Unique inflammatory signature in haemophilic arthropathy: miRNA changes due to interaction between blood and fibroblast‐like synoviocytes
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[1] M. Manetti,et al. TNF-α/TNF-R System May Represent a Crucial Mediator of Proliferative Synovitis in Hemophilia A , 2019, Journal of clinical medicine.
[2] M. Kjelgaard‐Hansen,et al. Rapid inflammation and early degeneration of bone and cartilage revealed in a time-course study of induced haemarthrosis in haemophilic rats , 2018, Rheumatology.
[3] Keyuan Zhou,et al. Upregulation of miR-196b-5p attenuates BCG uptake via targeting SOCS3 and activating STAT3 in macrophages from patients with long-term cigarette smoking-related active pulmonary tuberculosis , 2018, Journal of Translational Medicine.
[4] P. Declerck,et al. Defective TAFI activation in hemophilia A mice is a major contributor to joint bleeding. , 2018, Blood.
[5] C. Blobel,et al. Blood-induced bone loss in murine hemophilic arthropathy is prevented by blocking the iRhom2/ADAM17/TNF-α pathway. , 2018, Blood.
[6] F. Lammert,et al. Cytokine‐mediated modulation of the hepatic miRNome: miR‐146b‐5p is an IL‐6‐inducible miRNA with multiple targets , 2018, Journal of leukocyte biology.
[7] Luyi Wu,et al. The Role of Autophagy and Related MicroRNAs in Inflammatory Bowel Disease , 2018, Gastroenterology research and practice.
[8] K. Yoshiura,et al. MicroRNA-204-3p inhibits lipopolysaccharide-induced cytokines in familial Mediterranean fever via the phosphoinositide 3-kinase &ggr; pathway , 2018, Rheumatology.
[9] Z. Cai,et al. The role of miR-146b-5p in TLR4 pathway of glomerular mesangial cells with lupus nephritis. , 2018, European review for medical and pharmacological sciences.
[10] A. Radzikowska,et al. Monocyte-related biomarkers of rheumatoid arthritis development in undifferentiated arthritis patients – a pilot study , 2018, Reumatologia.
[11] C. Harris,et al. Mutant p53 cancers reprogram macrophages to tumor supporting macrophages via exosomal miR-1246 , 2018, Nature Communications.
[12] X. Mariette,et al. Deregulation of microRNA expression in purified T and B lymphocytes from patients with primary Sjögren’s syndrome , 2017, Annals of the rheumatic diseases.
[13] E. Semik,et al. Reduced levels of methyltransferase DNMT2 sensitize human fibroblasts to oxidative stress and DNA damage that is accompanied by changes in proliferation-related miRNA expression , 2017, Redox biology.
[14] Henry J González-Torres,et al. Profiling analysis of circulating microRNA in peripheral blood of patients with class IV lupus nephritis , 2017, PloS one.
[15] S. Skov,et al. Acute Haemarthrosis in the Haemophilia A Rat Generates a Local and Systemic Proinflammatory Response , 2017, Thrombosis and Haemostasis.
[16] F. Lafeber,et al. Differential effects of bleeds on the development of arthropathy – basic and applied issues , 2017, Haemophilia : the official journal of the World Federation of Hemophilia.
[17] A. Andriulli,et al. Functional Implications of MicroRNAs in Crohn’s Disease Revealed by Integrating MicroRNA and Messenger RNA Expression Profiling , 2017, International journal of molecular sciences.
[18] M. Evans,et al. MicroRNAs Modulate Oxidative Stress in Hypertension through PARP-1 Regulation , 2017, Oxidative medicine and cellular longevity.
[19] S. Wiemann,et al. miRNA-1246 induces pro-inflammatory responses in mesenchymal stem/stromal cells by regulating PKA and PP2A , 2017, Oncotarget.
[20] J. Knight,et al. miR-10b-5p is a novel Th17 regulator present in Th17 cells from ankylosing spondylitis , 2016, Annals of the rheumatic diseases.
[21] K. Bogunia-Kubik,et al. Significance of Polymorphism and Expression of miR-146a and NFkB1 Genetic Variants in Patients with Rheumatoid Arthritis , 2016, Archivum Immunologiae et Therapiae Experimentalis.
[22] F. Lafeber,et al. IL-1β, in contrast to TNFα, is pivotal in blood-induced cartilage damage and is a potential target for therapy. , 2015, Blood.
[23] C. Blobel,et al. Blood-Induced Arthropathy in Hemophilia: Mechanisms and Heterogeneity , 2015, Seminars in Thrombosis & Hemostasis.
[24] W. Fang,et al. Up-regulation of Serum MiR-130b-3p Level is Associated with Renal Damage in Early Lupus Nephritis , 2015, Scientific Reports.
[25] M. Morfini,et al. Agonistic anti‐human Fas monoclonal antibody induces fibroblast‐like synoviocyte apoptosis in haemophilic arthropathy: potential therapeutic implications , 2014, Haemophilia : the official journal of the World Federation of Hemophilia.
[26] K. Metzner,et al. Synthetic pre-microRNAs reveal dual-strand activity of miR-34a on TNF-α , 2014, RNA.
[27] T. Bateman,et al. IL‐6 receptor antagonist as adjunctive therapy with clotting factor replacement to protect against bleeding‐induced arthropathy in hemophilia , 2013, Journal of thrombosis and haemostasis : JTH.
[28] G. Yousef,et al. Pleiotropic action of renal cell carcinoma-dysregulated miRNAs on hypoxia-related signaling pathways. , 2012, The American journal of pathology.
[29] J. Gottenberg,et al. Synovial fibroblasts promote immunoglobulin class switching by a mechanism involving BAFF , 2011, European journal of immunology.
[30] S. Pfeffer,et al. miR-346 Controls Release of TNF-α Protein and Stability of Its mRNA in Rheumatoid Arthritis via Tristetraprolin Stabilization , 2011, PloS one.
[31] G. Firestein,et al. Fibroblast‐like synoviocytes: key effector cells in rheumatoid arthritis , 2010, Immunological reviews.
[32] C. Datz,et al. Synovial immunopathology in haemochromatosis arthropathy , 2009, Annals of the rheumatic diseases.
[33] L. Valentino,et al. Physiopathology of haemophilic arthropathy , 2008, Haemophilia : the official journal of the World Federation of Hemophilia.
[34] Alan R. Cohen,et al. Prophylaxis versus episodic treatment to prevent joint disease in boys with severe hemophilia. , 2007, The New England journal of medicine.
[35] L. Valentino,et al. Pathobiology of hemophilic synovitis I: overexpression of mdm2 oncogene. , 2004, Blood.
[36] L. Valentino,et al. c-myc proto-oncogene expression in hemophilic synovitis: in vitro studies of the effects of iron and ceramide. , 2002, Blood.
[37] P. Mannucci. Hemophilia and related bleeding disorders: a story of dismay and success. , 2002, Hematology. American Society of Hematology. Education Program.
[38] J. Sibilia,et al. NF‐κB and the MAP kinases/AP‐1 pathways are both involved in interleukin‐6 and interleukin‐8 expression in fibroblast‐like synoviocytes stimulated by protein I/II, a modulin from oral streptococci , 2001, Cellular microbiology.
[39] J. Bijlsma,et al. Blood-induced joint damage: a human in vitro study. , 1999, Arthritis and rheumatism.
[40] J. Ogier,et al. The A and the extended V N‐terminal regions of streptococcal protein I/IIf mediate the production of tumour necrosis factor α in the monocyte cell line THP‐1 , 1998, Molecular microbiology.
[41] J. Bijlsma,et al. Iron deposits and catabolic properties of synovial tissue from patients with haemophilia , 1998 .
[42] H. Pettersson,et al. A longitudinal study of orthopaedic outcomes for severe factor‐VIII‐deficient haemophiliacs , 1994, Journal of internal medicine.