TDAG51 deficiency attenuates dextran sulfate sodium-induced colitis in mice
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[1] J. Dou,et al. Elevated IL-35 level and iTr35 subset increase the bacterial burden and lung lesions in Mycobacterium tuberculosis-infected mice , 2022, Open life sciences.
[2] H. Uetake,et al. PHLDA1 expression in ulcerative colitis: A potential role in the management of dysplasia , 2021, Molecular and clinical oncology.
[3] F. Yang,et al. Loss of PHLDA1 has a protective role in OGD/R-injured neurons via regulation of the GSK-3β/Nrf2 pathway , 2021, Human & experimental toxicology.
[4] Zhongjun Wu,et al. miR-194 ameliorates hepatic ischemia/reperfusion injury via targeting PHLDA1 in a TRAF6-dependent manner. , 2021, International immunopharmacology.
[5] M. Takami,et al. Pax5 Negatively Regulates Osteoclastogenesis through Downregulation of Blimp1 , 2021, International journal of molecular sciences.
[6] Yongwon Choi,et al. T-Cell Death-Associated Gene 51 Is a Novel Negative Regulator of PPARγ That Inhibits PPARγ-RXRα Heterodimer Formation in Adipogenesis , 2020, Molecules and cells.
[7] G. Trinchieri,et al. FAM3D is essential for colon homeostasis and host defense against inflammation associated carcinogenesis , 2020, Nature Communications.
[8] Kaiping Wang,et al. Identification of Differential Intestinal Mucosa Transcriptomic Biomarkers for Ulcerative Colitis by Bioinformatics Analysis , 2020, Disease markers.
[9] Xuechu Zhen,et al. PHLDA1 promotes microglia-mediated neuroinflammation via regulating K63-linked ubiquitination of TRAF6 , 2020, Brain, Behavior, and Immunity.
[10] Yingxian Sun,et al. PHLDA1 is a new therapeutic target of oxidative stress and ischemia reperfusion-induced myocardial injury. , 2020, Life sciences.
[11] Yongwon Choi,et al. TDAG51 is a crucial regulator of maternal care and depressive-like behavior after parturition , 2019, PLoS genetics.
[12] S. Devaraj,et al. Anti-inflammatory activity of Alpinia officinarum hance on rat colon inflammation and tissue damage in DSS induced acute and chronic colitis models , 2018, Food Science and Human Wellness.
[13] H. Aburatani,et al. PHLDA1, another PHLDA family protein that inhibits Akt , 2018, Cancer science.
[14] Giovanni Monteleone,et al. Advances in understanding the role of cytokines in inflammatory bowel disease , 2018, Expert review of gastroenterology & hepatology.
[15] Xinrui Li,et al. Toll-like Receptors and Inflammatory Bowel Disease , 2018, Front. Immunol..
[16] M. Cho,et al. Immunological pathogenesis of inflammatory bowel disease , 2018, Intestinal research.
[17] J. Rho,et al. Generation of an osteoblast-based artificial niche that supports in vitro B lymphopoiesis , 2017, Experimental & Molecular Medicine.
[18] Q. Guan,et al. Recent Advances: The Imbalance of Cytokines in the Pathogenesis of Inflammatory Bowel Disease , 2017, Mediators of inflammation.
[19] Jie Fan,et al. PHLDA1 Promotes Lung Contusion by Regulating the Toll-Like Receptor 2 Signaling Pathway , 2016, Cellular Physiology and Biochemistry.
[20] Hyeon-Beom Seo,et al. Grim19 Attenuates DSS Induced Colitis in an Animal Model , 2016, PloS one.
[21] Mark S. Sundrud,et al. Cytokine Networks and T-Cell Subsets in Inflammatory Bowel Diseases , 2016, Inflammatory bowel diseases.
[22] M. Nagai. Pleckstrin homology-like domain, family A, member 1 (PHLDA1) and cancer. , 2016, Biomedical reports.
[23] L. Du,et al. Up-regulation of TDAG51 is a dependent factor of LPS-induced RAW264.7 macrophages proliferation and cell cycle progression , 2016, Immunopharmacology and immunotoxicology.
[24] S. Baek,et al. Regulation of PHLDA1 Expression by JAK2‐ERK1/2‐STAT3 Signaling Pathway , 2016, Journal of cellular biochemistry.
[25] A. Fanning,et al. Differential expression of key regulators of Toll‐like receptors in ulcerative colitis and Crohn's disease: a role for Tollip and peroxisome proliferator‐activated receptor gamma? , 2015, Clinical and experimental immunology.
[26] Jingbo Zhai,et al. IL-33 alleviates DSS-induced chronic colitis in C57BL/6 mice colon lamina propria by suppressing Th17 cell response as well as Th1 cell response. , 2015, International immunopharmacology.
[27] B. Coombes,et al. Convergence of External Crohn’s Disease Risk Factors on Intestinal Bacteria , 2015, Front. Immunol..
[28] W. Strober,et al. Experimental Models of Inflammatory Bowel Diseases , 2015, Cellular and molecular gastroenterology and hepatology.
[29] S. Moossavi,et al. Toll-like receptor expression in crypt epithelial cells, putative stem cells and intestinal myofibroblasts isolated from controls and patients with inflammatory bowel disease , 2014, Clinical and experimental immunology.
[30] C. Loddenkemper,et al. A guide to histomorphological evaluation of intestinal inflammation in mouse models. , 2014, International journal of clinical and experimental pathology.
[31] M. Coskun. Intestinal Epithelium in Inflammatory Bowel Disease , 2014, Front. Med..
[32] Neville E. Sanjana,et al. Improved vectors and genome-wide libraries for CRISPR screening , 2014, Nature Methods.
[33] Markus F. Neurath,et al. Cytokines in inflammatory bowel disease , 2014, Nature Reviews Immunology.
[34] B. Chassaing,et al. Dextran Sulfate Sodium (DSS)‐Induced Colitis in Mice , 2014, Current protocols in immunology.
[35] D. Merlin,et al. Dextran sodium sulfate inhibits the activities of both polymerase and reverse transcriptase: lithium chloride purification, a rapid and efficient technique to purify RNA , 2013, BMC Research Notes.
[36] Jong-Soon Choi,et al. TDAG51 deficiency promotes oxidative stress-induced apoptosis through the generation of reactive oxygen species in mouse embryonic fibroblasts , 2013, Experimental & Molecular Medicine.
[37] J. Capone,et al. Deficiency of TDAG51 Protects Against Atherosclerosis by Modulating Apoptosis, Cholesterol Efflux, and Peroxiredoxin‐1 Expression , 2013, Journal of the American Heart Association.
[38] S. Mani,et al. Protective effect of naringenin against experimental colitis via suppression of Toll-like receptor 4/NF-κB signalling , 2013, British Journal of Nutrition.
[39] Yu-Chen Hou,et al. Effects of alanyl-glutamine dipeptide on the expression of colon-inflammatory mediators during the recovery phase of colitis induced by dextran sulfate sodium , 2013, European Journal of Nutrition.
[40] R. Palanivel,et al. Loss of TDAG51 Results in Mature-Onset Obesity, Hepatic Steatosis, and Insulin Resistance by Regulating Lipogenesis , 2012, Diabetes.
[41] S. Lecleire,et al. Role of Toll Like Receptors in Irritable Bowel Syndrome: Differential Mucosal Immune Activation According to the Disease Subtype , 2012, PloS one.
[42] M. Perše,et al. Dextran Sodium Sulphate Colitis Mouse Model: Traps and Tricks , 2012, Journal of biomedicine & biotechnology.
[43] R. Jorissen,et al. PHLDA1 expression marks the putative epithelial stem cells and contributes to intestinal tumorigenesis. , 2011, Cancer research.
[44] S. Akira,et al. The role of pattern-recognition receptors in innate immunity: update on Toll-like receptors , 2010, Nature Immunology.
[45] A. Loukas,et al. Therapeutic potential of helminth soluble proteins in TNBS‐induced colitis in mice , 2009, Inflammatory bowel diseases.
[46] B. Necela,et al. Toll‐like receptor 4 mediates cross‐talk between peroxisome proliferator‐activated receptor γ and nuclear factor‐κB in macrophages , 2008, Immunology.
[47] Chundong Yu,et al. Establishment of a cell-based assay for examining the expression of tumor necrosis factor alpha (TNF-α) gene , 2008, Applied Microbiology and Biotechnology.
[48] W. Farrar,et al. A Role for PPARγ in the Regulation of Cytokines in Immune Cells and Cancer , 2008, PPAR research.
[49] M. Neurath,et al. NF‐κB in inflammatory bowel disease , 2008, Journal of internal medicine.
[50] H. Izu,et al. A novel HSF1‐mediated death pathway that is suppressed by heat shock proteins , 2006, The EMBO journal.
[51] Yasser El Miedany,et al. The Gastrointestinal Safety and Effect on Disease Activity of Etoricoxib, a Selective Cox-2 Inhibitor in Inflammatory Bowel Diseases , 2006, The American Journal of Gastroenterology.
[52] R. Xu,et al. Toll-like receptor-4 is required for intestinal response to epithelial injury and limiting bacterial translocation in a murine model of acute colitis. , 2005, American journal of physiology. Gastrointestinal and liver physiology.
[53] E. Falk,et al. TDAG51 Is Induced by Homocysteine, Promotes Detachment-mediated Programmed Cell Death, and Contributes to the Development of Atherosclerosis in Hyperhomocysteinemia* , 2003, Journal of Biological Chemistry.
[54] R. Cross,et al. Nitric Oxide in Inflammatory Bowel Disease , 2003, Inflammatory bowel diseases.
[55] J. Udagawa,et al. Strategic Compartmentalization of Toll-Like Receptor 4 in the Mouse Gut1 , 2003, The Journal of Immunology.
[56] C. Vinson,et al. CD40-Mediated Transcriptional Regulation of the IL-6 Gene in B Lymphocytes: Involvement of NF-κB, AP-1, and C/EBP1 , 2003, The Journal of Immunology.
[57] G. Rogler,et al. Toll-like receptors 2 and 4 are up-regulated during intestinal inflammation. , 2001, Gastroenterology.
[58] Yongwon Choi,et al. TDAG51 Is Not Essential for Fas/CD95 Regulation and Apoptosis In Vivo , 2001, Molecular and Cellular Biology.
[59] Richard Graham Knowles,et al. Suppression of acute experimental colitis by a highly selective inducible nitric-oxide synthase inhibitor, N-[3-(aminomethyl)benzyl]acetamidine. , 2001, The Journal of pharmacology and experimental therapeutics.
[60] I. Singer,et al. Cyclooxygenase 2 is induced in colonic epithelial cells in inflammatory bowel disease. , 1998, Gastroenterology.
[61] C. Park,et al. A novel gene product that couples TCR signaling to Fas(CD95) expression in activation-induced cell death. , 1996, Immunity.
[62] A. Ferguson,et al. Ulcerative colitis and Crohn's disease , 1994, BMJ.