Pathophysiology of sepsis and multiple organ failure: pro- versus anti-inflammatory aspects.

[1]  C. Hack,et al.  Time course of cytokine levels in sepsis , 1995, Intensive Care Medicine.

[2]  E. Lien,et al.  Toll-like receptors. , 2002, Critical care medicine.

[3]  M. Pinsky,et al.  Suppression of cytokine‐mediated β2‐integrin activation on circulating neutrophils in critically ill patients , 1999, Journal of leukocyte biology.

[4]  C. Terregino,et al.  Impaired neutrophil adherence as an early marker of systemic inflammatory response syndrome and severe sepsis. , 1997, Annals of emergency medicine.

[5]  A. Malugin,et al.  Nitric oxide donor induces HSP70 accumulation in the heart and in cultured cells , 1996, FEBS letters.

[6]  T. Hennet,et al.  TNF alpha alters mitochondrial membrane potential in L929 but not in TNF alpha-resistant L929.12 cells: relationship with the expression of stress proteins, annexin 1 and superoxide dismutase activity. , 1996, Free radical research.

[7]  J. Christman,et al.  Sepsis and cytokines: current status. , 1996, British journal of anaesthesia.

[8]  G. Schlag,et al.  Mediators of Injury and Inflammation , 1996, World Journal of Surgery.

[9]  T. Takakuwa,et al.  Changes in adhesion molecule levels in sepsis. , 1996, Research communications in molecular pathology and pharmacology.

[10]  R. Bone,et al.  Toward a theory regarding the pathogenesis of the systemic inflammatory response syndrome: what we do and do not know about cytokine regulation. , 1996, Critical care medicine.

[11]  B. Crestani,et al.  Compartmentalized IL-8 and elastase release within the human lung in unilateral pneumonia. , 1996, American journal of respiratory and critical care medicine.

[12]  J. Cavaillon.  The nonspecific nature of endotoxin tolerance. , 1995, Trends in microbiology.

[13]  G. Ramsay,et al.  Natural cytokine antagonists and endogenous antiendotoxin core antibodies in sepsis syndrome. The Sepsis Intervention Group. , 1995, JAMA.

[14]  J. Bryant,et al.  Leukocyte activation in the peripheral blood of patients with cirrhosis of the liver and SIRS. Correlation with serum interleukin-6 levels and organ dysfunction. , 1995, JAMA.

[15]  H. Kolb,et al.  Heat shock induces resistance in rat pancreatic islet cells against nitric oxide, oxygen radicals and streptozotocin toxicity in vitro. , 1995, The Journal of clinical investigation.

[16]  J. Haas,et al.  Tolerance to lipopolysaccharide involves mobilization of nuclear factor kappa B with predominance of p50 homodimers. , 1994, The Journal of biological chemistry.

[17]  A S Slutsky,et al.  Induction of the heat shock response reduces mortality rate and organ damage in a sepsis‐induced acute lung injury model , 1994, Critical care medicine.

[18]  K. Asadullah,et al.  Improvement of Monocyte Function — A New Therapeutic Approach? , 1994 .

[19]  E. Rackow,et al.  Altered leukocyte immunophenotypes in septic shock. Studies of HLA-DR, CD11b, CD14, and IL-2R expression. , 1993, Chest.

[20]  C. McCall,et al.  Tolerance to endotoxin-induced expression of the interleukin-1 beta gene in blood neutrophils of humans with the sepsis syndrome. , 1993, The Journal of clinical investigation.

[21]  J. Vincent,et al.  Serum cytokine levels in human septic shock. Relation to multiple-system organ failure and mortality. , 1993, Chest.

[22]  M. Diamond,et al.  A subpopulation of Mac-1 (CD11b/CD18) molecules mediates neutrophil adhesion to ICAM-1 and fibrinogen , 1993, The Journal of cell biology.

[23]  M. Jäättelä,et al.  Heat-shock proteins protect cells from monocyte cytotoxicity: possible mechanism of self-protection , 1993, The Journal of experimental medicine.

[24]  F. Trautinger,et al.  Respiratory Burst Capability of Polymorphonuclear Neutrophils and TNF‐α Serum Levels in Relationship to the Development of Septic Syndrome in Critically III Patients , 1991, Journal of leukocyte biology.