Elevated glycolytic metabolism of monocytes limits the generation of HIF-1α-driven migratory dendritic cells in tuberculosis
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O. Neyrolles | M. Vermeulen | L. Balboa | M. Sasiain | Sandra Inwentarz | D. Palmero | J. L. Marín Franco | Melanie Genoula | Lorena M. Ciallella | R. Musella | R. Argüello | M. B. Vecchione | M. Joly | F. Fuentes | Sarah C. Monard | Mariano Maio | M. Quiroga | P. G. Montaner | C. Vérollet | Z. Vahlas | Virginia González Polo | Joaquina Barros | Geanncarlo Lugo Villarino
[1] L. Butterfield,et al. Distinct metabolic states guide maturation of inflammatory and tolerogenic dendritic cells , 2022, Nature Communications.
[2] J. Philips,et al. Immune evasion and provocation by Mycobacterium tuberculosis , 2022, Nature Reviews Microbiology.
[3] D. Finlay,et al. Dendritic Cells metabolism - a strategic path to improve antitumoral DC vaccination. , 2022, Clinical and experimental immunology.
[4] Ping-Chih Ho,et al. Metabolic programming in dendritic cells tailors immune responses and homeostasis , 2021, Cellular & Molecular Immunology.
[5] M. Thangaraju,et al. Lactate-Dependent Regulation of Immune Responses by Dendritic Cells and Macrophages , 2021, Frontiers in Immunology.
[6] Xuetao Cao,et al. Dendritic cell migration in inflammation and immunity , 2021, Cellular & Molecular Immunology.
[7] D. Figarella-Branger,et al. SCENITH: A Flow Cytometry-Based Method to Functionally Profile Energy Metabolism with Single-Cell Resolution. , 2020, Cell metabolism.
[8] A. Fernie,et al. Cytoskeleton Architecture Regulates Glycolysis Coupling Cellular Metabolism to Mechanical Cues. , 2020, Trends in biochemical sciences.
[9] J. Pedraza-Chaverri,et al. Host-derived lipids from tuberculous pleurisy impair macrophage microbicidal-associated metabolic activity , 2020, bioRxiv.
[10] R. Deberardinis,et al. Mechanical regulation of glycolysis via cytoskeleton architecture , 2020, Nature.
[11] K. Moore,et al. Mycobacterium tuberculosis Limits Host Glycolysis and IL-1β by Restriction of PFK-M via MicroRNA-21. , 2020, Cell reports.
[12] I. D. de Vries,et al. Dendritic Cells Require PINK1-Mediated Phosphorylation of BCKDE1α to Promote Fatty Acid Oxidation for Immune Function , 2019, Front. Immunol..
[13] Sanjay Tyagi,et al. Immunometabolism of Phagocytes During Mycobacterium tuberculosis Infection , 2019, Front. Mol. Biosci..
[14] Xuetao Cao,et al. CCR7 Chemokine Receptor‐Inducible lnc‐Dpf3 Restrains Dendritic Cell Migration by Inhibiting HIF‐1&agr;‐Mediated Glycolysis , 2019, Immunity.
[15] Nicole M. Chapman,et al. Emerging Roles of Cellular Metabolism in Regulating Dendritic Cell Subsets and Function , 2018, Front. Cell Dev. Biol..
[16] D. Sancho,et al. Human Dendritic Cell Subsets Undergo Distinct Metabolic Reprogramming for Immune Response , 2018, Front. Immunol..
[17] J. Ernst. Mechanisms of M. tuberculosis Immune Evasion as Challenges to TB Vaccine Design. , 2018, Cell host & microbe.
[18] E. Ma,et al. Glycolytic metabolism is essential for CCR7 oligomerization and dendritic cell migration , 2018, Nature Communications.
[19] R. Poincloux,et al. Podosomes, But Not the Maturation Status, Determine the Protease-Dependent 3D Migration in Human Dendritic Cells , 2018, Front. Immunol..
[20] R. Hernández-Pando,et al. Formation of Foamy Macrophages by Tuberculous Pleural Effusions Is Triggered by the Interleukin-10/Signal Transducer and Activator of Transcription 3 Axis through ACAT Upregulation , 2018, Front. Immunol..
[21] M. Jeyanathan,et al. CD11b+ Dendritic Cell–Mediated Anti–Mycobacterium tuberculosis Th1 Activation Is Counterregulated by CD103+ Dendritic Cells via IL-10 , 2018, The Journal of Immunology.
[22] D. Goletti,et al. Impaired IFN-α-mediated signal in dendritic cells differentiates active from latent tuberculosis , 2018, PloS one.
[23] Linda V. Sinclair,et al. Glucose represses dendritic cell-induced T cell responses , 2017, Nature Communications.
[24] Maxim N. Artyomov,et al. Targeting dendritic cells to accelerate T-cell activation overcomes a bottleneck in tuberculosis vaccine efficacy , 2016, Nature Communications.
[25] L. Balboa,et al. Monocyte-derived dendritic cells early exposed to Mycobacterium tuberculosis induce an enhanced T helper 17 response and transfer mycobacterial antigens. , 2016, International journal of medical microbiology : IJMM.
[26] I. Komuro,et al. HIF-1α-PDK1 axis-induced active glycolysis plays an essential role in macrophage migratory capacity , 2016, Nature Communications.
[27] E. Cummins,et al. The role of HIF in immunity and inflammation. , 2016, Molecular aspects of medicine.
[28] J. Ernst,et al. Antigen Export Reduces Antigen Presentation and Limits T Cell Control of M. tuberculosis. , 2016, Cell host & microbe.
[29] J. Harding,et al. Mycobacterium-Infected Dendritic Cells Disseminate Granulomatous Inflammation , 2015, Scientific Reports.
[30] S. Nikonov,et al. Impairments of Antigen-Presenting Cells in Pulmonary Tuberculosis , 2015, Journal of immunology research.
[31] U. Schaible,et al. Macrophage defense mechanisms against intracellular bacteria , 2015, Immunological reviews.
[32] F. Ginhoux,et al. Monocytes and macrophages: developmental pathways and tissue homeostasis , 2014, Nature Reviews Immunology.
[33] Maxim N. Artyomov,et al. TLR-driven early glycolytic reprogramming via the kinases TBK1-IKKɛ supports the anabolic demands of dendritic cell activation , 2014, Nature Immunology.
[34] Cory M. Robinson,et al. Mycobacterium tuberculosis infection of human dendritic cells decreases integrin expression, adhesion and migration to chemokines , 2014, Immunology.
[35] H. A. Schreiber,et al. Essential yet limited role for CCR2+ inflammatory monocytes during Mycobacterium tuberculosis-specific T cell priming , 2013, eLife.
[36] L. Balboa,et al. Impaired dendritic cell differentiation of CD16‐positive monocytes in tuberculosis: Role of p38 MAPK , 2013, European journal of immunology.
[37] K. Lage,et al. Differential protein pathways in 1,25-dihydroxyvitamin d(3) and dexamethasone modulated tolerogenic human dendritic cells. , 2012, Journal of proteome research.
[38] Wing-Cheong Wong,et al. Gene expression profiling reveals the defining features of the classical, intermediate, and nonclassical human monocyte subsets. , 2011, Blood.
[39] L. Balboa,et al. Paradoxical role of CD16+CCR2+CCR5+ monocytes in tuberculosis: efficient APC in pleural effusion but also mark disease severity in blood , 2011, Journal of leukocyte biology.
[40] J. Ernst,et al. Initiation and regulation of T-cell responses in tuberculosis , 2011, Mucosal Immunology.
[41] Silvano Sozzani,et al. Nomenclature of monocytes and dendritic cells in blood. , 2010, Blood.
[42] L. Balboa,et al. Mycobacterium tuberculosis impairs dendritic cell response by altering CD1b, DC‐SIGN and MR profile , 2010, Immunology and cell biology.
[43] J. Casanova,et al. Human CD14dim Monocytes Patrol and Sense Nucleic Acids and Viruses via TLR7 and TLR8 Receptors , 2010, Immunity.
[44] T. Holowka,et al. Toll-like receptor-induced changes in glycolytic metabolism regulate dendritic cell activation. , 2010, Blood.
[45] C. Harding,et al. Regulation of antigen presentation by Mycobacterium tuberculosis: a role for Toll-like receptors , 2010, Nature Reviews Microbiology.
[46] F. Gauffre,et al. Matrix Architecture Dictates Three-Dimensional Migration Modes of Human Macrophages: Differential Involvement of Proteases and Podosome-Like Structures , 2009, The Journal of Immunology.
[47] K. Lage,et al. Proteome analysis demonstrates profound alterations in human dendritic cell nature by TX527, an analogue of vitamin D , 2009, Proteomics.
[48] A. Cooper,et al. Cell-mediated immune responses in tuberculosis. , 2009, Annual review of immunology.
[49] Sulochana D. Das,et al. Differential migration of human monocyte-derived dendritic cells after infection with prevalent clinical strains of Mycobacterium tuberculosis. , 2008, Immunobiology.
[50] Alan D. Roberts,et al. ESAT-6-specific CD4 T cell responses to aerosol Mycobacterium tuberculosis infection are initiated in the mediastinal lymph nodes , 2008, Proceedings of the National Academy of Sciences.
[51] Kai Zacharowski,et al. The role of toll-like receptors , 2008 .
[52] J. Ernst,et al. Initiation of the adaptive immune response to Mycobacterium tuberculosis depends on antigen production in the local lymph node, not the lungs , 2008, The Journal of experimental medicine.
[53] J. Ernst,et al. Mycobacterium tuberculosis Infects Dendritic Cells with High Frequency and Impairs Their Function In Vivo1 , 2007, The Journal of Immunology.
[54] D. Jarrossay,et al. Surface phenotype and antigenic specificity of human interleukin 17–producing T helper memory cells , 2007, Nature Immunology.
[55] S. Swain,et al. Interleukin 12p40 is required for dendritic cell migration and T cell priming after Mycobacterium tuberculosis infection , 2006, The Journal of experimental medicine.
[56] T. Blankenstein,et al. CCR7 governs skin dendritic cell migration under inflammatory and steady-state conditions. , 2004, Immunity.
[57] B. Ryffel,et al. Toll-like receptor pathways in the immune responses to mycobacteria. , 2004, Microbes and infection.
[58] Antonio Lanzavecchia,et al. Regulation of Dendritic Cell Migration to the Draining Lymph Node , 2003, The Journal of experimental medicine.
[59] A. Aderem,et al. Toll-like receptor-2 mediates mycobacteria-induced proinflammatory signaling in macrophages. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[60] M. Rothe,et al. Peptidoglycan- and Lipoteichoic Acid-induced Cell Activation Is Mediated by Toll-like Receptor 2* , 1999, The Journal of Biological Chemistry.
[61] F. Gusovsky,et al. Toll-like Receptor-4 Mediates Lipopolysaccharide-induced Signal Transduction* , 1999, The Journal of Biological Chemistry.
[62] P. Trinder,et al. An improved colour reagent for the determination of blood glucose by the oxidase system. , 1972, The Analyst.