Calcium signaling of thyrocytes is modulated by TSH through calcium binding protein expression.
暂无分享,去创建一个
R. Paschke | M. Eszlinger | K. Krohn | G. Aust | D. Führer | M. Weick | S. Lorenz
[1] Lei Guo,et al. Cross-platform comparison of SYBR® Green real-time PCR with TaqMan PCR, microarrays and other gene expression measurement technologies evaluated in the MicroArray Quality Control (MAQC) study , 2008, BMC Genomics.
[2] P. Santisteban,et al. Gbetagamma dimers released in response to thyrotropin activate phosphoinositide 3-kinase and regulate gene expression in thyroid cells. , 2008, Molecular endocrinology.
[3] H. Seo,et al. Thyroid hormone activates adenosine 5'-monophosphate-activated protein kinase via intracellular calcium mobilization and activation of calcium/calmodulin-dependent protein kinase kinase-beta. , 2008, Molecular endocrinology.
[4] I. So,et al. Ghrelin enhances the proliferating effect of thyroid stimulating hormone in FRTL-5 thyroid cells , 2008, Molecular and Cellular Endocrinology.
[5] Kazuki Nakamura,et al. Effect of P2 receptor on the intracellular calcium increase by cancer cells in human umbilical vein endothelial cells , 2008, Naunyn-Schmiedeberg's Archives of Pharmacology.
[6] V. Detours,et al. Roles of hydrogen peroxide in thyroid physiology and disease. , 2007, The Journal of clinical endocrinology and metabolism.
[7] R. Paschke,et al. Mechanisms of Disease: hydrogen peroxide, DNA damage and mutagenesis in the development of thyroid tumors , 2007, Nature Clinical Practice Endocrinology &Metabolism.
[8] S. Tunaru,et al. Thyrocyte-specific Gq/G11 deficiency impairs thyroid function and prevents goiter development. , 2007, The Journal of clinical investigation.
[9] R. Paschke,et al. Iodine deficiency activates antioxidant genes and causes DNA damage in the thyroid gland of rats and mice. , 2007, Biochimica et biophysica acta.
[10] P. Hogan,et al. Dissecting ICRAC, a store-operated calcium current. , 2007, Trends in biochemical sciences.
[11] J. Dumont,et al. Thyrotropin stimulates the generation of inositol 1,4,5-trisphosphate in human thyroid cells. , 2006, The Journal of clinical endocrinology and metabolism.
[12] Mitsuhiko Ikura,et al. Genetic polymorphism and protein conformational plasticity in the calmodulin superfamily: Two ways to promote multifunctionality , 2006, Proceedings of the National Academy of Sciences of the United States of America.
[13] M. Zou,et al. S100A4 (Mts1) gene overexpression is associated with invasion and metastasis of papillary thyroid carcinoma , 2005, British Journal of Cancer.
[14] L. Puskás,et al. Gene profiling identifies genes specific for well-differentiated epithelial thyroid tumors. , 2005, Cellular and molecular biology.
[15] L. Coletto,et al. Exporting calcium from cells. , 2005, Cell calcium.
[16] J. Boucher,et al. Dual Oxidase-2 Has an Intrinsic Ca2+-dependent H2O2-generating Activity* , 2005, Journal of Biological Chemistry.
[17] I. Ekiel,et al. Metastasis-associated Protein S100A4 Induces Angiogenesis through Interaction with Annexin II and Accelerated Plasmin Formation* , 2005, Journal of Biological Chemistry.
[18] Ann-Charlotte Westerdahl,et al. Effects of sphingosine 1-phosphate on calcium signaling, proliferation and S1P2 receptor expression in PC Cl3 rat thyroid cells , 2005, Molecular and Cellular Endocrinology.
[19] A. Miyauchi,et al. S100A4 Expression Is an Early Event of Papillary Carcinoma of the Thyroid , 2005, Oncology.
[20] M. Zou,et al. Microarray analysis of metastasis-associated gene expression profiling in a murine model of thyroid carcinoma pulmonary metastasis: identification of S100A4 (Mts1) gene overexpression as a poor prognostic marker for thyroid carcinoma. , 2004, The Journal of clinical endocrinology and metabolism.
[21] C. Heizmann,et al. S100 proteins in mouse and man: from evolution to function and pathology (including an update of the nomenclature). , 2004, Biochemical and biophysical research communications.
[22] R. Paschke,et al. RGS 2 expression is regulated by TSH and inhibits TSH receptor signaling. , 2004, European journal of endocrinology.
[23] M. Grigorian,et al. Extracellular S100A4(mts1) stimulates invasive growth of mouse endothelial cells and modulates MMP-13 matrix metalloproteinase activity , 2004, Oncogene.
[24] F. Pacifico,et al. TSH/cAMP up-regulate sarco/endoplasmic reticulum Ca2+-ATPases expression and activity in PC Cl3 thyroid cells. , 2004, European journal of endocrinology.
[25] R. Paschke,et al. Biological activity of activating thyroid-stimulating hormone receptor mutants depends on the cellular context. , 2003, Endocrinology.
[26] Brad T. Sherman,et al. DAVID: Database for Annotation, Visualization, and Integrated Discovery , 2003, Genome Biology.
[27] A. Fusco,et al. Regulation of thyroid cell proliferation by TSH and other factors: a critical evaluation of in vitro models. , 2001, Endocrine reviews.
[28] J. Simon,et al. Mechanisms of P2 receptor‐evoked DNA synthesis in thyroid FRTL‐5 cells , 2001, Journal of cellular physiology.
[29] J. Mazat,et al. From calcium signaling to cell death: two conformations for the mitochondrial permeability transition pore. Switching from low- to high-conductance state. , 1998, Biochimica et biophysica acta.
[30] R. Paschke,et al. Somatic Mutations in the Thyrotropin Receptor Gene and Not in the Gsα Protein Gene in 31 Toxic Thyroid Nodules1 , 1997 .
[31] P. Rocmans,et al. Diversity and prevalence of somatic mutations in the thyrotropin receptor and Gs alpha genes as a cause of toxic thyroid adenomas. , 1997, The Journal of clinical endocrinology and metabolism.
[32] M. Sheppard,et al. Thyroid-stimulating hormone rapidly stimulates inositol polyphosphate formation in FRTL-5 thyrocytes without activating phosphoinositidase C. , 1996, The Biochemical journal.
[33] B. Dugué,et al. Purinergic agonist ATP is a comitogen in thyroid FRTL‐5 cells , 1996, Journal of cellular physiology.
[34] F. Cetani,et al. Differential effects of NaCl concentration on the constitutive activity of the thyrotropin and the luteinizing hormone/chorionic gonadotropin receptors , 1996, FEBS letters.
[35] M. Grigorian,et al. Non-muscle myosin heavy chain as a possible target for protein encoded by metastasis-related mts-1 gene. , 1994, The Journal of biological chemistry.
[36] R. Miller,et al. Calcium buffering properties of calbindin D28k and parvalbumin in rat sensory neurones. , 1993, The Journal of physiology.
[37] J. Parma,et al. Somatic mutations in the thyrotropin receptor gene cause hyperfunctioning thyroid adenomas , 1993, Nature.
[38] J. Parma,et al. The thyrotropin receptor and the regulation of thyrocyte function and growth. , 1992, Endocrine reviews.
[39] E. Laurent,et al. ATP, bradykinin, TRH and TSH activate the Ca2+-phosphatidylinositol cascade of human thyrocytes in primary culture , 1991, Molecular and Cellular Endocrinology.
[40] J. Lytton,et al. Thapsigargin inhibits the sarcoplasmic or endoplasmic reticulum Ca-ATPase family of calcium pumps. , 1991, The Journal of biological chemistry.
[41] M. Parmentier,et al. Molecular cloning of the thyrotropin receptor. , 1989, Science.
[42] T. Harden,et al. Agonist-induced desensitization of a P2Y-purinergic receptor-regulated phospholipase C. , 1989, Journal of Biological Chemistry.
[43] Koichi Sato,et al. P2‐purinergic agonists activate phospholipase C in a guanine nucleotide‐ and Ca2+‐dependent manner in FRTL‐5 thyroid cell membranes , 1989, FEBS letters.
[44] E. Rapaport. Experimental cancer therapy in mice by adenine nucleotides. , 1988, European journal of cancer & clinical oncology.
[45] H. Towbin,et al. Electrophoretic transfer of proteins from polyacrylamide gels to nitrocellulose sheets: procedure and some applications. , 1979, Proceedings of the National Academy of Sciences of the United States of America.
[46] U. K. Laemmli,et al. Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4 , 1970, Nature.
[47] Brad T. Sherman,et al. Systematic and integrative analysis of large gene lists using DAVID bioinformatics resources , 2008, Nature Protocols.
[48] X. Qian,et al. Immunohistochemical separation of follicular variant of papillary thyroid carcinoma from follicular adenoma , 2006, Endocrine pathology.
[49] Carine Maenhaut,et al. The phylogeny, ontogeny, anatomy and regulation of the iodine metabolizing thyroid , 2005 .
[50] R. Paschke,et al. Somatic mutations in the thyrotropin receptor gene and not in the Gs alpha protein gene in 31 toxic thyroid nodules. , 1997, The Journal of clinical endocrinology and metabolism.
[51] 木村 孝穂. Thyrotropin-induced hydrogen peroxide production in FRTL-5 thyroid cells is mediated not by adenosine 3',5'-monophosphate, but by Ca[2+] signaling followed by phospholipase-A2 activation and potentiated by an adenosine derivative , 1996 .
[52] J. Estrela,et al. Elimination of Ehrlich tumours by ATP-induced growth inhibition, glutathione depletion and X-rays , 1995, Nature Medicine.
[53] F. Okajima,et al. Thyrotropin-induced hydrogen peroxide production in FRTL-5 thyroid cells is mediated not by adenosine 3',5'-monophosphate, but by Ca2+ signaling followed by phospholipase-A2 activation and potentiated by an adenosine derivative. , 1995, Endocrinology.
[54] F. Delange,et al. The disorders induced by iodine deficiency. , 1994, Thyroid : official journal of the American Thyroid Association.
[55] S. Orrenius,et al. The calcium ion and cell death. , 1994, Journal of neural transmission. Supplementum.