Serial soluble neurofilament heavy chain in plasma as a marker of brain injury after cardiac arrest

[1]  W. Freeman,et al.  Neuron-specific enolase correlates with other prognostic markers after cardiac arrest , 2011, Neurology.

[2]  M. Brown,et al.  Hyperacute Detection of Neurofilament Heavy Chain in Serum Following Stroke: A Transient Sign , 2011, Neurochemical Research.

[3]  P. Mccombe,et al.  Levels of phosphorylated axonal neurofilament subunit H (pNfH) are increased in acute ischemic stroke , 2011, Journal of the Neurological Sciences.

[4]  B. Uitdehaag,et al.  Blood and CSF Biomarker Dynamics in Multiple Sclerosis: Implications for Data Interpretation , 2011, Multiple sclerosis international.

[5]  L. Kappos,et al.  Neurofilament heavy chain in CSF correlates with relapses and disability in multiple sclerosis , 2011, Neurology.

[6]  V. Tsang,et al.  Remote Ischemic Preconditioning Protects the Brain Against Injury After Hypothermic Circulatory Arrest , 2011, Circulation.

[7]  L. Martinian,et al.  In vivo monitoring of neuronal loss in traumatic brain injury: a microdialysis study , 2011, Brain : a journal of neurology.

[8]  R. White,et al.  Predictors of neurologic outcome in hypothermia after cardiac arrest , 2010, Annals of neurology.

[9]  M. Blankenstein,et al.  Batch prepared protein standards for cerebrospinal fluid (CSF) biomarkers for neurodegeneration , 2010, Journal of Neuroscience Methods.

[10]  Mauro Oddo,et al.  Prognostication after cardiac arrest and hypothermia: A prospective study , 2010, Annals of neurology.

[11]  R. Neumar,et al.  Post-cardiac arrest syndrome: Epidemiology, pathophysiology, treatment, and prognostication: A scientific statement from the International Liaison Committee on Resuscitation; the American Heart Association Emergency Cardiovascular Care Committee; the Council on Cardiovascular Surgery and Anesthesia; , 2009, International emergency nursing.

[12]  T. Cronberg,et al.  Neuron specific enolase and S-100B as predictors of outcome after cardiac arrest and induced hypothermia. , 2009, Resuscitation.

[13]  T. Kaneko,et al.  Serum glial fibrillary acidic protein as a predictive biomarker of neurological outcome after cardiac arrest. , 2009, Resuscitation.

[14]  B. Hemmer,et al.  The Value of the Serum Neurofilament Protein Heavy Chain as a Biomarker for Peri-operative Brain Injury After Carotid Endarterectomy , 2009, Neurochemical Research.

[15]  R. Neumar,et al.  Consensus Process , 2022 .

[16]  B. Young,et al.  Predictors of poor neurologic outcome after induced mild hypothermia following cardiac arrest , 2008, Neurology.

[17]  M. Schluep,et al.  Glial and axonal body fluid biomarkers are related to infarct volume, severity, and outcome. , 2008, Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association.

[18]  P. Kochanek,et al.  Effects of hypothermia on drug disposition, metabolism, and response: A focus of hypothermia-mediated alterations on the cytochrome P450 enzyme system , 2007, Critical care medicine.

[19]  T. Zima,et al.  Performance Characteristics of Seven Neuron-Specific Enolase Assays , 2007, Tumor Biology.

[20]  G. B. Young,et al.  Practice Parameter: Prediction of outcome in comatose survivors after cardiopulmonary resuscitation (an evidence-based review) , 2006, Neurology.

[21]  G Keir,et al.  Axonal damage and outcome in subarachnoid haemorrhage , 2006, Journal of Neurology, Neurosurgery & Psychiatry.

[22]  Ingmar Rosén,et al.  Amplitude-integrated EEG (aEEG) predicts outcome after cardiac arrest and induced hypothermia , 2006, Intensive Care Medicine.

[23]  A. Hart,et al.  Prediction of poor outcome within the first 3 days of postanoxic coma , 2006, Neurology.

[24]  A. Petzold Neurofilament phosphoforms: Surrogate markers for axonal injury, degeneration and loss , 2005, Journal of the Neurological Sciences.

[25]  B. Romner,et al.  Raised serum S100B levels after acute bone fractures without cerebral injury. , 2005, The Journal of trauma.

[26]  J. Nolan,et al.  Mode of death after admission to an intensive care unit following cardiac arrest , 2004, Intensive Care Medicine.

[27]  L. Rosengren,et al.  CSF levels of neurofilament is a valuable predictor of long-term outcome after cardiac arrest , 2004, Journal of the Neurological Sciences.

[28]  S. Ridley,et al.  Drugs in anaesthesia , 2003, Anaesthesia.

[29]  G. Giovannoni,et al.  A specific ELISA for measuring neurofilament heavy chain phosphoforms. , 2003, Journal of immunological methods.

[30]  R. Anderson,et al.  Increase in serum S100A1-B and S100BB during cardiac surgery arises from extracerebral sources. , 2001, The Annals of thoracic surgery.

[31]  C. Alling,et al.  Neuron-specific enolase increases in plasma during and immediately after extracorporeal circulation. , 2000, The Annals of thoracic surgery.

[32]  S. Kelsey,et al.  Assessment of neurological prognosis in comatose survivors of cardiac arrest , 1994, The Lancet.

[33]  Fred Plum,et al.  Temporal profile of neuronal damage in a model of transient forebrain ischemia , 1982, Annals of neurology.

[34]  B. Jennett,et al.  ASSESSMENT OF OUTCOME AFTER SEVERE BRAIN DAMAGE A Practical Scale , 1975, The Lancet.

[35]  B Jennett,et al.  Assessment of outcome after severe brain damage. , 1975, Lancet.