Pregnancy and obesity modify the epithelial composition and hormone signaling state of the human breast
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R. J. Weber | Zev J. Gartner | Christopher S. McGinnis | Z. Gartner | A. Borowsky | P. Gascard | T. Tlsty | Lyndsay M. Murrow | Joseph A. Caruso | M. Thomson | T. Desai
[1] Daniel J. Gaffney,et al. souporcell: Robust clustering of single cell RNAseq by genotype and ambient RNA inference without reference genotypes , 2019, bioRxiv.
[2] Jennifer L Hu,et al. MULTI-seq: sample multiplexing for single-cell RNA sequencing using lipid-tagged indices , 2019, Nature Methods.
[3] R. Satija,et al. Normalization and variance stabilization of single-cell RNA-seq data using regularized negative binomial regression , 2019, Genome Biology.
[4] B. Rosner,et al. Parity, breastfeeding, and breast cancer risk by hormone receptor status and molecular phenotype: results from the Nurses’ Health Studies , 2019, Breast Cancer Research.
[5] Christoph Hafemeister,et al. Comprehensive integration of single cell data , 2018, bioRxiv.
[6] Eliah R. Shamir,et al. Myoepithelial cells are a dynamic barrier to epithelial dissemination , 2018, The Journal of cell biology.
[7] W. Willett,et al. Association of Body Mass Index and Age With Subsequent Breast Cancer Risk in Premenopausal Women , 2018, JAMA oncology.
[8] Z. Werb,et al. Profiling human breast epithelial cells using single cell RNA sequencing identifies cell diversity , 2018, Nature Communications.
[9] P. Hannaford,et al. Contemporary Hormonal Contraception and the Risk of Breast Cancer , 2017, The New England journal of medicine.
[10] L. Arendt,et al. Obesity reversibly depletes the basal cell population and enhances mammary epithelial cell estrogen receptor alpha expression and progenitor activity , 2017, Breast Cancer Research.
[11] A. Van Keymeulen,et al. Lineage-Restricted Mammary Stem Cells Sustain the Development, Homeostasis, and Regeneration of the Estrogen Receptor Positive Lineage , 2017, Cell reports.
[12] Wenjun Guo,et al. Lineage-Biased Stem Cells Maintain Estrogen-Receptor-Positive and -Negative Mouse Mammary Luminal Lineages. , 2017, Cell reports.
[13] C. Ng,et al. Pregnancy at early age is associated with a reduction of progesterone-responsive cells and epithelial Wnt signaling in human breast tissue , 2017, Oncotarget.
[14] Philippe Andrey,et al. MorphoLibJ: integrated library and plugins for mathematical morphology with ImageJ , 2016, Bioinform..
[15] O. Petersen,et al. Propagation of oestrogen receptor-positive and oestrogen-responsive normal human breast cells in culture , 2015, Nature Communications.
[16] May Yin Lee,et al. Reactivation of multipotency by oncogenic PIK3CA induces breast tumour heterogeneity , 2015, Nature.
[17] Michael B. Stadler,et al. PIK3CAH1047R induces multipotency and multi-lineage mammary tumours , 2015, Nature.
[18] J. Rougemont,et al. Progesterone and Wnt4 control mammary stem cells via myoepithelial crosstalk , 2015, The EMBO journal.
[19] W. Huber,et al. Moderated estimation of fold change and dispersion for RNA-seq data with DESeq2 , 2014, Genome Biology.
[20] D. Buist,et al. Recent oral contraceptive use by formulation and breast cancer risk among women 20 to 49 years of age. , 2014, Cancer research.
[21] Alan Mackay,et al. Identification of cellular and genetic drivers of breast cancer heterogeneity in genetically engineered mouse tumour models , 2014, The Journal of pathology.
[22] S. Swan,et al. Differences in ovarian hormones in relation to parity and time since last birth. , 2014, Fertility and sterility.
[23] H. Brinkhaus,et al. Early but not late pregnancy induces lifelong reductions in the proportion of mammary progesterone sensing cells and epithelial Wnt signaling , 2014, Breast Cancer Research.
[24] Piotr J. Balwierz,et al. Parity induces differentiation and reduces Wnt/Notch signaling ratio and proliferation potential of basal stem/progenitor cells isolated from mouse mammary epithelium , 2013, Breast Cancer Research.
[25] Wassim Raffoul,et al. Progesterone/RANKL Is a Major Regulatory Axis in the Human Breast , 2013, Science Translational Medicine.
[26] Heather Eliassen,et al. Menarche, menopause, and breast cancer risk: individual participant meta-analysis, including 118 964 women with breast cancer from 117 epidemiological studies , 2012, The Lancet. Oncology.
[27] J. Russo,et al. Defining the genomic signature of the parous breast , 2012, BMC Medical Genomics.
[28] C. Perou,et al. Defining the cellular precursors to human breast cancer , 2011, Proceedings of the National Academy of Sciences.
[29] E. Lander,et al. Genetic predisposition directs breast cancer phenotype by dictating progenitor cell fate. , 2011, Cell stem cell.
[30] A. Ashworth,et al. BRCA1 basal-like breast cancers originate from luminal epithelial progenitors and not from basal stem cells. , 2010, Cell stem cell.
[31] B. Vojtesek,et al. The pro-metastatic protein anterior gradient-2 predicts poor prognosis in tamoxifen-treated breast cancers , 2010, Oncogene.
[32] C. Brisken,et al. Two distinct mechanisms underlie progesterone-induced proliferation in the mammary gland , 2010, Proceedings of the National Academy of Sciences.
[33] S. Fox,et al. Aberrant luminal progenitors as the candidate target population for basal tumor development in BRCA1 mutation carriers , 2009, Nature Medicine.
[34] A. Ashworth,et al. Pregnancy and the risk of breast cancer. , 2007, Endocrine-related cancer.
[35] V. Beral,et al. Cancer incidence and mortality in relation to body mass index in the Million Women Study: cohort study , 2007, BMJ : British Medical Journal.
[36] J. Hanoune. Amphiregulin is an essential mediator of estrogen receptor a function in mammary gland development. , 2007 .
[37] Romayne A. Thompson,et al. Age-related lobular involution and risk of breast cancer. , 2006, Journal of the National Cancer Institute.
[38] M. Pike,et al. Reproductive factors and breast cancer risk according to joint estrogen and progesterone receptor status: a meta-analysis of epidemiological studies , 2006, Breast Cancer Research.
[39] T. Kodadek. Faculty Opinions recommendation of Estrogen receptor-alpha directs ordered, cyclical, and combinatorial recruitment of cofactors on a natural target promoter. , 2004 .
[40] Mark Garton,et al. Breast cancer and hormone-replacement therapy: the Million Women Study , 2003, The Lancet.
[41] J R Marshall,et al. Pooled analysis of prospective cohort studies on height, weight, and breast cancer risk. , 2000, American journal of epidemiology.
[42] A. Nardulli,et al. Sp1 binding sites and an estrogen response element half-site are involved in regulation of the human progesterone receptor A promoter. , 2000, Molecular endocrinology.
[43] F. Kittrell,et al. Murine mammary gland carcinogenesis is critically dependent on progesterone receptor function. , 1999, Cancer research.
[44] A. Howell,et al. Dissociation between steroid receptor expression and cell proliferation in the human breast. , 1997, Cancer research.
[45] Z. Shao,et al. The human myoepithelial cell is a natural tumor suppressor. , 1997, Clinical cancer research : an official journal of the American Association for Cancer Research.
[46] H. Adami,et al. Transient increase in the risk of breast cancer after giving birth. , 1994, The New England journal of medicine.
[47] K. McPherson,et al. Influence of menstrual cycle, parity and oral contraceptive use on steroid hormone receptors in normal breast. , 1992, British Journal of Cancer.
[48] P Chambon,et al. Two distinct estrogen‐regulated promoters generate transcripts encoding the two functionally different human progesterone receptor forms A and B. , 1990, The EMBO journal.
[49] Röbbe Wünschiers,et al. Computational Biology , 2013, Springer Berlin Heidelberg.
[50] K. Kliche,et al. Premenopausal Breast Cancer , 2012, Drugs.
[51] J. Russo,et al. Influence of age and parity on the development of the human breast , 2005, Breast Cancer Research and Treatment.