Collagen synthesis is suppressed in dermal fibroblasts by the human antimicrobial peptide LL-37.
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A. Di Nardo | D. Cho | R. Gallo | B. Cho | S. Song | K. Yamasaki | H. Park | S. Bang | Jun Young Lee | Heejong Kim
[1] K. Nagata,et al. Alpha-defensin enhances expression of HSP47 and collagen-1 in human lung fibroblasts. , 2007, Life sciences.
[2] David Abraham,et al. Systemic sclerosis: a prototypic multisystem fibrotic disorder. , 2007, The Journal of clinical investigation.
[3] C. Hogaboam,et al. Infectious disease, the innate immune response, and fibrosis. , 2007, The Journal of clinical investigation.
[4] T. Wynn,et al. Common and unique mechanisms regulate fibrosis in various fibroproliferative diseases. , 2007, The Journal of clinical investigation.
[5] Y. Tada,et al. Antifibrotic effects of hepatocyte growth factor on scleroderma fibroblasts and analysis of its mechanism , 2006, Modern rheumatology.
[6] M. Raghunath,et al. Defensins HNP1 and HBD2 stimulation of wound-associated responses in human conjunctival fibroblasts. , 2006, Investigative ophthalmology & visual science.
[7] H. Kawachi,et al. Transcription factor Ets-1 is essential for mesangial matrix remodeling. , 2006, Kidney international.
[8] T. Luger,et al. Melanocortins in fibroblast biology – current update and future perspective for dermatology , 2004, Experimental dermatology.
[9] H. Hammes,et al. Regulation of neovascularization by human neutrophil peptides (α‐defensins): a link between inflammation and angiogenesis , 2004, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[10] M. Raghunath,et al. Collagen Metabolism Is a Novel Target of the Neuropeptide α-Melanocyte-stimulating Hormone* , 2004, Journal of Biological Chemistry.
[11] M. Raghunath,et al. Collagen metabolism is a novel target of the neuropeptide alpha-melanocyte-stimulating hormone. , 2004, The Journal of biological chemistry.
[12] P. Elsbach. What is the real role of antimicrobial polypeptides that can mediate several other inflammatory responses? , 2003, The Journal of clinical investigation.
[13] S. Zahler,et al. An angiogenic role for the human peptide antibiotic LL-37/hCAP-18. , 2003, The Journal of clinical investigation.
[14] A. Vitiello,et al. Cutting Edge: Mast Cell Antimicrobial Activity Is Mediated by Expression of Cathelicidin Antimicrobial Peptide 1 , 2003, The Journal of Immunology.
[15] Tomas Ganz,et al. Endogenous antimicrobial peptides and skin infections in atopic dermatitis. , 2002, The New England journal of medicine.
[16] V. Sementchenko,et al. Ets1 Is an Effector of the Transforming Growth Factor β (TGF-β) Signaling Pathway and an Antagonist of the Profibrotic Effects of TGF-β* , 2002, The Journal of Biological Chemistry.
[17] H. Akiyama,et al. Effects of human neutrophil peptide-1 on the expression of interstitial collagenase and type I collagen in human dermal fibroblasts , 2002, Archives of Dermatological Research.
[18] Takaaki Ohtake,et al. Innate antimicrobial peptide protects the skin from invasive bacterial infection , 2001, Nature.
[19] V. Nizet,et al. Cutaneous injury induces the release of cathelicidin anti-microbial peptides active against group A Streptococcus. , 2001, The Journal of investigative dermatology.
[20] K A Arndt,et al. Keloids and hypertrophic scars: review and treatment strategies. , 1999, Seminars in cutaneous medicine and surgery.
[21] Domenico Romeo,et al. Cathelicidins: a novel protein family with a common proregion and a variable C‐terminal antimicrobial domain , 1995, FEBS letters.
[22] M. Klagsbrun,et al. Syndecans, cell surface heparan sulfate proteoglycans, are induced by a proline-rich antimicrobial peptide from wounds. , 1994, Proceedings of the National Academy of Sciences of the United States of America.