subgroup analyses across four randomized trials , 2009, Alimentary pharmacology & therapeutics.
[28] E. Sasso,et al. Comparisons of affinities, avidities, and complement activation of adalimumab, infliximab, and etanercept in binding to soluble and membrane tumor necrosis factor. , 2009, Clinical immunology.
[29] J. Gisbert,et al. Loss of Response and Requirement of Infliximab Dose Intensification in Crohn's Disease: A Review , 2009, The American Journal of Gastroenterology.
[30] K. Bendtzen,et al. Individual medicine in inflammatory bowel disease: Monitoring bioavailability, pharmacokinetics and immunogenicity of anti-tumour necrosis factor-alpha antibodies , 2009, Scandinavian journal of gastroenterology.
[31] E. Kuipers,et al. Immunogenicity negatively influences the outcome of adalimumab treatment in Crohn’s disease , 2008, Alimentary pharmacology & therapeutics.
[32] P. Rutgeerts,et al. Withdrawal of immunosuppression in Crohn's disease treated with scheduled infliximab maintenance: a randomized trial. , 2008, Gastroenterology.
[33] K. Bendtzen,et al. Tumor Necrosis Factor-Alpha Binding Capacity and Anti-Infliximab Antibodies Measured by Fluid-Phase Radioimmunoassays as Predictors of Clinical Efficacy of Infliximab in Crohn's Disease , 2008, The American Journal of Gastroenterology.
[34] G. D'Haens,et al. Adalimumab Induction Therapy for Crohn Disease Previously Treated with Infliximab: A Randomized Trial , 2008 .
[35] G. Kaplan,et al. Infliximab dose escalation vs. initiation of adalimumab for loss of response in Crohn’s disease: a cost‐effectiveness analysis , 2007, Alimentary pharmacology & therapeutics.
[36] S. Targan,et al. Natalizumab for the treatment of active Crohn's disease: results of the ENCORE Trial. , 2007, Gastroenterology.
[37] P. Rutgeerts,et al. Effectiveness of concomitant immunosuppressive therapy in suppressing the formation of antibodies to infliximab in Crohn’s disease , 2007, Gut.
[38] S. Hanauer,et al. General principles and pharmacology of biologics in inflammatory bowel disease. , 2006, Gastroenterology clinics of North America.
[39] J. Gisbert,et al. Monitorización de la tiopurina metiltransferasa y de los metabolitos tiopurínicos para optimizar el tratamiento con azatioprina en la enfermedad inflamatoria intestinal , 2006 .
[40] M. Silverberg,et al. Association of trough serum infliximab to clinical outcome after scheduled maintenance treatment for Crohn's disease. , 2006, Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association.
[41] J. Mate,et al. [Monitoring of thiopurine methyltransferase and thiopurine metabolites to optimize azathioprine therapy in inflammatory bowel disease]. , 2006, Gastroenterologia y hepatologia.
[42] C. Aybay,et al. Demonstration of specific antibodies against infliximab induced during treatment of a patient with ankylosing spondylitis , 2006, Rheumatology International.
[43] S. Targan,et al. Natalizumab induction and maintenance therapy for Crohn's disease. , 2005, The New England journal of medicine.
[44] S. Schreiber,et al. A randomized, placebo-controlled trial of certolizumab pegol (CDP870) for treatment of Crohn's disease. , 2005, Gastroenterology.
[45] M. Bala,et al. Incidence and importance of antibody responses to infliximab after maintenance or episodic treatment in Crohn's disease. , 2004, Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association.
[46] P. Rutgeerts,et al. Optimizing anti-TNF treatment in inflammatory bowel disease. , 2004, Gastroenterology.
[47] M. Peppercorn,et al. Intravenous hydrocortisone premedication reduces antibodies to infliximab in Crohn's disease: a randomized controlled trial. , 2003, Gastroenterology.
[48] An Carbonez,et al. Influence of immunogenicity on the long-term efficacy of infliximab in Crohn's disease. , 2003, The New England journal of medicine.
[49] S. Hanauer,et al. For Personal Use. Only Reproduce with Permission from the Lancet Publishing Group , 2022 .
[50] S. Targan,et al. Tumor necrosis factor: biology and therapeutic inhibitors. , 2000, Gastroenterology.
[51] J. Bonnefoy,et al. Human NK cells constitutively express membrane TNF‐α (mTNFα) and present mTNFα‐dependent cytotoxic activity , 1999 .
[52] Y. Goltsev,et al. Tumor necrosis factor receptor and Fas signaling mechanisms. , 1999, Annual review of immunology.
[53] G. Kollias,et al. Impaired on/off regulation of TNF biosynthesis in mice lacking TNF AU-rich elements: implications for joint and gut-associated immunopathologies. , 1999, Immunity.
[54] J. Bonnefoy,et al. Human NK cells constitutively express membrane TNF-alpha (mTNFalpha) and present mTNFalpha-dependent cytotoxic activity. , 1999, European journal of immunology.
[55] J. Keane,et al. Pathogenic Mycobacterium tuberculosis evades apoptosis of host macrophages by release of TNF-R2, resulting in inactivation of TNF-alpha. , 1998, Journal of immunology.
[56] S. Targan,et al. A Short-Term Study of Chimeric Monoclonal Antibody cA2 to Tumor Necrosis Factor α for Crohn's Disease , 1997 .
[57] M. Feldmann,et al. Contact with T cells modulates monocyte IL-10 production: role of T cell membrane TNF-alpha. , 1997, Journal of immunology.
[58] Nicole Nelson,et al. A metalloproteinase disintegrin that releases tumour-necrosis factor-α from cells , 1997, Nature.
[59] S. Targan,et al. A short-term study of chimeric monoclonal antibody cA2 to tumor necrosis factor alpha for Crohn's disease. Crohn's Disease cA2 Study Group. , 1997, The New England journal of medicine.
[60] B. Castner,et al. A metalloproteinase disintegrin that releases tumour-necrosis factor-alpha from cells. , 1997, Nature.
[61] T. Decker,et al. Cell-associated tumor necrosis factor (TNF) as a killing mechanism of activated cytotoxic macrophages. , 1987, Journal of immunology.