Distinct striatum pathways connected to salience network predict symptoms improvement and resilient functioning in schizophrenia following risperidone monotherapy
暂无分享,去创建一个
Huafu Chen | W. Liao | Xujun Duan | Xiaogang Chen | Qian Cui | Shaoqiang Han | Rong Li | Yangyang Yu | Mi Yang | X. Zong | F. Xin | Maolin Hu | B. Becker | Mi Yang
[1] M. Menon,et al. Aberrant modulation of brain activation by emotional valence during self-referential processing among patients with delusions of reference. , 2017, Journal of behavior therapy and experimental psychiatry.
[2] Valentin Riedl,et al. Changes in extra-striatal functional connectivity in patients with schizophrenia in a psychotic episode , 2017, British Journal of Psychiatry.
[3] J. Downar,et al. Cortico-Striatal-Thalamic Loop Circuits of the Salience Network: A Central Pathway in Psychiatric Disease and Treatment , 2016, Front. Syst. Neurosci..
[4] J. Mann,et al. Short-term Effects of Risperidone Monotherapy on Spontaneous Brain Activity in First-episode Treatment-naïve Schizophrenia Patients: A Longitudinal fMRI Study , 2016, Scientific Reports.
[5] J. Mann,et al. Risperidone-induced topological alterations of anatomical brain network in first-episode drug-naive schizophrenia patients: a longitudinal diffusion tensor imaging study , 2016, Psychological Medicine.
[6] Andrew Zalesky,et al. Delayed Development of Brain Connectivity in Adolescents With Schizophrenia and Their Unaffected Siblings. , 2015, JAMA psychiatry.
[7] A. Malhotra,et al. Baseline Striatal Functional Connectivity as a Predictor of Response to Antipsychotic Drug Treatment. , 2015, The American journal of psychiatry.
[8] L. Uddin. Salience processing and insular cortical function and dysfunction , 2014, Nature Reviews Neuroscience.
[9] Jimmy Lee,et al. Altered striatal functional connectivity in subjects with an at-risk mental state for psychosis. , 2014, Schizophrenia bulletin.
[10] Michael W. Cole,et al. Altered global brain signal in schizophrenia , 2014, Proceedings of the National Academy of Sciences.
[11] Peter B. Jones,et al. Functional dysconnectivity of corticostriatal circuitry as a risk phenotype for psychosis. , 2013, JAMA psychiatry.
[12] Melissa J. Green,et al. Task-related fronto-striatal functional connectivity during working memory performance in schizophrenia , 2013, Schizophrenia Research.
[13] Dimitris Mavridis,et al. Comparative efficacy and tolerability of 15 antipsychotic drugs in schizophrenia: a multiple-treatments meta-analysis , 2013, The Lancet.
[14] W. Honer,et al. Effects of eight weeks of atypical antipsychotic treatment on middle frontal thickness in drug-naïve first-episode psychosis patients , 2013, Schizophrenia Research.
[15] Marc Joliot,et al. Links among resting-state default-mode network, salience network, and symptomatology in schizophrenia , 2013, Schizophrenia Research.
[16] S. Kapur,et al. Alterations of the Brain Reward System in Antipsychotic Naïve Schizophrenia Patients , 2012, Biological Psychiatry.
[17] P. Szeszko,et al. Magnetic resonance imaging predictors of treatment response in first-episode schizophrenia. , 2012, Schizophrenia bulletin.
[18] O. Grimm,et al. Reduced striatal activation during reward anticipation due to appetite-provoking cues in chronic schizophrenia: A fMRI study , 2012, Schizophrenia Research.
[19] T. Eichele,et al. Increased Intrinsic Brain Activity in the Striatum Reflects Symptom Dimensions in Schizophrenia , 2012, Schizophrenia bulletin.
[20] H. Critchley,et al. An Interoceptive Predictive Coding Model of Conscious Presence , 2011, Front. Psychology.
[21] E. Bullmore,et al. Disrupted Axonal Fiber Connectivity in Schizophrenia , 2011, Biological Psychiatry.
[22] V. Menon,et al. Saliency, switching, attention and control: a network model of insula function , 2010, Brain Structure and Function.
[23] A. Heinz,et al. Dopaminergic dysfunction in schizophrenia: salience attribution revisited. , 2010, Schizophrenia bulletin.
[24] H. Critchley,et al. A common role of insula in feelings, empathy and uncertainty , 2009, Trends in Cognitive Sciences.
[25] S. Kapur,et al. The dopamine hypothesis of schizophrenia: version III--the final common pathway. , 2009, Schizophrenia bulletin.
[26] Kevin Murphy,et al. The impact of global signal regression on resting state correlations: Are anti-correlated networks introduced? , 2009, NeuroImage.
[27] B. Biswal,et al. Functional connectivity of human striatum: a resting state FMRI study. , 2008, Cerebral cortex.
[28] V. Menon,et al. A critical role for the right fronto-insular cortex in switching between central-executive and default-mode networks , 2008, Proceedings of the National Academy of Sciences.
[29] Peter Williamson,et al. Are anticorrelated networks in the brain relevant to schizophrenia? , 2007, Schizophrenia bulletin.
[30] P. Fletcher,et al. From prediction error to psychosis: ketamine as a pharmacological model of delusions , 2007, Journal of psychopharmacology.
[31] Brian Knutson,et al. Dysfunction of ventral striatal reward prediction in schizophrenic patients treated with typical, not atypical, neuroleptics , 2006, Psychopharmacology.
[32] S. Kapur,et al. From dopamine to salience to psychosis—linking biology, pharmacology and phenomenology of psychosis , 2005, Schizophrenia Research.
[33] S. Kapur,et al. Half a century of antipsychotics and still a central role for dopamine D2 receptors , 2003, Progress in Neuro-Psychopharmacology and Biological Psychiatry.
[34] R. Conley,et al. Management of treatment resistance in schizophrenia , 2001, Biological Psychiatry.
[35] J. Pekar,et al. A method for making group inferences from functional MRI data using independent component analysis , 2001, Human brain mapping.
[36] E. Bullmore,et al. Differences in frontal cortical activation by a working memory task after substitution of risperidone for typical antipsychotic drugs in patients with schizophrenia. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[37] T. Robbins,et al. Frontal-striatal cognitive deficits in patients with chronic schizophrenia. , 1997, Brain : a journal of neurology.
[38] P. Seeman,et al. Antipsychotic drugs: direct correlation between clinical potency and presynaptic action on dopamine neurons. , 1975, Science.
[39] A. Malhotra,et al. Antipsychotic treatment and functional connectivity of the striatum in first-episode schizophrenia. , 2015, JAMA psychiatry.
[40] P. Liddle,et al. Does the salience network play a cardinal role in psychosis? An emerging hypothesis of insular dysfunction. , 2012, Journal of psychiatry & neuroscience : JPN.
[41] R. Buckner,et al. The organization of the human striatum estimated by intrinsic functional connectivity. , 2012, Journal of neurophysiology.
[42] P. McGuire,et al. Abnormal prefrontal activation directly related to pre-synaptic striatal dopamine dysfunction in people at clinical high risk for psychosis , 2011, Molecular Psychiatry.
[43] Marisa O. Hollinshead,et al. The organization of the human cerebral cortex estimated by intrinsic functional connectivity , 2011 .
[44] H. Holcomb,et al. Clozapine but not Haloperidol Re-establishes Normal Task-Activated rCBF Patterns in Schizophrenia within the Anterior Cingulate Cortex , 2004, Neuropsychopharmacology.
[45] S. Kapur. Psychosis as a state of aberrant salience: a framework linking biology, phenomenology, and pharmacology in schizophrenia. , 2003, The American journal of psychiatry.
[46] S. Tanada,et al. Decreased dopamine D2 receptor binding in the anterior cingulate cortex in schizophrenia. , 2002, Archives of general psychiatry.
[47] Karl J. Friston,et al. Schizophrenia: a disconnection syndrome? , 1995, Clinical neuroscience.