TDAG51 deficiency attenuates dextran sulfate sodium-induced colitis in mice

[1]  J. Dou,et al.  Elevated IL-35 level and iTr35 subset increase the bacterial burden and lung lesions in Mycobacterium tuberculosis-infected mice , 2022, Open life sciences.

[2]  H. Uetake,et al.  PHLDA1 expression in ulcerative colitis: A potential role in the management of dysplasia , 2021, Molecular and clinical oncology.

[3]  F. Yang,et al.  Loss of PHLDA1 has a protective role in OGD/R-injured neurons via regulation of the GSK-3β/Nrf2 pathway , 2021, Human & experimental toxicology.

[4]  Zhongjun Wu,et al.  miR-194 ameliorates hepatic ischemia/reperfusion injury via targeting PHLDA1 in a TRAF6-dependent manner. , 2021, International immunopharmacology.

[5]  M. Takami,et al.  Pax5 Negatively Regulates Osteoclastogenesis through Downregulation of Blimp1 , 2021, International journal of molecular sciences.

[6]  Yongwon Choi,et al.  T-Cell Death-Associated Gene 51 Is a Novel Negative Regulator of PPARγ That Inhibits PPARγ-RXRα Heterodimer Formation in Adipogenesis , 2020, Molecules and cells.

[7]  G. Trinchieri,et al.  FAM3D is essential for colon homeostasis and host defense against inflammation associated carcinogenesis , 2020, Nature Communications.

[8]  Kaiping Wang,et al.  Identification of Differential Intestinal Mucosa Transcriptomic Biomarkers for Ulcerative Colitis by Bioinformatics Analysis , 2020, Disease markers.

[9]  Xuechu Zhen,et al.  PHLDA1 promotes microglia-mediated neuroinflammation via regulating K63-linked ubiquitination of TRAF6 , 2020, Brain, Behavior, and Immunity.

[10]  Yingxian Sun,et al.  PHLDA1 is a new therapeutic target of oxidative stress and ischemia reperfusion-induced myocardial injury. , 2020, Life sciences.

[11]  Yongwon Choi,et al.  TDAG51 is a crucial regulator of maternal care and depressive-like behavior after parturition , 2019, PLoS genetics.

[12]  S. Devaraj,et al.  Anti-inflammatory activity of Alpinia officinarum hance on rat colon inflammation and tissue damage in DSS induced acute and chronic colitis models , 2018, Food Science and Human Wellness.

[13]  H. Aburatani,et al.  PHLDA1, another PHLDA family protein that inhibits Akt , 2018, Cancer science.

[14]  Giovanni Monteleone,et al.  Advances in understanding the role of cytokines in inflammatory bowel disease , 2018, Expert review of gastroenterology & hepatology.

[15]  Xinrui Li,et al.  Toll-like Receptors and Inflammatory Bowel Disease , 2018, Front. Immunol..

[16]  M. Cho,et al.  Immunological pathogenesis of inflammatory bowel disease , 2018, Intestinal research.

[17]  J. Rho,et al.  Generation of an osteoblast-based artificial niche that supports in vitro B lymphopoiesis , 2017, Experimental & Molecular Medicine.

[18]  Q. Guan,et al.  Recent Advances: The Imbalance of Cytokines in the Pathogenesis of Inflammatory Bowel Disease , 2017, Mediators of inflammation.

[19]  Jie Fan,et al.  PHLDA1 Promotes Lung Contusion by Regulating the Toll-Like Receptor 2 Signaling Pathway , 2016, Cellular Physiology and Biochemistry.

[20]  Hyeon-Beom Seo,et al.  Grim19 Attenuates DSS Induced Colitis in an Animal Model , 2016, PloS one.

[21]  Mark S. Sundrud,et al.  Cytokine Networks and T-Cell Subsets in Inflammatory Bowel Diseases , 2016, Inflammatory bowel diseases.

[22]  M. Nagai Pleckstrin homology-like domain, family A, member 1 (PHLDA1) and cancer. , 2016, Biomedical reports.

[23]  L. Du,et al.  Up-regulation of TDAG51 is a dependent factor of LPS-induced RAW264.7 macrophages proliferation and cell cycle progression , 2016, Immunopharmacology and immunotoxicology.

[24]  S. Baek,et al.  Regulation of PHLDA1 Expression by JAK2‐ERK1/2‐STAT3 Signaling Pathway , 2016, Journal of cellular biochemistry.

[25]  A. Fanning,et al.  Differential expression of key regulators of Toll‐like receptors in ulcerative colitis and Crohn's disease: a role for Tollip and peroxisome proliferator‐activated receptor gamma? , 2015, Clinical and experimental immunology.

[26]  Jingbo Zhai,et al.  IL-33 alleviates DSS-induced chronic colitis in C57BL/6 mice colon lamina propria by suppressing Th17 cell response as well as Th1 cell response. , 2015, International immunopharmacology.

[27]  B. Coombes,et al.  Convergence of External Crohn’s Disease Risk Factors on Intestinal Bacteria , 2015, Front. Immunol..

[28]  W. Strober,et al.  Experimental Models of Inflammatory Bowel Diseases , 2015, Cellular and molecular gastroenterology and hepatology.

[29]  S. Moossavi,et al.  Toll-like receptor expression in crypt epithelial cells, putative stem cells and intestinal myofibroblasts isolated from controls and patients with inflammatory bowel disease , 2014, Clinical and experimental immunology.

[30]  C. Loddenkemper,et al.  A guide to histomorphological evaluation of intestinal inflammation in mouse models. , 2014, International journal of clinical and experimental pathology.

[31]  M. Coskun Intestinal Epithelium in Inflammatory Bowel Disease , 2014, Front. Med..

[32]  Neville E. Sanjana,et al.  Improved vectors and genome-wide libraries for CRISPR screening , 2014, Nature Methods.

[33]  Markus F. Neurath,et al.  Cytokines in inflammatory bowel disease , 2014, Nature Reviews Immunology.

[34]  B. Chassaing,et al.  Dextran Sulfate Sodium (DSS)‐Induced Colitis in Mice , 2014, Current protocols in immunology.

[35]  D. Merlin,et al.  Dextran sodium sulfate inhibits the activities of both polymerase and reverse transcriptase: lithium chloride purification, a rapid and efficient technique to purify RNA , 2013, BMC Research Notes.

[36]  Jong-Soon Choi,et al.  TDAG51 deficiency promotes oxidative stress-induced apoptosis through the generation of reactive oxygen species in mouse embryonic fibroblasts , 2013, Experimental & Molecular Medicine.

[37]  J. Capone,et al.  Deficiency of TDAG51 Protects Against Atherosclerosis by Modulating Apoptosis, Cholesterol Efflux, and Peroxiredoxin‐1 Expression , 2013, Journal of the American Heart Association.

[38]  S. Mani,et al.  Protective effect of naringenin against experimental colitis via suppression of Toll-like receptor 4/NF-κB signalling , 2013, British Journal of Nutrition.

[39]  Yu-Chen Hou,et al.  Effects of alanyl-glutamine dipeptide on the expression of colon-inflammatory mediators during the recovery phase of colitis induced by dextran sulfate sodium , 2013, European Journal of Nutrition.

[40]  R. Palanivel,et al.  Loss of TDAG51 Results in Mature-Onset Obesity, Hepatic Steatosis, and Insulin Resistance by Regulating Lipogenesis , 2012, Diabetes.

[41]  S. Lecleire,et al.  Role of Toll Like Receptors in Irritable Bowel Syndrome: Differential Mucosal Immune Activation According to the Disease Subtype , 2012, PloS one.

[42]  M. Perše,et al.  Dextran Sodium Sulphate Colitis Mouse Model: Traps and Tricks , 2012, Journal of biomedicine & biotechnology.

[43]  R. Jorissen,et al.  PHLDA1 expression marks the putative epithelial stem cells and contributes to intestinal tumorigenesis. , 2011, Cancer research.

[44]  S. Akira,et al.  The role of pattern-recognition receptors in innate immunity: update on Toll-like receptors , 2010, Nature Immunology.

[45]  A. Loukas,et al.  Therapeutic potential of helminth soluble proteins in TNBS‐induced colitis in mice , 2009, Inflammatory bowel diseases.

[46]  B. Necela,et al.  Toll‐like receptor 4 mediates cross‐talk between peroxisome proliferator‐activated receptor γ and nuclear factor‐κB in macrophages , 2008, Immunology.

[47]  Chundong Yu,et al.  Establishment of a cell-based assay for examining the expression of tumor necrosis factor alpha (TNF-α) gene , 2008, Applied Microbiology and Biotechnology.

[48]  W. Farrar,et al.  A Role for PPARγ in the Regulation of Cytokines in Immune Cells and Cancer , 2008, PPAR research.

[49]  M. Neurath,et al.  NF‐κB in inflammatory bowel disease , 2008, Journal of internal medicine.

[50]  H. Izu,et al.  A novel HSF1‐mediated death pathway that is suppressed by heat shock proteins , 2006, The EMBO journal.

[51]  Yasser El Miedany,et al.  The Gastrointestinal Safety and Effect on Disease Activity of Etoricoxib, a Selective Cox-2 Inhibitor in Inflammatory Bowel Diseases , 2006, The American Journal of Gastroenterology.

[52]  R. Xu,et al.  Toll-like receptor-4 is required for intestinal response to epithelial injury and limiting bacterial translocation in a murine model of acute colitis. , 2005, American journal of physiology. Gastrointestinal and liver physiology.

[53]  E. Falk,et al.  TDAG51 Is Induced by Homocysteine, Promotes Detachment-mediated Programmed Cell Death, and Contributes to the Development of Atherosclerosis in Hyperhomocysteinemia* , 2003, Journal of Biological Chemistry.

[54]  R. Cross,et al.  Nitric Oxide in Inflammatory Bowel Disease , 2003, Inflammatory bowel diseases.

[55]  J. Udagawa,et al.  Strategic Compartmentalization of Toll-Like Receptor 4 in the Mouse Gut1 , 2003, The Journal of Immunology.

[56]  C. Vinson,et al.  CD40-Mediated Transcriptional Regulation of the IL-6 Gene in B Lymphocytes: Involvement of NF-κB, AP-1, and C/EBP1 , 2003, The Journal of Immunology.

[57]  G. Rogler,et al.  Toll-like receptors 2 and 4 are up-regulated during intestinal inflammation. , 2001, Gastroenterology.

[58]  Yongwon Choi,et al.  TDAG51 Is Not Essential for Fas/CD95 Regulation and Apoptosis In Vivo , 2001, Molecular and Cellular Biology.

[59]  Richard Graham Knowles,et al.  Suppression of acute experimental colitis by a highly selective inducible nitric-oxide synthase inhibitor, N-[3-(aminomethyl)benzyl]acetamidine. , 2001, The Journal of pharmacology and experimental therapeutics.

[60]  I. Singer,et al.  Cyclooxygenase 2 is induced in colonic epithelial cells in inflammatory bowel disease. , 1998, Gastroenterology.

[61]  C. Park,et al.  A novel gene product that couples TCR signaling to Fas(CD95) expression in activation-induced cell death. , 1996, Immunity.

[62]  A. Ferguson,et al.  Ulcerative colitis and Crohn's disease , 1994, BMJ.