Perivascular Arrest of CD8+ T Cells Is a Signature of Experimental Cerebral Malaria
暂无分享,去创建一个
Ana Rodriguez | Michael Loran Dustin | A. Villegas-Méndez | K. Couper | N. Van Rooijen | J. A. Coles | P. Stewart-Hutchinson | J. Brewer | T. Shaw | Egor Zindy | Patrick Strangward | J. Gallego-Delgado | D. Dandamudi | A. Villegas‐Mendez | Tovah N. Shaw
[1] B. Igyártó,et al. Quantifying Memory CD8 T Cells Reveals Regionalization of Immunosurveillance , 2015, Cell.
[2] C. Poh,et al. Pathogenic CD8+ T cells in experimental cerebral malaria , 2015, Seminars in Immunopathology.
[3] P. Loke,et al. Experimental Cerebral Malaria Pathogenesis—Hemodynamics at the Blood Brain Barrier , 2014, PLoS pathogens.
[4] Ulrike Böhme,et al. A comprehensive evaluation of rodent malaria parasite genomes and gene expression , 2014, BMC Biology.
[5] C. Poh,et al. Damage to the Blood-Brain Barrier during Experimental Cerebral Malaria Results from Synergistic Effects of CD8+ T Cells with Different Specificities , 2014, Infection and Immunity.
[6] N. Hunt,et al. Real-Time Imaging Reveals the Dynamics of Leukocyte Behaviour during Experimental Cerebral Malaria Pathogenesis , 2014, PLoS pathogens.
[7] C. Hempel,et al. Systemic and Cerebral Vascular Endothelial Growth Factor Levels Increase in Murine Cerebral Malaria along with Increased Calpain and Caspase Activity and Can be Reduced by Erythropoietin Treatment , 2014, Front. Immunol..
[8] G. McFadden,et al. CD8+ T Cells from a Novel T Cell Receptor Transgenic Mouse Induce Liver-Stage Immunity That Can Be Boosted by Blood-Stage Infection in Rodent Malaria , 2014, PLoS pathogens.
[9] G. Grau,et al. Electron microscopic features of brain edema in rodent cerebral malaria in relation to glial fibrillary acidic protein expression. , 2014, International journal of clinical and experimental pathology.
[10] R. Spaccapelo,et al. Protective or pathogenic effects of vascular endothelial growth factor (VEGF) as potential biomarker in cerebral malaria , 2014, Pathogens and global health.
[11] Ronald N Germain,et al. Tuning of antigen sensitivity by T cell receptor-dependent negative feedback controls T cell effector function in inflamed tissues. , 2014, Immunity.
[12] J. Yates,et al. Microglia Promote Learning-Dependent Synapse Formation through Brain-Derived Neurotrophic Factor , 2013, Cell.
[13] B. Brown,et al. Clinical and laboratory predictors of outcome in cerebral malaria in suburban Nigeria. , 2013, Journal of infection in developing countries.
[14] Michael P. Barrett,et al. In Vivo Imaging of Trypanosome-Brain Interactions and Development of a Rapid Screening Test for Drugs against CNS Stage Trypanosomiasis , 2013, PLoS neglected tropical diseases.
[15] F. Ginhoux,et al. Brain microvessel cross-presentation is a hallmark of experimental cerebral malaria , 2013, EMBO molecular medicine.
[16] Darren J Obbard,et al. Molecular evolution and phylogenetics of rodent malaria parasites , 2012, BMC Evolutionary Biology.
[17] S. Kappe,et al. Neuroimmunological Blood Brain Barrier Opening in Experimental Cerebral Malaria , 2012, PLoS pathogens.
[18] C. Hunter,et al. IFN-γ–Producing CD4+ T Cells Promote Experimental Cerebral Malaria by Modulating CD8+ T Cell Accumulation within the Brain , 2012, The Journal of Immunology.
[19] Andrea J. Liu,et al. Generalized Lévy walks and the role of chemokines in migration of effector CD8+ T cells , 2012, Nature.
[20] Nancy Fullman,et al. Global malaria mortality between 1980 and 2010: a systematic analysis , 2012, The Lancet.
[21] M. V. von Herrath,et al. Intravital imaging of CTLs killing islet cells in diabetic mice. , 2012, The Journal of clinical investigation.
[22] A. Papenfuss,et al. Blood-stage Plasmodium berghei infection generates a potent, specific CD8+ T-cell response despite residence largely in cells lacking MHC I processing machinery. , 2011, The Journal of infectious diseases.
[23] E. Riley,et al. Heterogeneous and Tissue-Specific Regulation of Effector T Cell Responses by IFN-γ during Plasmodium berghei ANKA Infection , 2011, The Journal of Immunology.
[24] Shuxian Hu,et al. Memory T cells persisting in the brain following MCMV infection induce long-term microglial activation via interferon-γ , 2011, Journal of NeuroVirology.
[25] N. Anstey,et al. Granzyme B Expression by CD8+ T Cells Is Required for the Development of Experimental Cerebral Malaria , 2011, The Journal of Immunology.
[26] P. Maffia,et al. In Vivo Real-Time Multiphoton Imaging of T Lymphocytes in the Mouse Brain After Experimental Stroke , 2011, Stroke.
[27] F. Ginhoux,et al. CD8+ T Cells and IFN-γ Mediate the Time-Dependent Accumulation of Infected Red Blood Cells in Deep Organs during Experimental Cerebral Malaria , 2011, PloS one.
[28] Kamolrat Silamut,et al. Artesunate versus quinine in the treatment of severe falciparum malaria in African children (AQUAMAT): an open-label, randomised trial , 2010, The Lancet.
[29] A. Loukas,et al. Immune-Mediated Mechanisms of Parasite Tissue Sequestration during Experimental Cerebral Malaria , 2010, The Journal of Immunology.
[30] C. Hempel,et al. In vivo expression of neuroglobin in reactive astrocytes during neuropathology in murine models of traumatic brain injury, cerebral malaria, and autoimmune encephalitis , 2010, Glia.
[31] A. Flügel,et al. Knocking at the brain’s door: intravital two-photon imaging of autoreactive T cell interactions with CNS structures , 2010, Seminars in Immunopathology.
[32] M. Mota,et al. Accumulation of Plasmodium berghei-Infected Red Blood Cells in the Brain Is Crucial for the Development of Cerebral Malaria in Mice , 2010, Infection and Immunity.
[33] Jaime Grutzendler,et al. Thinned-skull cranial window technique for long-term imaging of the cortex in live mice , 2010, Nature Protocols.
[34] J. Harty,et al. Tracking the Total CD8 T Cell Response to Infection Reveals Substantial Discordance in Magnitude and Kinetics between Inbred and Outbred Hosts1 , 2009, The Journal of Immunology.
[35] H. Wekerle,et al. Effector T cell interactions with meningeal vascular structures in nascent autoimmune CNS lesions , 2009, Nature.
[36] P. Kaye,et al. Alveolar Macrophages Transport Pathogens to Lung Draining Lymph Nodes1 , 2009, The Journal of Immunology.
[37] M. Norman,et al. IP-10-Mediated T Cell Homing Promotes Cerebral Inflammation over Splenic Immunity to Malaria Infection , 2009, PLoS pathogens.
[38] R. Ransohoff,et al. Localizing central nervous system immune surveillance: Meningeal antigen‐presenting cells activate T cells during experimental autoimmune encephalomyelitis , 2009, Annals of neurology.
[39] I. Bechmann,et al. Perivascular Spaces and the Two Steps to Neuroinflammation , 2008, Journal of neuropathology and experimental neurology.
[40] Michael Loran Dustin,et al. Protein Kinase Cθ Regulates Stability of the Peripheral Adhesion Ring Junction and Contributes to the Sensitivity of Target Cell Lysis by CTL1 , 2008, The Journal of Immunology.
[41] N. Van Rooijen,et al. Ly6c+ “inflammatory monocytes” are microglial precursors recruited in a pathogenic manner in West Nile virus encephalitis , 2008, The Journal of experimental medicine.
[42] G. Belz,et al. Blood-stage Plasmodium infection induces CD8+ T lymphocytes to parasite-expressed antigens, largely regulated by CD8α+ dendritic cells , 2008, Proceedings of the National Academy of Sciences.
[43] Aaron J. Johnson,et al. Induction of Blood Brain Barrier Tight Junction Protein Alterations by CD8 T Cells , 2008, PloS one.
[44] A. Haque,et al. Common Strategies To Prevent and Modulate Experimental Cerebral Malaria in Mouse Strains with Different Susceptibilities , 2008, Infection and Immunity.
[45] Philippe Bousso,et al. Two-photon imaging of intratumoral CD8+ T cell cytotoxic activity during adoptive T cell therapy in mice. , 2008, The Journal of clinical investigation.
[46] Catherine Q Nie,et al. NK Cells Stimulate Recruitment of CXCR3+ T Cells to the Brain during Plasmodium berghei-Mediated Cerebral Malaria1 , 2007, The Journal of Immunology.
[47] Sebastian Amigorena,et al. In vivo imaging of cytotoxic T cell infiltration and elimination of a solid tumor , 2007, The Journal of experimental medicine.
[48] R. Helbok,et al. Scanning electron microscopy of the neuropathology of murine cerebral malaria , 2006, Malaria Journal.
[49] Ralph Weissleder,et al. Regulatory T cells reversibly suppress cytotoxic T cell function independent of effector differentiation. , 2006, Immunity.
[50] L. Rénia,et al. Pathogenic T cells in cerebral malaria. , 2006, International journal for parasitology.
[51] H. Ball,et al. Perforin mediated apoptosis of cerebral microvascular endothelial cells during experimental cerebral malaria. , 2006, International journal for parasitology.
[52] F. Nosten,et al. Artesunate versus quinine for severe falciparum malaria – Authors' reply , 2006, The Lancet.
[53] Arjen Dondorp,et al. Artesunate versus quinine for treatment of severe falciparum malaria: a randomised trial , 2005, The Lancet.
[54] T. F. Pais,et al. Brain macrophage activation in murine cerebral malaria precedes accumulation of leukocytes and CD8+ T cell proliferation , 2005, Journal of Neuroimmunology.
[55] B. Becher,et al. Dendritic cells permit immune invasion of the CNS in an animal model of multiple sclerosis , 2005, Nature Medicine.
[56] V. Perry,et al. Mannose receptor expression specifically reveals perivascular macrophages in normal, injured, and diseased mouse brain , 2005, Glia.
[57] M. Gertsenstein,et al. Mouse in red: Red fluorescent protein expression in mouse ES cells, embryos, and adult animals , 2004, Genesis.
[58] Chris J Janse,et al. A Plasmodium berghei reference line that constitutively expresses GFP at a high level throughout the complete life cycle. , 2004, Molecular and biochemical parasitology.
[59] J. Russell,et al. TNFR1-dependent VCAM-1 expression by astrocytes exposes the CNS to destructive inflammation , 2004, Journal of Neuroimmunology.
[60] Wenjiang J. Fu,et al. Differentiating the pathologies of cerebral malaria by postmortem parasite counts , 2004, Nature Medicine.
[61] L. Rénia,et al. Chemokine Receptor CCR2 Is Not Essential for the Development of Experimental Cerebral Malaria , 2003, Infection and Immunity.
[62] D. Seilhean,et al. Perforin-Dependent Brain-Infiltrating Cytotoxic CD8+ T Lymphocytes Mediate Experimental Cerebral Malaria Pathogenesis 1 , 2003, The Journal of Immunology.
[63] L. Rénia,et al. On the Pathogenic Role of Brain-Sequestered αβ CD8+ T Cells in Experimental Cerebral Malaria1 , 2002, The Journal of Immunology.
[64] A. Nagy,et al. Embryonic stem cells and mice expressing different GFP variants for multiple non-invasive reporter usage within a single animal , 2002, BMC biotechnology.
[65] R. Ganju,et al. Lipopolysaccharide-Induced Apoptosis of Endothelial Cells and Its Inhibition by Vascular Endothelial Growth Factor , 2002, The Journal of Immunology.
[66] N. Rooijen,et al. The role of perivascular and meningeal macrophages in experimental allergic encephalomyelitis , 2002, Journal of Neuroimmunology.
[67] K. Jin,et al. Caspase-3 and the regulation of hypoxic neuronal death by vascular endothelial growth factor , 2001, Neuroscience.
[68] G. Grau,et al. Pathogenesis of Cerebral Malaria: Recent Experimental Data and Possible Applications for Humans , 2001, Clinical Microbiology Reviews.
[69] R. Ménard,et al. Fluorescent Plasmodium berghei sporozoites and pre‐erythrocytic stages: a new tool to study mosquito and mammalian host interactions with malaria parasites , 2001, Cellular Microbiology.
[70] T. Chan-Ling,et al. Central nervous system in cerebral malaria: ‘Innocent bystander’ or active participant in the induction of immunopathology? , 2001, Immunology and cell biology.
[71] P. Kubes,et al. Visualization of Plasmodium falciparum–Endothelium Interactions in Human Microvasculature , 2000, The Journal of experimental medicine.
[72] N. Rayment,et al. Immunopathology of Cerebral Malaria: Morphological Evidence of Parasite Sequestration in Murine Brain Microvasculature , 2000, Infection and Immunity.
[73] L. Rénia,et al. Involvement of IFN‐γ receptor‐mediated signaling in pathology and anti‐malarial immunity induced by Plasmodium berghei infection , 2000, European journal of immunology.
[74] A. Sher,et al. Analysis of Fractalkine Receptor CX3CR1 Function by Targeted Deletion and Green Fluorescent Protein Reporter Gene Insertion , 2000, Molecular and Cellular Biology.
[75] G. Grau,et al. Expression of major histocompatibility complex antigens on mouse brain microvascular endothelial cells in relation to susceptibility to cerebral malaria , 1997, Immunology.
[76] C. Cetrulo,et al. Vascular endothelial growth factor inhibits endothelial cell apoptosis induced by tumor necrosis factor-alpha: balance between growth and death signals. , 1997, Journal of molecular and cellular cardiology.
[77] David D. Manning,et al. Participation of lymphocyte subpopulations in the pathogenesis of experimental murine cerebral malaria. , 1996, Journal of immunology.
[78] Davis,et al. An immunohistochemical study of the pathology of fatal malaria. Evidence for widespread endothelial activation and a potential role for intercellular adhesion molecule-1 in cerebral sequestration. , 1994, The American journal of pathology.
[79] Kristin A. Hogquist,et al. T cell receptor antagonist peptides induce positive selection , 1994, Cell.
[80] H. Pircher,et al. Involvement of both T cell receptor Vα and Vβ variable region domains and α chain junctional region in viral antigen recognition , 1991 .
[81] O. Steinwall,et al. SELECTIVE VULNERABILITY OF THE BLOOD‐BRAIN BARRIER IN CHEMICALLY INDUCED LESIONS , 1966, Journal of neuropathology and experimental neurology.
[82] J. Stockman. Artesunate versus quinine in the treatment of severe falciparum malaria in African children (AQUAMAT): an open-label, randomised trial , 2012 .
[83] D. Sullivan,et al. Glial activation and matrix metalloproteinase release in cerebral malaria , 2011, Journal of NeuroVirology.
[84] T. Furuta,et al. Elevated levels of vascular endothelial growth factor (VEGF) and soluble vascular endothelial growth factor receptor (VEGFR)-2 in human malaria. , 2010, The American journal of tropical medicine and hygiene.
[85] H. Ball,et al. Interferon-gamma synergises with tumour necrosis factor and lymphotoxin-alpha to enhance the mRNA and protein expression of adhesion molecules in mouse brain endothelial cells. , 2007, Cytokine.
[86] V. Perry,et al. What is the blood-brain barrier (not)? , 2007, Trends in immunology.
[87] H. Ball,et al. Interferon-γ synergises with tumour necrosis factor and lymphotoxin-α to enhance the mRNA and protein expression of adhesion molecules in mouse brain endothelial cells , 2007 .
[88] L. Rénia,et al. On the pathogenic role of brain-sequestered alphabeta CD8+ T cells in experimental cerebral malaria. , 2002, Journal of immunology.
[89] H. Pircher,et al. Involvement of both T cell receptor V alpha and V beta variable region domains and alpha chain junctional region in viral antigen recognition. , 1991, European journal of immunology.
[90] Richard Kronland-Martinet,et al. A real-time algorithm for signal analysis with the help of the wavelet transform , 1989 .