Transcriptional activation of TRADD mediates p53-independent radiation-induced apoptosis of glioma cells
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F. McCormick | S. Ries | D. Haas-Kogan | G. Yount | Noga Shalev | D. Basila | M. Korn | G. Afshar
[1] D. Faller,et al. A Novel DNA Damage Checkpoint Involving Post-transcriptional Regulation of Cyclin A Expression* , 2000, The Journal of Biological Chemistry.
[2] E. Wagner,et al. c-Jun-Dependent CD95-L Expression Is a Rate-Limiting Step in the Induction of Apoptosis by Alkylating Agents , 2000, Molecular and Cellular Biology.
[3] V. Rotter,et al. Epithelial cells of different organs exhibit distinct patterns of p53-dependent and p53-independent apoptosis following DNA insult. , 1999, Experimental cell research.
[4] Matthew W. Strobeck,et al. Cyclin A Is a Functional Target of Retinoblastoma Tumor Suppressor Protein-mediated Cell Cycle Arrest* , 1999, The Journal of Biological Chemistry.
[5] D. Bar-Sagi,et al. Suppression of Ras-Induced Apoptosis by the Rac GTPase , 1999, Molecular and Cellular Biology.
[6] T. Nomura,et al. Signaling through the antigen receptor of B lymphocytes activates a p53-independent pathway of c-Myc-induced apoptosis , 1999, Oncogene.
[7] A. Weiss,et al. Induction of NF-κB by the Akt/PKB kinase , 1999, Current Biology.
[8] V. Rotter,et al. Accentuated apoptosis in normally developing p53 knockout mouse embryos following genotoxic stress , 1999, Oncogene.
[9] K. Varga,et al. Unbound states by analytic continuation in the coupling constant , 1999 .
[10] R. Brown,et al. p53-oriented cancer therapies: current progress. , 1999, Annals of oncology : official journal of the European Society for Medical Oncology.
[11] Lars E. French,et al. The TRAIL to selective tumor death , 1999, Nature Medicine.
[12] C. Rauch,et al. Tumoricidal activity of tumor necrosis factor–related apoptosis–inducing ligand in vivo , 1999, Nature Medicine.
[13] E. Reddy,et al. Modulation of life and death by the TNF receptor superfamily , 1998, Oncogene.
[14] D. Geschwind,et al. Genomic organization, 5' flanking enhancer region, and chromosomal assignment of the cell cycle gene, p55Cdc. , 1998, Molecular genetics and metabolism.
[15] R. Weichselbaum,et al. Activation of protein kinase C δ by the c-Abl tyrosine kinase in response to ionizing radiation , 1998, Oncogene.
[16] G. Evan,et al. Induction of TNF‐sensitive cellular phenotype by c‐Myc involves p53 and impaired NF‐κB activation , 1997, The EMBO journal.
[17] V. D’Agati,et al. C-MYC–induced Apoptosis in Polycystic Kidney Disease Is Bcl-2 and p53 Independent , 1997, The Journal of experimental medicine.
[18] J Weinstein,et al. Cell Cycle-regulated Expression, Phosphorylation, and Degradation of p55Cdc , 1997, The Journal of Biological Chemistry.
[19] I. Herr,et al. Activation of CD95 (APO‐1/Fas) signaling by ceramide mediates cancer therapy‐induced apoptosis , 1997, The EMBO journal.
[20] I. Herr,et al. The CD95 (APO-1/Fas) system mediates drug-induced apoptosis in neuroblastoma cells. , 1997, Cancer research.
[21] R. Gentz,et al. An antagonist decoy receptor and a death domain-containing receptor for TRAIL. , 1997, Science.
[22] T. Gura. How TRAIL Kills Cancer Cells, But Not Normal Cells , 1997, Science.
[23] C. Potten,et al. Apoptosis in small intestinal epithelia from p53-null mice: evidence for a delayed, p53-indepdendent G2/M-associated cell death after γ-irradiation , 1997, Oncogene.
[24] Arul M. Chinnaiyan,et al. The Receptor for the Cytotoxic Ligand TRAIL , 1997, Science.
[25] P. Galle,et al. Drug-induced apoptosis in hepatoma cells is mediated by the CD95 (APO-1/Fas) receptor/ligand system and involves activation of wild-type p53. , 1997, The Journal of clinical investigation.
[26] S. Marsters,et al. Induction of Apoptosis by Apo-2 Ligand, a New Member of the Tumor Necrosis Factor Cytokine Family* , 1996, The Journal of Biological Chemistry.
[27] I. Herr,et al. Involvement of the CD95 (APO–1/Fas) receptor/ligand system in drug–induced apoptosis in leukemia cells , 1996, Nature Medicine.
[28] C. Hunt,et al. The cell cycle-coupled expression of topoisomerase IIalpha during S phase is regulated by mRNA stability and is disrupted by heat shock or ionizing radiation , 1996, Molecular and cellular biology.
[29] N. Zaffaroni,et al. Effect of ionizing radiation on cell‐cycle progression and cyclin B1 expression in human melanoma cells , 1996, International journal of cancer.
[30] A. Chinnaiyan,et al. FADD/MORT1 Is a Common Mediator of CD95 (Fas/APO-1) and Tumor Necrosis Factor Receptor-induced Apoptosis (*) , 1996, The Journal of Biological Chemistry.
[31] Hong-Bing Shu,et al. TRADD–TRAF2 and TRADD–FADD Interactions Define Two Distinct TNF Receptor 1 Signal Transduction Pathways , 1996, Cell.
[32] C A Smith,et al. Identification and characterization of a new member of the TNF family that induces apoptosis. , 1995, Immunity.
[33] M. Akashi,et al. Irradiation Induces WAF1 Expression through a p53-independent Pathway in KG-1 Cells (*) , 1995, The Journal of Biological Chemistry.
[34] M. Prados,et al. Constitutional p53 mutations associated with brain tumors in young adults. , 1995, Cancer genetics and cytogenetics.
[35] J. Roth,et al. Wild-type human p53 and a temperature-sensitive mutant induce Fas/APO-1 expression , 1995, Molecular and cellular biology.
[36] D. Goeddel,et al. The TNF receptor 1-associated protein TRADD signals cell death and NF-κB activation , 1995, Cell.
[37] A. Planas,et al. Evidence of internucleosomal DNA fragmentation and identification of dying cells in X-ray-induced cell death in the developing brain , 1995, International Journal of Developmental Neuroscience.
[38] N. Copeland,et al. Induction of apoptosis by the mouse Nedd2 gene, which encodes a protein similar to the product of the Caenorhabditis elegans cell death gene ced-3 and the mammalian IL-1 beta-converting enzyme. , 1994, Genes & development.
[39] P. Barr,et al. Apoptosis and Its Role in Human Disease , 1994, Bio/Technology.
[40] L. Tartaglia,et al. A novel domain within the 55 kd TNF receptor signals cell death , 1993, Cell.
[41] R. Weichselbaum,et al. Down-regulation of cell cycle control genes by ionizing radiation. , 1992, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.
[42] I. Ferrer. The effect of cycloheximide on natural and X-ray-induced cell death in the developing cerebral cortex , 1992, Brain Research.
[43] R. Muschel,et al. Cyclin B expression in HeLa cells during the G2 block induced by ionizing radiation. , 1991, Cancer research.
[44] P. Steinbok,et al. Superfractionated radiotherapy in grade III, IV intracranial gliomas. , 1988, International journal of radiation oncology, biology, physics.
[45] N Albright,et al. Computer programs for the analysis of cellular survival data. , 1987, Radiation research.
[46] B. Fertil,et al. Intrinsic radiosensitivity of human cell lines is correlated with radioresponsiveness of human tumors: analysis of 101 published survival curves. , 1985, International journal of radiation oncology, biology, physics.
[47] G. Sheline. Radiation therapy of brain tumors , 1977, Cancer.
[48] W. El-Deiry,et al. Regulation of p53 downstream genes. , 1998, Seminars in cancer biology.
[49] M. Prados,et al. Radiation therapy and hydroxyurea followed by the combination of 6-thioguanine and BCNU for the treatment of primary malignant brain tumors. , 1998, International journal of radiation oncology, biology, physics.
[50] I. Krantz,et al. KILLER/DR5 is a DNA damage–inducible p53–regulated death receptor gene , 1997, Nature Genetics.
[51] W. Dewey,et al. p53-dependent G1 arrest and p53-independent apoptosis influence the radiobiologic response of glioblastoma. , 1996, International journal of radiation oncology, biology, physics.
[52] A. Taghian,et al. In vitro intrinsic radiation sensitivity of glioblastoma multiforme. , 1992, International journal of radiation oncology, biology, physics.
[53] S. Murphy. Generation of Astrocyte Cultures from Normal and Neoplastic Central Nervous System , 1990 .