High-threshold calcium channel activity in rat hippocampal neurones during hypoxia 1 Published on the World Wide Web on 19 May 1999. 1
暂无分享,去创建一个
[1] D. Richter,et al. L‐type Ca2+ channels in inspiratory neurones of mice and their modulation by hypoxia , 1998, The Journal of physiology.
[2] E. Lukyanetz,et al. Calcineurin involvement in the regulation of high‐threshold Ca2+ channels in NG108–15 (rodent neuroblastoma × glioma hybrid) cells , 1998, The Journal of physiology.
[3] E. Lukyanetz. Diversity and properties of calcium channel types in NG108-15 hybrid cells , 1998, Neuroscience.
[4] K. Sawada,et al. Role of glutamate receptors and voltage-dependent calcium and sodium channels in the extracellular glutamate/aspartate accumulation and subsequent neuronal injury induced by oxygen/glucose deprivation in cultured hippocampal neurons. , 1998, The Journal of pharmacology and experimental therapeutics.
[5] N. Prabhakar,et al. Ca2+ current in rabbit carotid body glomus cells is conducted by multiple types of high-voltage-activated Ca2+ channels. , 1997, Journal of neurophysiology.
[6] William A Catterall,et al. Persistent Sodium Currents through Brain Sodium Channels Induced by G Protein βγ Subunits , 1997, Neuron.
[7] S. Waxman,et al. Downregulation of Tetrodotoxin-Resistant Sodium Currents and Upregulation of a Rapidly Repriming Tetrodotoxin-Sensitive Sodium Current in Small Spinal Sensory Neurons after Nerve Injury , 1997, The Journal of Neuroscience.
[8] A. Buchan,et al. Identification of calcium channels involved in neuronal injury in rat hippocampal slices subjected to oxygen and glucose deprivation , 1997, Brain Research.
[9] E. Lukyanetz. Evidence for Colocalization of Calcineurin and Calcium Channels in Dorsal Root Ganglion Neurons , 1997, Neuroscience.
[10] P. Calabresi,et al. Hypoxia in striatal and cortical neurones: membrane potential and Ca2+ measurements , 1997, Neuroreport.
[11] L. Sundstrom,et al. Selective N-type calcium channel antagonist omega conotoxin MVIIA is neuroprotective against hypoxic neurodegeneration in organotypic hippocampal-slice cultures. , 1996, Stroke.
[12] A. Schousboe,et al. Characterization of a chemical anoxia model in cerebellar granule neurons using sodium azide: Protection by nifedipine and MK‐801 , 1996, Journal of neuroscience research.
[13] D. Linden,et al. Reduced nicotinamide adenine dinucleotide-selective stimulation of inositol 1,4,5-trisphosphate receptors mediates hypoxic mobilization of calcium , 1996, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[14] R. S. Payne,et al. Synergism between diltiazem and MK-801 but not APV in protecting hippocampal slices against hypoxic damage , 1995, Brain Research.
[15] Shigenori Watanabe,et al. Calcium channel blockers improve hypoxia/hypoglycemia-induced impairment of rat hippocampal 2-deoxyglucose uptake in vitro after ethanol withdrawal , 1995, Brain Research.
[16] G. Erdemli,et al. Nitric oxide may be a mediator of effects of prolonged but not brief anoxia in CA1 neurons in slices , 1995, Neuropharmacology.
[17] P. Bickler,et al. Causes of calcium accumulation in rat cortical brain slices during hypoxia and ischemia: role of ion channels and membrane damage , 1994, Brain Research.
[18] F. Sigworth,et al. Oxygen deprivation activates an ATP-inhibitable K+ channel in substantia nigra neurons , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[19] D. Reis,et al. Hypoxia‐activated Ca2+ currents in pacemaker neurones of rat rostral ventrolateral medulla in vitro. , 1994, The Journal of physiology.
[20] B. Bell,et al. Effects of metabolic blockers on Ca2+‐dependent currents in cultured sensory neurones from neonatal rats , 1994, British journal of pharmacology.
[21] P. Kostyuk,et al. Ionic mechanism of electrical excitability in rat sensory neurons during postnatal ontogenesis , 1991, Neuroscience.
[22] K. Krnjević,et al. Hypoxic changes in hippocampal neurons. , 1989, Journal of neurophysiology.
[23] D. Choi,et al. Glutamate neurotoxicity and diseases of the nervous system , 1988, Neuron.
[24] G. Haddad,et al. O2-sensing mechanisms in excitable cells: role of plasma membrane K+ channels. , 1997, Annual review of physiology.