Neomycin blocks dihydropyridine-insensitive Ca2+ influx in bovine adrenal chromaffin cells.

[1]  C. Duarte,et al.  A Toxin Fraction (FTX) from the Funnel‐Web Spider Poison Inhibits Dihydropyridine‐Insensitive Ca2+ Channels Coupled to Catecholamine Release in Bovine Adrenal Chromaffin Cells , 1993, Journal of neurochemistry.

[2]  A. Fox,et al.  ω-Conotoxin GVIA blocks a Ca2+ current in bovine chromaffin cells that is not of the “classic” N type , 1992, Neuron.

[3]  A. Fox,et al.  Three types of bovine chromaffin cell Ca2+ channels: facilitation increases the opening probability of a 27 pS channel. , 1991, The Journal of physiology.

[4]  R. Burgoyne Control of exocytosis in adrenal chromaffin cells. , 1991, Biochimica et biophysica acta.

[5]  J. Bossu,et al.  Inactivation characteristics reveal two calcium currents in adult bovine chromaffin cells. , 1991, The Journal of physiology.

[6]  J. Bossu,et al.  Two types of calcium channels are expressed in adult bovine chromaffin cells. , 1991, The Journal of physiology.

[7]  N. Weiner,et al.  Ca2(+)-evoked [3H]dopamine release from synaptosomes is dependent on neuronal type Ca2+ channels and is not mediated by acetylcholine, glutamate or aspartate release. , 1990, The Journal of pharmacology and experimental therapeutics.

[8]  K. Takeda,et al.  The blocking effects of ω-conotoxin on Ca current in bovine chromaffin cells , 1990, Neuroscience Letters.

[9]  M. Titeler,et al.  Identification of ω‐Conotoxin Binding Sites on Adrenal Medullary Membranes: Possibility of Multiple Calcium Channels in Chromaffin Cells , 1990, Journal of neurochemistry.

[10]  G. Feuerstein,et al.  Voltage-sensitive calcium flux into bovine chromaffin cells occurs through dihydropyridine-sensitive and dihydropyridine- and ω-conotoxin-insensitive pathways , 1989, Neuroscience.

[11]  J. Reig,et al.  Separate Binding and Functional Sites for ω co‐Conotoxin and Nitrendipine Suggest Two Types of Calcium Channels in Bovine Chromaffin Cells , 1989, Journal of neurochemistry.

[12]  M. Boarder,et al.  Evidence for a dihydropyridine‐sensitive and conotoxin‐insensitive release of noradrenaline and uptake of calcium in adrenal chromaffin cells , 1989, British journal of pharmacology.

[13]  R. Burgoyne,et al.  A comparison of bradykinin, angiotensin II and muscarinic stimulation of cultured bovine adrenal chromaffin cells , 1989, Bioscience reports.

[14]  R Llinás,et al.  Blocking and isolation of a calcium channel from neurons in mammals and cephalopods utilizing a toxin fraction (FTX) from funnel-web spider poison. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[15]  L. Rosário,et al.  Role of intracellular pH in secretion from adrenal medulla chromaffin cells. , 1989, The Journal of biological chemistry.

[16]  R. Plevin,et al.  Stimulation of Formation of Inositol Phosphates in Primary Cultures of Bovine Adrenal Chromaffin Cells by Angiotensin II, Histamine, Bradykinin, and Carbachol , 1988, Journal of neurochemistry.

[17]  M. Gustin,et al.  Neomycin inhibits the calcium current of Paramecium. , 1988, Biochimica et biophysica acta.

[18]  W. Atchison,et al.  Effects of antibiotics on uptake of calcium into isolated nerve terminals. , 1988, The Journal of pharmacology and experimental therapeutics.

[19]  S. Yamamoto,et al.  Inositol trisphosphate accumulation by high K+ stimulation in cultured adrenal chromaffin cells , 1987, FEBS letters.

[20]  D. Eberhard,et al.  Cholinergic Stimulation of Inositol Phosphate Formation in Bovine Adrenal Chromaffin Cells: Distinct Nicotinic and Muscarinic Mechanisms , 1987, Journal of neurochemistry.

[21]  R. Tsien,et al.  Omega-conotoxin: direct and persistent blockade of specific types of calcium channels in neurons but not muscle. , 1987, Proceedings of the National Academy of Sciences of the United States of America.

[22]  M. Levine Ascorbic acid specifically enhances dopamine beta-monooxygenase activity in resting and stimulated chromaffin cells. , 1986, The Journal of biological chemistry.

[23]  M. Prentki,et al.  Neomycin: a specific drug to study the inositol‐phospholipid signalling system? , 1986, FEBS letters.

[24]  R. Tsien,et al.  A new generation of Ca2+ indicators with greatly improved fluorescence properties. , 1985, The Journal of biological chemistry.

[25]  Michael R. Whitaker,et al.  Calcium‐dependent polyphosphoinositide hydrolysis is associated with exocytosis in vitro , 1985, FEBS letters.

[26]  F. Sala,et al.  Dihydropyridine BAY-K-8644 activates chromaffin cell calcium channels , 1984, Nature.

[27]  P. Sánchez-García,et al.  Pharmacological dissection of receptor-associated and voltage-sensitive ionic channels involved in catecholamine release , 1983, Neuroscience.

[28]  J. Fiekers Effects of the aminoglycoside antibiotics, streptomycin and neomycin, on neuromuscular transmission. II. Postsynaptic considerations. , 1983, The Journal of pharmacology and experimental therapeutics.

[29]  S. Feldman,et al.  Inhibition of gentamicin uptake in rat renal cortex in vivo by aminoglycosides and organic polycations. , 1982, The Journal of pharmacology and experimental therapeutics.

[30]  D. Sanders,et al.  The neuromuscular blocking effects of therapeutic concentrations of various antibiotics on normal rat skeletal muscle: a quantitative comparison. , 1981, The Journal of pharmacology and experimental therapeutics.

[31]  J. Schacht,et al.  Purification of polyphosphoinositides by chromatography on immobilized neomycin. , 1978, Journal of lipid research.

[32]  J. Wright,et al.  The effects of neomycin upon transmitter release and action. , 1977, The Journal of pharmacology and experimental therapeutics.

[33]  A. Fox,et al.  Two types of Ca2+ currents are found in bovine chromaffin cells: facilitation is due to the recruitment of one type. , 1991, The Journal of physiology.

[34]  C. Pittinger,et al.  Antibiotic blockade of neuromuscular function. , 1972, Annual review of pharmacology.