Immunohistochemical, genetic and clinical characterization of sporadic aldosterone-producing adenomas

[1]  C. Gomez-Sanchez,et al.  Adrenal vein sampling in primary aldosteronism: towards a standardised protocol. , 2015, The lancet. Diabetes & endocrinology.

[2]  P. Mulatero,et al.  Understanding primary aldosteronism: impact of next generation sequencing and expression profiling , 2015, Molecular and Cellular Endocrinology.

[3]  H. Sasano,et al.  Dissecting the molecular pathways of primary aldosteronism , 2014, Pathology international.

[4]  S. Ito,et al.  Aldosterone suppression on contralateral adrenal during adrenal vein sampling does not predict blood pressure response after adrenalectomy. , 2014, The Journal of clinical endocrinology and metabolism.

[5]  S. Ito,et al.  Different Expression of 11&bgr;-Hydroxylase and Aldosterone Synthase Between Aldosterone-Producing Microadenomas and Macroadenomas , 2014, Hypertension.

[6]  T. Strom,et al.  Genetic Spectrum and Clinical Correlates of Somatic Mutations in Aldosterone-Producing Adenoma , 2014, Hypertension.

[7]  B. Tops,et al.  Adrenal nodularity and somatic mutations in primary aldosteronism: one node is the culprit? , 2014, The Journal of clinical endocrinology and metabolism.

[8]  H. Sasano,et al.  Development of monoclonal antibodies against human CYP11B1 and CYP11B2 , 2014, Molecular and Cellular Endocrinology.

[9]  F. Beuschlein,et al.  Somatic ATP1A1, ATP2B3, and KCNJ5 Mutations in Aldosterone-Producing Adenomas , 2014, Hypertension.

[10]  F. Veglio,et al.  Long-term cardio- and cerebrovascular events in patients with primary aldosteronism. , 2013, The Journal of clinical endocrinology and metabolism.

[11]  James Hadfield,et al.  Somatic mutations in ATP1A1 and CACNA1D underlie a common subtype of adrenal hypertension , 2013, Nature Genetics.

[12]  Tetsurou Yamamoto,et al.  Histopathological diagnosis of primary aldosteronism using CYP11B2 immunohistochemistry. , 2013, The Journal of clinical endocrinology and metabolism.

[13]  T. Wieland,et al.  Somatic mutations in ATP1A1 and ATP2B3 lead to aldosterone-producing adenomas and secondary hypertension , 2013, Nature Genetics.

[14]  A. Fassina,et al.  Unilateral adrenal hyperplasia: a novel cause of surgically correctable primary hyperaldosteronism. , 2012, Surgery.

[15]  S. Ito,et al.  Effect of Adrenocorticotropic Hormone Stimulation During Adrenal Vein Sampling in Primary Aldosteronism , 2012, Hypertension.

[16]  Morris J. Brown,et al.  Microarray, qPCR, and KCNJ5 sequencing of aldosterone-producing adenomas reveal differences in genotype and phenotype between zona glomerulosa- and zona fasciculata-like tumors. , 2012, The Journal of clinical endocrinology and metabolism.

[17]  F. Beuschlein,et al.  Prevalence, Clinical, and Molecular Correlates of KCNJ5 Mutations in Primary Aldosteronism , 2012, Hypertension.

[18]  C. Gomez-Sanchez,et al.  Mutations of the potassium channel KCNJ5 causing aldosterone-producing adenomas: one or two hits? , 2012, Hypertension.

[19]  A. Fassina,et al.  Concurrent primary aldosteronism and subclinical cortisol hypersecretion: a prospective study , 2011, Journal of hypertension.

[20]  S. Mane,et al.  K+ Channel Mutations in Adrenal Aldosterone-Producing Adenomas and Hereditary Hypertension , 2011, Science.

[21]  X. Jeunemaître,et al.  Adrenal Cortex Remodeling and Functional Zona Glomerulosa Hyperplasia in Primary Aldosteronism , 2010, HYPERTENSION.

[22]  G. Altavilla,et al.  Zona fasciculata-like histotype and aldosterone response to upright posture are not related in aldosterone-producing adenomas. , 2009, Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association.

[23]  W. Young,et al.  Case detection, diagnosis, and treatment of patients with primary aldosteronism: an endocrine society clinical practice guideline. , 2008, The Journal of clinical endocrinology and metabolism.

[24]  F. Veglio,et al.  Roles of clinical criteria, computed tomography scan, and adrenal vein sampling in differential diagnosis of primary aldosteronism subtypes. , 2008, The Journal of clinical endocrinology and metabolism.

[25]  P. Bernante,et al.  Heterogeneity of Aldosterone-Producing Adenomas Revealed by a Whole Transcriptome Analysis , 2007, Hypertension.

[26]  S. Ishidoya,et al.  Laparoscopic partial versus total adrenalectomy for aldosterone producing adenoma. , 2005, The Journal of urology.

[27]  W. Young,et al.  Role for adrenal venous sampling in primary aldosteronism. , 2004, Surgery.

[28]  W. Young,et al.  Increased Diagnosis of Primary Aldosteronism, Including Surgically Correctable Forms, in Centers from Five Continents , 2004 .

[29]  H. Sasano,et al.  Unique cases of unilateral hyperaldosteronemia due to multiple adrenocortical micronodules, which can only be detected by selective adrenal venous sampling. , 2002, Metabolism: clinical and experimental.

[30]  X. Jeunemaître,et al.  Recombinant CYP11B genes encode enzymes that can catalyze conversion of 11-deoxycortisol to cortisol, 18-hydroxycortisol, and 18-oxocortisol. , 1998, The Journal of clinical endocrinology and metabolism.

[31]  T. Mune,et al.  Reduced response to metoclopramide and anomalous rising response to upright posture of plasma aldosterone concentration in Japanese patients with aldosterone-producing adenoma. , 1993, The Journal of clinical endocrinology and metabolism.

[32]  R. Gordon,et al.  Histological and biochemical distinctiveness of atypical aldosterone‐producing adenomas responsive to upright posture and angiotensin , 1991, Clinical endocrinology.

[33]  A. Neville,et al.  Histopathology of the human adrenal cortex. , 1985, Clinics in endocrinology and metabolism.

[34]  E B Cox,et al.  Estrogen receptor analyses. Correlation of biochemical and immunohistochemical methods using monoclonal antireceptor antibodies. , 1985, Archives of pathology & laboratory medicine.

[35]  K. Nakagawa,et al.  Adrenocortical zonation in humans under normal and pathological conditions. , 2010, The Journal of clinical endocrinology and metabolism.