Aquaporin1–3 expression in normal and hydronephrotic kidneys in the human fetus

[1]  Julie B. Lindower.  Water balance in the fetus and neonate. , 2017, Seminars in fetal & neonatal medicine.

[2]  R. Nørregaard,et al.  Influence of sex on aquaporin1–4 and vasopressin V2 receptor expression in the pig kidney during development , 2016, Pediatric Research.

[3]  J. Djurhuus,et al.  Ontogeny of the mammalian kidney: expression of aquaporins 1, 2, 3, and 4 , 2014, World Journal of Pediatrics.

[4]  A. Chandra,et al.  Decrease of renal aquaporins 1–4 is associated with renal function impairment in pediatric congenital hydronephrosis , 2012, World Journal of Pediatrics.

[5]  Richard S. Lee,et al.  The Society for Fetal Urology consensus statement on the evaluation and management of antenatal hydronephrosis. , 2010, Journal of pediatric urology.

[6]  J. Frøkiaer,et al.  Expression of renal aquaporins is down-regulated in children with congenital hydronephrosis , 2009, Scandinavian journal of urology and nephrology.

[7]  H. Debaix,et al.  Expression patterns of the aquaporin gene family during renal development: influence of genetic variability , 2009, Pflügers Archiv - European Journal of Physiology.

[8]  J. Frøkiaer,et al.  Aquaporins in the kidney. , 2009, Handbook of experimental pharmacology.

[9]  F. Matsui,et al.  Urological emergency in neonates with congenital hydronephrosis , 2007, International journal of urology : official journal of the Japanese Urological Association.

[10]  J. Frøkiaer,et al.  COX-2 activity transiently contributes to increased water and NaCl excretion in the polyuric phase after release of ureteral obstruction. , 2007, American journal of physiology. Renal physiology.

[11]  R. Chevalier.  Chronic partial ureteral obstruction and the developing kidney , 2007, Pediatric Radiology.

[12]  W. Manucha.  Biochemical-molecular markers in unilateral ureteral obstruction. , 2007, Biocell : official journal of the Sociedades Latinoamericanas de Microscopia Electronica ... et. al.

[13]  P. Deen,et al.  Cell biological aspects of the vasopressin type-2 receptor and aquaporin 2 water channel in nephrogenic diabetes insipidus. , 2006, American journal of physiology. Renal physiology.

[14]  Feng Chen,et al.  Congenital progressive hydronephrosis (cph) is caused by an S256L mutation in aquaporin-2 that affects its phosphorylation and apical membrane accumulation. , 2006, Proceedings of the National Academy of Sciences of the United States of America.

[15]  A. Aperia,et al.  Water Channels (Aquaporins) and Their Role for Postnatal Adaptation , 2005, Pediatric Research.

[16]  J. Frøkiaer,et al.  Neonatal ureteral obstruction alters expression of renal sodium transporters and aquaporin water channels. , 2004, Kidney international.

[17]  D. Milewicz,et al.  Characterization of an aquaporin-2 water channel gene mutation causing partial nephrogenic diabetes insipidus in a Mexican family: evidence of increased frequency of the mutation in the town of origin. , 2004, Journal of the American Society of Nephrology : JASN.

[18]  S. Hooper,et al.  Aquaporin gene expression and regulation in the ovine fetal lung , 2003, The Journal of physiology.

[19]  G. Ardissino,et al.  Epidemiology of chronic renal failure in children: data from the ItalKid project. , 2003, Pediatrics.

[20]  J. Frøkiaer,et al.  Renal aquaporins and sodium transporters with special focus on urinary tract obstruction. , 2003, APMIS. Supplementum.

[21]  J. Frøkiaer,et al.  Aquaporins in the kidney: from molecules to medicine. , 2002, Physiological reviews.

[22]  Thomas D. Schmittgen,et al.  Analysis of relative gene expression data using real-time quantitative PCR and the 2(-Delta Delta C(T)) Method. , 2001, Methods.

[23]  C J Epstein,et al.  Nephrogenic diabetes insipidus in mice lacking aquaporin-3 water channels. , 2000, Proceedings of the National Academy of Sciences of the United States of America.

[24]  Robert L. Chevalier,et al.  Molecular and cellular pathophysiology of obstructive nephropathy , 1999, Pediatric Nephrology.

[25]  A. Verkman,et al.  Reduced water permeability and altered ultrastructure in thin descending limb of Henle in aquaporin-1 null mice. , 1999, The Journal of clinical investigation.

[26]  E. Wintour,et al.  Ovine aquaporin-2: cDNA cloning, ontogeny and control of renal gene expression , 1998, Pediatric Nephrology.

[27]  A. Verkman,et al.  Defective proximal tubular fluid reabsorption in transgenic aquaporin-1 null mice. , 1998, Proceedings of the National Academy of Sciences of the United States of America.

[28]  M. K. Ruddy,et al.  The Perinatal Expression of Aquaporin-2 and Aquaporin-3 in Developing Kidney , 1998, Pediatric Research.

[29]  C. Epstein,et al.  Severely Impaired Urinary Concentrating Ability in Transgenic Mice Lacking Aquaporin-1 Water Channels* , 1998, The Journal of Biological Chemistry.

[30]  E. Wintour,et al.  Ovine AQP1: cDNA cloning, ontogeny, and control of renal gene expression , 1998, Pediatric Nephrology.

[31]  F. Marumo,et al.  Expression of AQP family in rat kidneys during development and maturation. , 1997, The American journal of physiology.

[32]  S. Nielsen,et al.  Aquaporin-3 water channel localization and regulation in rat kidney. , 1995, The American journal of physiology.

[33]  C. Turck,et al.  Immunolocalization of the mercurial-insensitive water channel and glycerol intrinsic protein in epithelial cell plasma membranes. , 1995, Proceedings of the National Academy of Sciences of the United States of America.

[34]  P. Agre,et al.  Concurrent expression of erythroid and renal aquaporin CHIP and appearance of water channel activity in perinatal rats. , 1993, The Journal of clinical investigation.