Improved myocardial oxygen utilization following propranolol infusion in adolescents with postburn hypermetabolism.

[1]  R. Barrow,et al.  Effect of Propranolol Administration on Hemodynamic and Metabolic Responses of Burned Pediatric Patients , 1988, Annals of surgery.

[2]  R. Gillum Heart rate of black and white youths aged 12-17 years: associations with blood pressure and other cardiovascular risk factors. , 1988, Journal of the National Medical Association.

[3]  J. F. Southern,et al.  Case 15-1988 , 1988 .

[4]  S. Gelman,et al.  Lack of immediate effects of wound excision on the hyperdynamic circulation of burned patients. , 1988, The Journal of burn care & rehabilitation.

[5]  R. Demling,et al.  The effect of complete burn wound excision and closure on postburn oxygen consumption. , 1987, Surgery.

[6]  R. S. Eliot,et al.  Experimental catecholamine-induced myocardial necrosis. I. Morphology, quantification and regional distribution of acute contraction band lesions. , 1985, Journal of molecular and cellular cardiology.

[7]  M. Caron,et al.  Beta-adrenergic receptors: biochemical mechanisms of physiological regulation. , 1984, Physiological reviews.

[8]  G. Berenson,et al.  Resting heart rate and pressure-rate product of children in a total biracial community: the Bogalusa Heart Study. , 1982, American journal of epidemiology.

[9]  D C Harrison,et al.  Decreased catecholamine sensitivity and beta-adrenergic-receptor density in failing human hearts. , 1982, The New England journal of medicine.

[10]  G. A. Rooke,et al.  Work as a Correlate of Canine Left Ventricular Oxygen Consumption, and the Problem of Catecholamine Oxygen Wasting , 1982, Circulation research.

[11]  P. Cryer,et al.  Epinephrine plasma metabolic clearance rates and physiologic thresholds for metabolic and hemodynamic actions in man. , 1980, The Journal of clinical investigation.

[12]  G. Hellige,et al.  Validity of myocardial oxygen consumption parameters , 1979, Clinical cardiology.

[13]  YANG WANG,et al.  The Rate-Pressure Product as an Index of Myocardial Oxygen Consumption during Exercise in Patients with Angina Pectoris , 1978, Circulation.

[14]  A. Mason,et al.  Catecholamines: Znediator of the Hypermetabolic Response to Thermal Injury , 1974, Annals of surgery.

[15]  D. Hunt,et al.  Myocardial lesions following experimental intracranial hemorrhage: prevention with propranolol. , 1972, American heart journal.

[16]  D. Reichenbach,et al.  Catecholamines and cardiomyopathy: The pathogenesis and potential importance of myofibrillar degeneration , 1970 .

[17]  W. Shoemaker,et al.  Cardiovascular function in hemorrhage, trauma and sepsis: determinants of cardiac output and cardiac work. , 1969, Annals of surgery.

[18]  J. Seaton,et al.  Relationship of Increased Oxygen Consumption to Catecholamine Excretion in Thermal Bums , 1967, Annals of surgery.

[19]  J. Moncrief Effect of various fluid regimens and pharmacologic agents on the circulatory hemodynamics of the immediate postburn period. , 1966, Annals of surgery.

[20]  H. Burchell,et al.  Focal myocarditis associated with pheochromocytoma. , 1966, The New England journal of medicine.

[21]  W. Raab,et al.  Sympathogenic origin and antiadrenergic prevention of stress-induced myocardial lesions. , 1961, The American journal of cardiology.

[22]  W. Raab Key position of catecholamines in functional and degenerative cardiovascular pathology. , 1960, The American journal of cardiology.

[23]  R. Scully,et al.  Case records of the Massachusetts General Hospital. , 1990 .