L-DOPA Reverses the Increased Free Amino Acids Tissue Levels Induced by Dopamine Depletion and Rises GABA and Tyrosine in the Striatum
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[1] R. Moratalla,et al. Dopamine D3 Receptor Modulates l‐DOPA‐Induced Dyskinesia by Targeting D1 Receptor‐Mediated Striatal Signaling , 2015, Cerebral cortex.
[2] Qin Li,et al. Widespread Monoaminergic Dysregulation of Both Motor and Non-Motor Circuits in Parkinsonism and Dyskinesia. , 2015, Cerebral cortex.
[3] R. Moratalla,et al. Dopaminergic regulation of olfactory type G‐protein α subunit expression in the striatum , 2015, Movement disorders : official journal of the Movement Disorder Society.
[4] Hongbin Sun,et al. Correlations between plasma levels of amino acids and nonmotor symptoms in Parkinson’s disease , 2015, Journal of Neural Transmission.
[5] M. Casarejos,et al. Optimal excitation and emission wavelengths to analyze amino acids and optimize neurotransmitters quantification using precolumn OPA-derivatization by HPLC , 2015, Amino Acids.
[6] S. Hitchcock,et al. Enhanced histamine H2 excitation of striatal cholinergic interneurons in L-DOPA-induced dyskinesia , 2015, Neurobiology of Disease.
[7] R. Moratalla,et al. Nitric oxide synthase inhibition decreases l-DOPA-induced dyskinesia and the expression of striatal molecular markers in Pitx3−/− aphakia mice , 2015, Neurobiology of Disease.
[8] R. Moratalla,et al. Activation of DREAM (Downstream Regulatory Element Antagonistic Modulator), a Calcium-Binding Protein, Reduces L-DOPA-Induced Dyskinesias in Mice , 2015, Biological Psychiatry.
[9] P. Lewitt,et al. Levodopa therapy for Parkinson's disease: Pharmacokinetics and pharmacodynamics , 2015, Movement disorders : official journal of the Movement Disorder Society.
[10] Joshua L. Plotkin,et al. Cell type-specific plasticity of striatal projection neurons in parkinsonism and L-DOPA-induced dyskinesia , 2014, Nature Communications.
[11] P. Cowen,et al. Tyrosine-free amino acid mixtures reduce physiologically-evoked release of dopamine in a selective and activity-dependent manner , 2014, Journal of psychopharmacology.
[12] R. Moratalla,et al. L-DOPA Treatment Selectively Restores Spine Density in Dopamine Receptor D2–Expressing Projection Neurons in Dyskinetic Mice , 2014, Biological Psychiatry.
[13] R. Moratalla,et al. Oleoylethanolamide reduces L-DOPA-induced dyskinesia via TRPV1 receptor in a mouse model of Parkinson´s disease , 2014, Neurobiology of Disease.
[14] Lei Zhang,et al. Change in Plasma Levels of Amino Acid Neurotransmitters and its Correlation with Clinical Heterogeneity in Early Parkinson's Disease Patients , 2013, CNS neuroscience & therapeutics.
[15] D. Dexter,et al. Parkinson disease: from pathology to molecular disease mechanisms. , 2013, Free radical biology & medicine.
[16] R. Moratalla,et al. Methamphetamine and Parkinson's Disease , 2013, Parkinson's disease.
[17] Jun Shen. Modeling the glutamate–glutamine neurotransmitter cycle , 2012, Front. Neuroenerg..
[18] Eduardo D. Martín,et al. L-DOPA-induced increase in TH-immunoreactive striatal neurons in parkinsonian mice: Insights into regulation and function , 2012, Neurobiology of Disease.
[19] S. Dunnett,et al. Unilateral nigrostriatal 6-hydroxydopamine lesions in mice I: Motor impairments identify extent of dopamine depletion at three different lesion sites , 2012, Behavioural Brain Research.
[20] M. Cenci,et al. In vivo evidence for a differential contribution of striatal and nigral D1 and D2 receptors to l-DOPA induced dyskinesia and the accompanying surge of nigral amino acid levels , 2012, Neurobiology of Disease.
[21] S. Dunnett,et al. Unilateral nigrostriatal 6-hydroxydopamine lesions in mice II: Predicting l-DOPA-induced dyskinesia , 2012, Behavioural Brain Research.
[22] P. Calabresi,et al. Higher free d-aspartate and N-methyl-d-aspartate levels prevent striatal depotentiation and anticipate l-DOPA-induced dyskinesia , 2011, Experimental Neurology.
[23] M. Morari,et al. Amantadine attenuates levodopa‐induced dyskinesia in mice and rats preventing the accompanying rise in nigral GABA levels , 2011, Journal of neurochemistry.
[24] R. Moratalla,et al. Striatal Signaling in L-DOPA-Induced Dyskinesia: Common Mechanisms with Drug Abuse and Long Term Memory Involving D1 Dopamine Receptor Stimulation , 2011, Front. Neuroanat..
[25] D. Erlij,et al. l-DOPA-induced dyskinesia in hemiparkinsonian rats is associated with up-regulation of adenylyl cyclase type V/VI and increased GABA release in the substantia nigra reticulata , 2011, Neurobiology of Disease.
[26] V. Durkalski,et al. Relationships between large neutral amino acid levels in plasma, cerebrospinal fluid, brain microdialysate and brain tissue in the rat , 2010, Brain Research.
[27] U. Ungerstedt,et al. The rotational model and microdialysis: Significance for dopamine signalling, clinical studies, and beyond , 2010, Progress in Neurobiology.
[28] R. Moratalla,et al. Genetic Inactivation of Dopamine D1 but Not D2 Receptors Inhibits L-DOPA–Induced Dyskinesia and Histone Activation , 2009, Biological Psychiatry.
[29] D. Centonze,et al. d-Aspartate Prevents Corticostriatal Long-Term Depression and Attenuates Schizophrenia-Like Symptoms Induced by Amphetamine and MK-801 , 2008, The Journal of Neuroscience.
[30] M. L. de Ceballos,et al. Tyrosine hydroxylase cells appearing in the mouse striatum after dopamine denervation are likely to be projection neurones regulated by l‐DOPA , 2008, The European journal of neuroscience.
[31] H. Haas,et al. GABAA‐receptor modification in taurine transporter knockout mice causes striatal disinhibition , 2007, The Journal of physiology.
[32] R. Moratalla,et al. Metabolic interactions between glutamatergic and dopaminergic neurotransmitter systems are mediated through D1 dopamine receptors , 2007, Journal of neuroscience research.
[33] Gonzalo Flores,et al. Alterations in dendritic morphology of the prefrontal cortical and striatum neurons in the unilateral 6‐OHDA‐rat model of Parkinson's disease , 2007, Synapse.
[34] Anders Björklund,et al. Dopamine released from 5-HT terminals is the cause of L-DOPA-induced dyskinesia in parkinsonian rats. , 2007, Brain : a journal of neurology.
[35] A. Schousboe,et al. The glutamate/GABA‐glutamine cycle: aspects of transport, neurotransmitter homeostasis and ammonia transfer , 2006, Journal of neurochemistry.
[36] N. Pavón,et al. ERK Phosphorylation and FosB Expression Are Associated with L-DOPA-Induced Dyskinesia in Hemiparkinsonian Mice , 2006, Biological Psychiatry.
[37] J. Clements,et al. Comparison of Taurine- and Glycine-induced Conformational Changes in the M2-M3 Domain of the Glycine Receptor* , 2004, Journal of Biological Chemistry.
[38] S. Engelborghs,et al. Amino Acids and Biogenic Amines in Cerebrospinal Fluid of Patients with Parkinson's Disease , 2003, Neurochemical Research.
[39] L. Denoroy,et al. Glutamate and aspartate do not exhibit the same changes in their extracellular concentrations in the rat striatum after N‐methyl‐D‐aspartate local administration , 2003, Journal of neuroscience research.
[40] H. Haas,et al. Long‐lasting enhancement of corticostriatal neurotransmission by taurine , 2002, The European journal of neuroscience.
[41] H. Haas,et al. Expression and function of glycine receptors in striatal cholinergic interneurons from rat and mouse , 2001, Neuroscience.
[42] A. El Idrissi,et al. Growth Factors and Taurine Protect against Excitotoxicity by Stabilizing Calcium Homeostasis and Energy Metabolism , 1999, The Journal of Neuroscience.
[43] T. Hökfelt,et al. EVIDENCE FOR ASPARTATE-IMMUNOREACTIVE NEURONS IN THE NEOSTRIATUM OF THE RAT: MODULATION BY THE MESENCEPHALIC DOPAMINE PATHWAY VIA D1-SUBTYPE OF RECEPTOR , 1996, Neuroscience.
[44] W. Benzing,et al. Striatal glutamic acid decarboxylase immunoreactivity is increased after dopaminergic deafferentation: densitometric analysis , 1991, Neuroscience Letters.
[45] U. Ungerstedt,et al. Bilateral regulation of glutamate tissue and extracellular levels in caudate-putamen by midbrain dopamine neurons , 1990, Neuroscience Letters.
[46] T. Halonen,et al. Free amino acids in the brain of patients with Parkinson's disease , 1988, Neuroscience Letters.
[47] U. Ungerstedt,et al. Simultaneous measurement of dopamine release and rotational behaviour in 6-hydroxydopamine denervated rats using intracerebral dialysis , 1986, Brain Research.
[48] Y. Mizuno,et al. Changes in γ-aminobutyrate, glutamate, aspartate, glycine, and taurine contents in the striatum after unilateral nigrostriatal lesions in rats , 1986, Experimental Neurology.
[49] M. Mena,et al. Monoamine metabolites in human cerebrospinal fluid. HPLC/ED method , 1984, Acta neurologica Scandinavica.
[50] U. Ungerstedt,et al. Evidence that apomorphine and pergolide induce rotation in rats by different actions on D1 and D2 receptor sites. , 1984, European journal of pharmacology.
[51] F. Guimarães,et al. Counteraction by Nitric Oxide Synthase Inhibitor of Neurochemical Alterations of Dopaminergic System in 6-OHDA-Lesioned Rats Under l-DOPA Treatment , 2013, Neurotoxicity Research.
[52] Eduardo D. Martín,et al. D1 but not D5 dopamine receptors are critical for LTP, spatial learning, and LTP-Induced arc and zif268 expression in the hippocampus. , 2008, Cerebral cortex.