Thin-walled microvessels in human coronary atherosclerotic plaques show incomplete endothelial junctions relevance of compromised structural integrity for intraplaque microvascular leakage.
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Renu Virmani | R. Virmani | F. Kolodgie | M. Daemen | V. V. van Hinsbergh | A. Bijnens | H. Duimel | P. Frederik | Frank D Kolodgie | Mat J A P Daemen | Erica Pacheco | J. Sluimer | K. Maxfield | Bob Kutys | Victor W M van Hinsbergh | Ann P J J Bijnens | Judith C Sluimer | Kimberly Maxfield | Erica Pacheco | Bob Kutys | Hans Duimel | Peter M Frederik
[1] R. Reneman,et al. Local blood pressure rather than shear stress should be blamed for plaque rupture. , 2008, Journal of the American College of Cardiology.
[2] Mathijs Groeneweg,et al. Hypoxia, hypoxia-inducible transcription factor, and macrophages in human atherosclerotic plaques are correlated with intraplaque angiogenesis. , 2008, Journal of the American College of Cardiology.
[3] F. Meyer,et al. Placenta growth factor expression in human atherosclerotic carotid plaques is related to plaque destabilization. , 2008, Atherosclerosis.
[4] Amir Lerman,et al. The dynamic vasa vasorum. , 2007, Cardiovascular research.
[5] Stavros J. Baloyannis. Pathological alterations of the climbing fibres of the cerebellum in vascular dementia: A Golgi and electron microscope study , 2007, Journal of the Neurological Sciences.
[6] E. Dejana,et al. The role of junctional adhesion molecules in vascular inflammation , 2007, Nature Reviews Immunology.
[7] A. Zernecke,et al. Perivascular Mast Cells Promote Atherogenesis and Induce Plaque Destabilization in Apolipoprotein E–Deficient Mice , 2007, Circulation.
[8] A. Heagerty,et al. Hepatocyte growth factor and c‐Met expression in pericytes: implications for atherosclerotic plaque development , 2007, The Journal of pathology.
[9] A. Naylor,et al. Carotid plaque instability and ischemic symptoms are linked to immaturity of microvessels within plaques. , 2007, Journal of vascular surgery.
[10] L. Poston,et al. The oestrogen receptor β contributes to sex related differences in endothelial function of murine small arteries via EDHF , 2006, The Journal of physiology.
[11] R. Virmani,et al. Lipoprotein-Associated Phospholipase A2 Protein Expression in the Natural Progression of Human Coronary Atherosclerosis , 2006, Arteriosclerosis, thrombosis, and vascular biology.
[12] M. C. Epistolato,et al. Biological features (inflammation and neoangiogenesis) and atherosclerotic risk factors in carotid plaques and calcified aortic valve stenosis: two different sites of the same disease? , 2006, American journal of clinical pathology.
[13] S. Kuruvilla,et al. Images in cardiovascular medicine. Role of inflammation in atherosclerosis: immunohistochemical and electron microscopic images of a coronary endarterectomy specimen. , 2006, Circulation.
[14] J. Jukema,et al. Lesional Overexpression of Matrix Metalloproteinase-9 Promotes Intraplaque Hemorrhage in Advanced Lesions But Not at Earlier Stages of Atherogenesis , 2005, Arteriosclerosis, thrombosis, and vascular biology.
[15] Aloke V. Finn,et al. Atherosclerotic Plaque Progression and Vulnerability to Rupture: Angiogenesis as a Source of Intraplaque Hemorrhage , 2005, Arteriosclerosis, thrombosis, and vascular biology.
[16] Michael Simons,et al. Role of Angiogenesis in Cardiovascular Disease : a Critical Appraisal , 2022 .
[17] A. Luttun,et al. Placental Growth Factor Promotes Atherosclerotic Intimal Thickening and Macrophage Accumulation , 2005, Circulation.
[18] Gieri Cathomas,et al. Arterial Neovascularization and Inflammation in Vulnerable Patients: Early and Late Signs of Symptomatic Atherosclerosis , 2004, Circulation.
[19] Juan J. Badimon,et al. Plaque Neovascularization Is Increased in Ruptured Atherosclerotic Lesions of Human Aorta: Implications for Plaque Vulnerability , 2004, Circulation.
[20] A. Sajantila,et al. Mast Cells in Neovascularized Human Coronary Plaques Store and Secrete Basic Fibroblast Growth Factor, a Potent Angiogenic Mediator , 2004, Arteriosclerosis, thrombosis, and vascular biology.
[21] Elisabetta Dejana,et al. Endothelial cell–cell junctions: happy together , 2004, Nature Reviews Molecular Cell Biology.
[22] Renu Virmani,et al. Intraplaque hemorrhage and progression of coronary atheroma. , 2003, The New England journal of medicine.
[23] Holger Gerhardt,et al. Endothelial-pericyte interactions in angiogenesis , 2003, Cell and Tissue Research.
[24] Rakesh K Jain,et al. Molecular regulation of vessel maturation , 2003, Nature Medicine.
[25] Mark M. Kockx,et al. Phagocytosis and Macrophage Activation Associated With Hemorrhagic Microvessels in Human Atherosclerosis , 2003, Arteriosclerosis, thrombosis, and vascular biology.
[26] U. Schlötzer-Schrehardt,et al. Pericyte recruitment in human corneal angiogenesis: an ultrastructural study with clinicopathological correlation , 2003, The British journal of ophthalmology.
[27] G. Amerongen,et al. Intracellular signalling involved in modulating human endothelial barrier function , 2002 .
[28] V. V. van Hinsbergh,et al. Intracellular signalling involved in modulating human endothelial barrier function , 2002, Journal of anatomy.
[29] C. Betsholtz,et al. Analysis of Mural Cell Recruitment to Tumor Vessels , 2002, Circulation.
[30] Holger Gerhardt,et al. Lack of Pericytes Leads to Endothelial Hyperplasia and Abnormal Vascular Morphogenesis , 2001, The Journal of cell biology.
[31] K. Ballmer-Hofer,et al. VEGF transiently disrupts gap junctional communication in endothelial cells. , 2001, Journal of cell science.
[32] P. Carmeliet,et al. Angiogenesis in cancer and other diseases , 2000, Nature.
[33] O. Tricot,et al. Relation between endothelial cell apoptosis and blood flow direction in human atherosclerotic plaques. , 2000, Circulation.
[34] R. Virmani,et al. Lessons from sudden coronary death: a comprehensive morphological classification scheme for atherosclerotic lesions. , 2000, Arteriosclerosis, thrombosis, and vascular biology.
[35] R K Jain,et al. Openings between defective endothelial cells explain tumor vessel leakiness. , 2000, The American journal of pathology.
[36] J. Roelandt,et al. Images in Cardiovascular Medicine , 2000 .
[37] Y. Bobryshev,et al. Neovascular expression of VE-cadherin in human atherosclerotic arteries and its relation to intimal inflammation. , 1999, Cardiovascular research.
[38] D. McDonald,et al. Endothelial Gaps as Sites for Plasma Leakage in Inflammation , 1999, Microcirculation.
[39] R. Virmani,et al. When neoangiogenesis ricochets. , 1998, American heart journal.
[40] R. Stocker,et al. Presence of hypochlorite-modified proteins in human atherosclerotic lesions. , 1996, The Journal of clinical investigation.
[41] N. Simionescu,et al. Pathobiochemistry of combined diabetes and atherosclerosis studied on a novel animal model. The hyperlipemic-hyperglycemic hamster. , 1996, The American journal of pathology.
[42] C. Alpers,et al. Neovascular expression of E-selectin, intercellular adhesion molecule-1, and vascular cell adhesion molecule-1 in human atherosclerosis and their relation to intimal leukocyte content. , 1996, Circulation.
[43] K. Sueishi,et al. Intimal neovascularization in human coronary atherosclerosis: its origin and pathophysiological significance. , 1995, Human pathology.