MicroRNA-1 Negatively Regulates Expression of the Hypertrophy-Associated Calmodulin and Mef2a Genes
暂无分享,去创建一个
T. Golub | Jun Lu | S. Ikeda | Aibin He | S. Kong | R. Bejar | N. Bodyak | Kyu-Ho Lee | Qing Ma | P. Kang | W. Pu
[1] B. Nadal-Ginard,et al. Protooncogene induction and reprogramming of cardiac gene expression produced by pressure overload. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[2] A. Katz. The cardiomyopathy of overload: an unnatural growth response. , 1995, European heart journal.
[3] A. Habara-Ohkubo. Differentiation of beating cardiac muscle cells from a derivative of P19 embryonal carcinoma cells. , 1996, Cell structure and function.
[4] Jeffrey Robbins,et al. A Calcineurin-Dependent Transcriptional Pathway for Cardiac Hypertrophy , 1998, Cell.
[5] F. Charron,et al. GATA‐dependent recruitment of MEF2 proteins to target promoters , 2000, The EMBO journal.
[6] Mark A Sussman,et al. Reversal of cardiac hypertrophy in transgenic disease models by calcineurin inhibition. , 2000, Journal of molecular and cellular cardiology.
[7] E. Strehler,et al. The calmodulin multigene family as a unique case of genetic redundancy: multiple levels of regulation to provide spatial and temporal control of calmodulin pools? , 2000, Cell calcium.
[8] R. Passier,et al. CaM kinase signaling induces cardiac hypertrophy and activates the MEF2 transcription factor in vivo. , 2000, The Journal of clinical investigation.
[9] J. Molkentin,et al. The Transcription Factors GATA4 and GATA6 Regulate Cardiomyocyte Hypertrophy in Vitro and in Vivo * , 2001, The Journal of Biological Chemistry.
[10] Chun Li Zhang,et al. Class II Histone Deacetylases Act as Signal-Responsive Repressors of Cardiac Hypertrophy , 2002, Cell.
[11] T. Tuschl,et al. Identification of Tissue-Specific MicroRNAs from Mouse , 2002, Current Biology.
[12] K. Khrapko,et al. Gene expression profiling of the aging mouse cardiac myocytes. , 2002, Nucleic acids research.
[13] W. Pu,et al. NFAT Transcription Factors Are Critical Survival Factors That Inhibit Cardiomyocyte Apoptosis During Phenylephrine Stimulation In Vitro , 2003, Circulation research.
[14] Terry Speed,et al. Normalization of cDNA microarray data. , 2003, Methods.
[15] John D. Storey,et al. Statistical significance for genomewide studies , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[16] D. Bartel. MicroRNAs Genomics, Biogenesis, Mechanism, and Function , 2004, Cell.
[17] Jian Xu,et al. Calcineurin/NFAT Coupling Participates in Pathological, but not Physiological, Cardiac Hypertrophy , 2004, Circulation research.
[18] Anton J. Enright,et al. Human MicroRNA Targets , 2004, PLoS biology.
[19] Yong Zhao,et al. Serum response factor regulates a muscle-specific microRNA that targets Hand2 during cardiogenesis , 2005, Nature.
[20] K. Gunsalus,et al. Combinatorial microRNA target predictions , 2005, Nature Genetics.
[21] D. Bartel,et al. Microarray profiling of microRNAs reveals frequent coexpression with neighboring miRNAs and host genes. , 2005, RNA.
[22] C. Burge,et al. Conserved Seed Pairing, Often Flanked by Adenosines, Indicates that Thousands of Human Genes are MicroRNA Targets , 2005, Cell.
[23] H. Izawa,et al. Overexpression of calmodulin induces cardiac hypertrophy by a calcineurin-dependent pathway. , 2005, Biochemical and biophysical research communications.
[24] C. Burge,et al. The Widespread Impact of Mammalian MicroRNAs on mRNA Repression and Evolution , 2005, Science.
[25] E. Olson,et al. Toward transcriptional therapies for the failing heart: chemical screens to modulate genes. , 2005, The Journal of clinical investigation.
[26] C. V. Jongeneel,et al. An atlas of human gene expression from massively parallel signature sequencing (MPSS). , 2005, Genome research.
[27] H. Horvitz,et al. MicroRNA expression profiles classify human cancers , 2005, Nature.
[28] N. Rajewsky,et al. Silencing of microRNAs in vivo with ‘antagomirs’ , 2005, Nature.
[29] Jian-Fu Chen,et al. The role of microRNA-1 and microRNA-133 in skeletal muscle proliferation and differentiation , 2006, Nature Genetics.
[30] P. Doevendans,et al. MEF2 Activates a Genetic Program Promoting Chamber Dilation and Contractile Dysfunction in Calcineurin-Induced Heart Failure , 2006, Circulation.
[31] S. Ikeda,et al. Gata4 is required for maintenance of postnatal cardiac function and protection from pressure overload-induced heart failure , 2006, Proceedings of the National Academy of Sciences.
[32] B. Aronow,et al. Myocyte Enhancer Factors 2A and 2C Induce Dilated Cardiomyopathy in Transgenic Mice* , 2006, Journal of Biological Chemistry.
[33] E. Olson,et al. A signature pattern of stress-responsive microRNAs that can evoke cardiac hypertrophy and heart failure , 2006, Proceedings of the National Academy of Sciences.
[34] MEF 2 Activates a Genetic Program Promoting Chamber Dilation and Contractile Dysfunction in Calcineurin-Induced Heart Failure , 2006 .
[35] R. Schwartz,et al. Cardiac-Specific Deletion of Gata4 Reveals Its Requirement for Hypertrophy, Compensation, and Myocyte Viability , 2006, Circulation research.
[36] C. Croce,et al. MicroRNA-133 controls cardiac hypertrophy , 2007, Nature Medicine.
[37] Thomas Thum,et al. MicroRNAs in the Human Heart: A Clue to Fetal Gene Reprogramming in Heart Failure , 2007, Circulation.
[38] Sek Won Kong,et al. Altered microRNA expression in human heart disease. , 2007, Physiological genomics.
[39] Danish Sayed,et al. MicroRNAs Play an Essential Role in the Development of Cardiac Hypertrophy , 2007, Circulation research.
[40] Donald M Bers,et al. Free and bound intracellular calmodulin measurements in cardiac myocytes. , 2007, Cell calcium.
[41] Chunxiang Zhang,et al. MicroRNAs are aberrantly expressed in hypertrophic heart: do they play a role in cardiac hypertrophy? , 2007, The American journal of pathology.
[42] Danish Sayed,et al. MicroRNAs Play an Essential Role in the Development of Cardiac Hypertrophy , 2007 .
[43] Chaoqian Xu,et al. The muscle-specific microRNA miR-1 regulates cardiac arrhythmogenic potential by targeting GJA1 and KCNJ2 , 2011, Nature Medicine.
[44] Aibin He,et al. Overexpression of micro ribonucleic acid 29, highly up-regulated in diabetic rats, leads to insulin resistance in 3T3-L1 adipocytes. , 2007, Molecular endocrinology.
[45] Michael T. McManus,et al. Dysregulation of Cardiogenesis, Cardiac Conduction, and Cell Cycle in Mice Lacking miRNA-1-2 , 2007, Cell.
[46] Doron Betel,et al. The microRNA.org resource: targets and expression , 2007, Nucleic Acids Res..
[47] E. Olson,et al. MicroRNAs flex their muscles. , 2008, Trends in genetics : TIG.
[48] Annie L. Conery,et al. The MicroRNA miR-1 Regulates a MEF-2-Dependent Retrograde Signal at Neuromuscular Junctions , 2008, Cell.