Induction of Oxidative DNA Damage in the Peri‐Infarct Region After Permanent Focal Cerebral Ischemia
暂无分享,去创建一个
R. Simon | D. Henshall | Jun Chen | Dexi Chen | J. Lan | J. Chen | C. O'Horo | T. Nagayama | D. Chen | Tetsuya Nagayama | Cristine O'Horo | Cristine O'Horo
[1] R. Simon,et al. Activation of Poly(ADP‐Ribose) Polymerase in the Rat Hippocampus May Contribute to Cellular Recovery Following Sublethal Transient Global Ischemia , 2000, Journal of neurochemistry.
[2] R. Simon,et al. Formation of the Base Modification 8‐Hydroxyl‐2′ ‐ Deoxyguanosine and DNA Fragmentation Following Seizures Induced by Systemic Kainic Acid in the Rat , 2000, Journal of neurochemistry.
[3] E. Holmes,et al. Oxidative Damage to the c‐fos Gene and Reduction of Its Transcription After Focal Cerebral Ischemia , 1999, Journal of neurochemistry.
[4] S. Snyder,et al. Post-treatment with an inhibitor of poly(ADP-ribose) polymerase attenuates cerebral damage in focal ischemia , 1999, Brain Research.
[5] M. Fujimura,et al. Early Decrease of Apurinic/Apyrimidinic Endonuclease Expression after Transient Focal Cerebral Ischemia in Mice , 1999, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[6] U. Tuor,et al. Cerebral Ischemia Produces Laddered DNA Fragments Distinct from Cardiac Ischemia and Archetypal Apoptosis , 1999, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[7] S. Snyder,et al. Poly (ADP-ribose) polymerase, nitric oxide and cell death. , 1999, Trends in pharmacological sciences.
[8] M. Fujimura,et al. Reduction of apurinic/apyrimidinic endonuclease expression after transient global cerebral ischemia in rats: implication of the failure of DNA repair in neuronal apoptosis. , 1999, Stroke.
[9] K. Nozaki,et al. Enhanced Poly(ADP-ribosyl)ation after Focal Ischemia in Rat Brain , 1998, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[10] R. Simon,et al. Induction of Caspase-3-Like Protease May Mediate Delayed Neuronal Death in the Hippocampus after Transient Cerebral Ischemia , 1998, The Journal of Neuroscience.
[11] H. Cheng,et al. MDL 101,002, a free radical spin trap, is efficacious in permanent and transient focal ischemia models. , 1998, Life sciences.
[12] Ulrich Dirnagl,et al. Increased Formation of Reactive Oxygen Species after Permanent and Reversible Middle Cerebral Artery Occlusion in the Rat , 1998, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[13] Y. Shinohara,et al. Peroxynitrite Formation in Focal Cerebral Ischemia—Reperfusion in Rats Occurs Predominantly in the Peri-Infarct Region , 1998, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[14] P. Upton,et al. Highly sensitive apurinic/apyrimidinic site assay can detect spontaneous and chemically induced depurination under physiological conditions. , 1998, Cancer research.
[15] S. Nagata,et al. A caspase-activated DNase that degrades DNA during apoptosis, and its inhibitor ICAD , 1998, Nature.
[16] M. Moskowitz,et al. Ischemic Brain Injury is Mediated by the Activation of Poly(ADP-Ribose)Polymerase , 1997, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[17] S. Snyder,et al. Poly(ADP-ribose) polymerase gene disruption renders mice resistant to cerebral ischemia , 1997, Nature Medicine.
[18] B. Demple,et al. Interaction of human apurinic endonuclease and DNA polymerase beta in the base excision repair pathway. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[19] R. Simon,et al. Early Detection of DNA Strand Breaks in the Brain After Transient Focal Ischemia: Implications for the Role of DNA Damage in Apoptosis and Neuronal Cell Death , 1997, Journal of neurochemistry.
[20] Xiaodong Wang,et al. DFF, a Heterodimeric Protein That Functions Downstream of Caspase-3 to Trigger DNA Fragmentation during Apoptosis , 1997, Cell.
[21] R. Floyd,et al. Damage, Repair, and Mutagenesis in Nuclear Genes after Mouse Forebrain Ischemia–Reperfusion , 1996, The Journal of Neuroscience.
[22] P. Kochanek,et al. Polymorphonuclear leukocytes and microcirculatory perfusion in acute stroke in the SHR. , 1996, The Keio journal of medicine.
[23] M. Chopp,et al. DNA damage and repair in central nervous system injury: National Institute of Neurological Disorders and Stroke Workshop Summary. , 1996, Stroke.
[24] T. Yoshimine,et al. Generation of nitric oxide and superoxide during reperfusion after focal cerebral ischemia in rats. , 1996, The American journal of physiology.
[25] Y. Ben-Ari,et al. Apoptosis and Necrosis after Reversible Focal Ischemia: An in Situ DNA Fragmentation Analysis , 1996, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[26] K. Kogure,et al. Role of Neutrophils in Radical Production during Ischemia and Reperfusion of the Rat Brain: Effect of Neutrophil Depletion on Extracellular Ascorbyl Radical Formation , 1995, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[27] H. Bernstein,et al. Apoptosis overview emphasizing the role of oxidative stress, DNA damage and signal-transduction pathways. , 1995, Leukemia & lymphoma.
[28] P. Weinstein,et al. Attenuation of postischemic brain hypoperfusion and reperfusion injury by the cyclooxygenase-lipoxygenase inhibitor BW755C. , 1995, Journal of neurosurgery.
[29] M. Chopp,et al. Temporal Profile of in situ DNA Fragmentation after Transient Middle Cerebral Artery Occlusion in the Rat , 1995, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[30] Y. Ben-Ari,et al. Early Endonuclease Activation following Reversible Focal Ischemia in the Rat Brain , 1995, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[31] John Calvin Reed,et al. Tumor suppressor p53 is a direct transcriptional activator of the human bax gene , 1995, Cell.
[32] L. Loeb,et al. Reverse chemical mutagenesis: identification of the mutagenic lesions resulting from reactive oxygen species-mediated damage to DNA. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[33] E. Hall,et al. Dose-response analysis of the effect of 21-aminosteroid tirilazad mesylate (U-74006F) upon neurological outcome and ischemic brain damage in permanent focal cerebral ischemia , 1994, Brain Research.
[34] M. Kastan,et al. DNA strand breaks: the DNA template alterations that trigger p53-dependent DNA damage response pathways , 1994, Molecular and cellular biology.
[35] A. Buchan,et al. DNA damage consistent with apoptosis in transient focal ischaemic neocortex. , 1994, Neuroreport.
[36] M. Linnik,et al. Evidence Supporting a Role for Programmed Cell Death in Focal Cerebral Ischemia in Rats , 1993, Stroke.
[37] P. Borm,et al. Cell and tissue responses to oxidative damage. , 1993, Laboratory investigation; a journal of technical methods and pathology.
[38] K. Makino,et al. Synthesis and damage specificity of a novel probe for the detection of abasic sites in DNA. , 1993, Biochemistry.
[39] A. Grollman,et al. Mutagenesis by 8-oxoguanine: an enemy within. , 1993, Trends in genetics : TIG.
[40] S. Ben‐Sasson,et al. Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation , 1992, The Journal of cell biology.
[41] P. Kochanek,et al. Polymorphonuclear Leukocytes and Monocytes/Macrophages in the Pathogenesis of Cerebral Ischemia and Stroke , 1992, Stroke.
[42] S. Wallace,et al. A novel, sensitive, and specific assay for abasic sites, the most commonly produced DNA lesion. , 1992, Biochemistry.
[43] S. Tannenbaum,et al. DNA damage and mutation in human cells exposed to nitric oxide in vitro. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[44] A. Grollman,et al. Insertion of specific bases during DNA synthesis past the oxidation-damaged base 8-oxodG , 1991, Nature.
[45] E. Gajewski,et al. Modification of DNA bases in mammalian chromatin by radiation-generated free radicals. , 1990, Biochemistry.
[46] R. Floyd,et al. Role of oxygen free radicals in carcinogenesis and brain ischemia , 1990, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[47] E. Moran,et al. The role of specific DNA base damages in the X-ray-induced inactivation of bacteriophage PM2. , 1985, Mutation research.
[48] R. Schaaper,et al. Depurination causes mutations in SOS-induced cells. , 1981, Proceedings of the National Academy of Sciences of the United States of America.
[49] H. Ide,et al. Repair kinetics of abasic sites in mammalian cells selectively monitored by the aldehyde reactive probe (ARP). , 1998, Nucleosides & nucleotides.
[50] N. Huang,et al. Hydroxyl radical formation in hyperglycemic rats during middle cerebral artery occlusion/reperfusion. , 1997, Free radical biology & medicine.
[51] Y. Fukuuchi,et al. Leukocytes, macrophages and secondary brain damage following cerebral ischemia. , 1996, Acta neurochirurgica. Supplement.
[52] Ravi S. Sandhu,et al. Workshop summary , 1996, RBAC '95.
[53] C. Iadecola,et al. Inhibition of inducible nitric oxide synthase ameliorates cerebral ischemic damage. , 1995, The American journal of physiology.
[54] B Demple,et al. Repair of oxidative damage to DNA: enzymology and biology. , 1994, Annual review of biochemistry.
[55] J. Garcìa,et al. Influx of leukocytes and platelets in an evolving brain infarct (Wistar rat). , 1994, The American journal of pathology.
[56] M. Bednar,et al. The role of neutrophils and platelets in a rabbit model of thromboembolic stroke. , 1991, Stroke.
[57] M. Dizdaroglu. Chemical determination of free radical-induced damage to DNA. , 1991, Free radical biology & medicine.