TNFα reduces glutamate induced intracellular Ca2+ increase in cultured cortical astrocytes
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[1] G. Stoll,et al. Time course of inwardly rectifying K+ current reduction in glial cells surrounding ischemic brain lesions , 2000, Brain Research.
[2] W. Walz. Role of astrocytes in the clearance of excess extracellular potassium , 2000, Neurochemistry International.
[3] C. Steinhäuser,et al. Ion channels in glial cells , 2000, Brain Research Reviews.
[4] C. Jahr,et al. Glial Contribution to Glutamate Uptake at Schaffer Collateral–Commissural Synapses in the Hippocampus , 1998, The Journal of Neuroscience.
[5] G. Stoll,et al. TNFα induces a protein kinase C‐dependent reduction in astroglial K+ conductance , 1998 .
[6] W Reichelt,et al. Ionic mechanisms in glutamate‐induced astrocyte swelling: Role of K+ influx , 1998, Journal of neuroscience research.
[7] H. Kimelberg,et al. Glutamate receptor‐mediated calcium responses in acutely isolated hippocampal astrocytes , 1997, Glia.
[8] K. H. Backus,et al. Intracellular Calcium Transients and Potassium Current Oscillations Evoked by Glutamate in Cultured Rat Astrocytes , 1997, The Journal of Neuroscience.
[9] H. Köller. TNFα in cerebrospinal fluid of meningitis patients reduces astrocytes membrane potential , 1997, Journal of Neuroimmunology.
[10] H. Hartung,et al. IMMUNOLOGICALLY INDUCED ELECTROPHYSIOLOGICAL DYSFUNCTION: IMPLICATIONS FOR INFLAMMATORY DISEASES OF THE CNS AND PNS , 1997, Progress in Neurobiology.
[11] H. Köller,et al. Tumour necrosis factor-alpha increases intracellular Ca2+ and induces a depolarization in cultured astroglial cells. , 1996, Brain : a journal of neurology.
[12] H. Sontheimer,et al. Cytokine modulation of glial glutamate uptake: a possble involvement of nitric oxide , 1996, Neuroreport.
[13] C. Zorumski,et al. Components of glial responses to exogenous and synaptic glutamate in rat hippocampal microcultures , 1996, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[14] M. Mattson,et al. Brain injury and tumor necrosis factors induce calbindin D‐28K in astrocytes: Evidence for a cytoprotective response , 1995, Journal of neuroscience research.
[15] M. Ballestas,et al. Interleukin 1‐β‐ and tumor necrosis factor‐α‐mediated regulation of ICAM‐1 gene expression in astrocytes requires protein kinase C activity , 1995 .
[16] C. Raine,et al. The adhesion molecule and cytokine profile of multiple sclerosis lesions , 1995, Annals of neurology.
[17] H. Hartung,et al. Antigen presentation by astrocytes primes rat T-lymphocytes for apoptotic cell death , 1994, Journal of Neuroimmunology.
[18] Y. Agid,et al. Induction of calbindin‐D 28K gene and protein expression by physiological stimuli but not in calcium‐mediated degeneration in rat PC12 pheochromocytoma cells , 1994, FEBS letters.
[19] Fang Liu,et al. Glutamate-mediated astrocyte–neuron signalling , 1994, Nature.
[20] J. Holzwarth,et al. Glutamate receptor agonists stimulate diverse calcium responses in different types of cultured rat cortical glial cells , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[21] M. Nedergaard,et al. Direct signaling from astrocytes to neurons in cultures of mammalian brain cells. , 1994, Science.
[22] Steven Mennerick,et al. Glial contributions to excitatory neurotransmission in cultured hippocampal cells , 1994, Nature.
[23] V Balasingam,et al. Reactive astrogliosis in the neonatal mouse brain and its modulation by cytokines , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[24] E. Benveniste,et al. Signal transduction pathways mediating astrocyte IL-6 induction by IL-1 beta and tumor necrosis factor-alpha. , 1994, Journal of immunology.
[25] O. Selnes,et al. Clinical‐neuropathologic correlation in HIV‐associated dementia , 1993, Neurology.
[26] P. Kragsbjerg,et al. Tumor Necrosis Factor-α (TNFα) in Cerebrospinal Fluid from Patients with Meningitis of Different Etiologies: High Levels of TNFα Indicate Bacterial Meningitis , 1993 .
[27] G. Westbrook,et al. The time course of glutamate in the synaptic cleft. , 1992, Science.
[28] K. Frei,et al. Production of macrophage colony-stimulating factor by astrocytes and brain macrophages , 1992, Journal of Neuroimmunology.
[29] E. Thompson,et al. Blood-Brain Barrier Damage in Patients with Bacterial Meningitis: Association with Tumor Necrosis Factor-α but not Interleukin-Iβ , 1992 .
[30] Stephen J. Smith,et al. Neuronal activity triggers calcium waves in hippocampal astrocyte networks , 1992, Neuron.
[31] R. Ransohoff,et al. Human astrocytes proliferate in response to tumor necrosis factor alpha , 1990, Journal of Neuroimmunology.
[32] E. Benveniste,et al. Induction and regulation of interleukin-6 gene expression in rat astrocytes , 1990, Journal of Neuroimmunology.
[33] M. Caplan,et al. Cerebrospinal fluid cachectin/tumor necrosis factor-alpha and platelet-activating factor concentrations and severity of bacterial meningitis in children. , 1990, The Journal of infectious diseases.
[34] D. Faber,et al. Glutamate stimulates release of Ca2+ from internal stores in astroglia , 1990, Brain Research.
[35] A. Jensen,et al. Fluorescence measurement of changes in intracellular calcium induced by excitatory amino acids in cultured cortical astrocytes , 1990, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[36] H. Hartung,et al. Recombinant interleukin-1β stimulates eicosanoid production in rat primary culture astrocytes , 1989, Brain Research.
[37] H. Hartung,et al. Leukotriene production by cultured astroglial cells , 1987, Brain Research.
[38] H. Kimelberg,et al. Excitatory amino acids directly depolarize rat brain astrocytes in primary culture , 1984, Nature.
[39] W. Fierz,et al. Astrocytes present myelin basic protein to encephalitogenic T-cell lines , 1984, Nature.
[40] A. Verkhratsky,et al. Glial calcium: homeostasis and signaling function. , 1998, Physiological reviews.
[41] S. Perry,et al. Tumor Necrosis Factor (cid:97) Inhibits Glutamate Uptake by Primary Human Astrocytes FOR PATHOGENESIS OF HIV-1 , 2022 .