Amyloid-beta and tau pathology following repetitive mild traumatic brain injury.

[1]  Ann C. McKee,et al.  Antibody against early driver of neurodegeneration cis P-tau blocks brain injury and tauopathy , 2015, Nature.

[2]  M. Febo,et al.  Temporal MRI characterization, neurobiochemical and neurobehavioral changes in a mouse repetitive concussive head injury model , 2015, Scientific Reports.

[3]  Weiming Xia,et al.  Beta-amyloid deposition in chronic traumatic encephalopathy , 2015, Acta Neuropathologica.

[4]  L. Mucke,et al.  Tau Reduction Diminishes Spatial Learning and Memory Deficits after Mild Repetitive Traumatic Brain Injury in Mice , 2014, PloS one.

[5]  Corey T Walker,et al.  The pathophysiology underlying repetitive mild traumatic brain injury in a novel mouse model of chronic traumatic encephalopathy , 2014, Surgical neurology international.

[6]  K. Saatman,et al.  Regional Neurodegeneration and Gliosis Are Amplified by Mild Traumatic Brain Injury Repeated at 24-Hour Intervals , 2014, Journal of neuropathology and experimental neurology.

[7]  Corey T Walker,et al.  The spectrum of neurobehavioral sequelae after repetitive mild traumatic brain injury: a novel mouse model of chronic traumatic encephalopathy. , 2014, Journal of neurotrauma.

[8]  David C Viano,et al.  Animal models of sports‐related head injury: bridging the gap between pre‐clinical research and clinical reality , 2014, Journal of neurochemistry.

[9]  G. Beaupré,et al.  Long-Term Cognitive Impairments and Pathological Alterations in a Mouse Model of Repetitive Mild Traumatic Brain Injury , 2014, Front. Neurol..

[10]  T. Talavage,et al.  Functionally-detected cognitive impairment in high school football players without clinically-diagnosed concussion. , 2014, Journal of neurotrauma.

[11]  W. Stewart,et al.  Chronic neuropathological and neurobehavioral changes in a repetitive mild traumatic brain injury model , 2014, Annals of neurology.

[12]  N. Chen,et al.  Examining the relationship between head trauma and neurodegenerative disease: A review of epidemiology, pathology and neuroimaging techniques. , 2014, Journal of Alzheimer's disease & Parkinsonism.

[13]  Mathias Jucker,et al.  Self-propagation of pathogenic protein aggregates in neurodegenerative diseases , 2013, Nature.

[14]  C. Soto,et al.  Cross-Seeding of Misfolded Proteins: Implications for Etiology and Pathogenesis of Protein Misfolding Diseases , 2013, PLoS pathogens.

[15]  E. Lehman.  Epidemiology of neurodegeneration in American-style professional football players , 2013, Alzheimer's Research & Therapy.

[16]  Lee W. Tien,et al.  Clinical correlates in an experimental model of repetitive mild brain injury , 2013, Annals of neurology.

[17]  N. Greig,et al.  Changes in mouse cognition and hippocampal gene expression observed in a mild physical- and blast-traumatic brain injury , 2013, Neurobiology of Disease.

[18]  M. Nedergaard,et al.  ‘Hit & Run’ Model of Closed-Skull Traumatic Brain Injury (TBI) Reveals Complex Patterns of Post-Traumatic AQP4 Dysregulation , 2013, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[19]  Joshua J. Breunig,et al.  Brain injury, neuroinflammation and Alzheimer's disease , 2013, Front. Aging Neurosci..

[20]  Véronique D. Bohbot,et al.  Spatial navigational strategies correlate with gray matter in the hippocampus of healthy older adults tested in a virtual maze , 2013, Front. Ag. Neurosci..

[21]  M. Mullan,et al.  Repetitive mild traumatic brain injury augments tau pathology and glial activation in aged hTau mice. , 2013, Journal of neuropathology and experimental neurology.

[22]  Michael Chopp,et al.  Animal models of traumatic brain injury , 2013, Nature Reviews Neuroscience.

[23]  W. Stewart,et al.  Repetitive mild traumatic brain injury in a mouse model produces learning and memory deficits accompanied by histological changes. , 2012, Journal of neurotrauma.

[24]  S. Prusiner,et al.  A Unifying Role for Prions in Neurodegenerative Diseases , 2012, Science.

[25]  C. Soto.  Transmissible Proteins: Expanding the Prion Heresy , 2012, Cell.

[26]  Robin O Cleveland,et al.  Chronic Traumatic Encephalopathy in Blast-Exposed Military Veterans and a Blast Neurotrauma Mouse Model , 2012, Science Translational Medicine.

[27]  T. M. Sivanandam,et al.  Traumatic brain injury: A risk factor for Alzheimer's disease , 2012, Neuroscience & Biobehavioral Reviews.

[28]  J. Gilger,et al.  Biomechanical correlates of symptomatic and asymptomatic neurophysiological impairment in high school football. , 2012, Journal of biomechanics.

[29]  B. Greenwald,et al.  Chronic Traumatic Encephalopathy: A Review , 2012, Rehabilitation research and practice.

[30]  Johan Davidsson,et al.  Experimental Animal Models for Studies on the Mechanisms of Blast-Induced Neurotrauma , 2012, Front. Neur..

[31]  D. Cain,et al.  Sub-concussive brain injury in the Long-Evans rat induces acute neuroinflammation in the absence of behavioral impairments , 2012, Behavioural Brain Research.

[32]  Tong Zhu,et al.  Subject-specific changes in brain white matter on diffusion tensor imaging after sports-related concussion. , 2012, Magnetic resonance imaging.

[33]  D. Cain,et al.  Repeated mild lateral fluid percussion brain injury in the rat causes cumulative long-term behavioral impairments, neuroinflammation, and cortical loss in an animal model of repeated concussion. , 2012, Journal of neurotrauma.

[34]  O. Barbarash,et al.  The Influence of Low and Moderate Carotid Stenosis on Neurophysiologic Status of Patients Undergoing on-pump Coronary Artery Bypass Grafting , 2012, Front. Neur..

[35]  M. Kane,et al.  A mouse model of human repetitive mild traumatic brain injury , 2012, Journal of Neuroscience Methods.

[36]  M. Jucker,et al.  Pathogenic protein seeding in alzheimer disease and other neurodegenerative disorders , 2011, Annals of neurology.

[37]  A. McKee,et al.  Long‐term Consequences of Repetitive Brain Trauma: Chronic Traumatic Encephalopathy , 2011, PM & R : the journal of injury, function, and rehabilitation.

[38]  D. Brody,et al.  Distinct Temporal and Anatomical Distributions of Amyloid-β and Tau Abnormalities following Controlled Cortical Impact in Transgenic Mice , 2011, PloS one.

[39]  Claudio Soto,et al.  Misfolded protein aggregates: mechanisms, structures and potential for disease transmission. , 2011, Seminars in cell & developmental biology.

[40]  D. Holtzman,et al.  Controlled Cortical Impact Traumatic Brain Injury in 3xTg-AD Mice Causes Acute Intra-Axonal Amyloid-β Accumulation and Independently Accelerates the Development of Tau Abnormalities , 2011, The Journal of Neuroscience.

[41]  B. Masel,et al.  Traumatic brain injury: a disease process, not an event. , 2010, Journal of neurotrauma.

[42]  Douglas H. Smith,et al.  Traumatic brain injury and amyloid-β pathology: a link to Alzheimer's disease? , 2010, Nature Reviews Neuroscience.

[43]  J. Simard,et al.  Novel model of frontal impact closed head injury in the rat. , 2009, Journal of neurotrauma.

[44]  A. McKee,et al.  Chronic Traumatic Encephalopathy in Athletes: Progressive Tauopathy After Repetitive Head Injury , 2009, Journal of neuropathology and experimental neurology.

[45]  A. Hamberger,et al.  CONCUSSION IN PROFESSIONAL FOOTBALL: MORPHOLOGY OF BRAIN INJURIES IN THE NFL CONCUSSION MODEL—PART 16 , 2009, Neurosurgery.

[46]  A. Hamberger,et al.  CONCUSSION IN PROFESSIONAL FOOTBALL: ANIMAL MODEL OF BRAIN INJURY—PART 15 , 2009, Neurosurgery.

[47]  J. Trojanowski,et al.  A Lack of Amyloid β Plaques Despite Persistent Accumulation of Amyloid β in Axons of Long‐Term Survivors of Traumatic Brain Injury , 2009, Brain pathology.

[48]  Claudio Soto,et al.  Amyloids, prions and the inherent infectious nature of misfolded protein aggregates. , 2006, Trends in biochemical sciences.

[49]  John Q Trojanowski,et al.  Enhanced neurofibrillary tangle formation, cerebral atrophy, and cognitive deficits induced by repetitive mild brain injury in a transgenic tauopathy mouse model. , 2005, Journal of neurotrauma.

[50]  J. Trojanowski,et al.  Vitamin E reduces amyloidosis and improves cognitive function in Tg2576 mice following repetitive concussive brain injury , 2004, Journal of neurochemistry.

[51]  D. Viano,et al.  Concussion in Professional Football: Epidemiological Features of Game Injuries and Review of the Literature—Part 3 , 2004, Neurosurgery.

[52]  D. Okonkwo,et al.  Caspase-3-mediated cleavage of amyloid precursor protein and formation of amyloid Beta peptide in traumatic axonal injury. , 2002, Journal of neurotrauma.

[53]  J. Trojanowski,et al.  Repetitive Mild Brain Trauma Accelerates Aβ Deposition, Lipid Peroxidation, and Cognitive Impairment in a Transgenic Mouse Model of Alzheimer Amyloidosis , 2002, The Journal of Neuroscience.

[54]  K.,et al.  Mild head injury increasing the brain's vulnerability to a second concussive impact. , 2001, Journal of neurosurgery.

[55]  L. Murray,et al.  Beta amyloid protein deposition in the brain after severe head injury: implications for the pathogenesis of Alzheimer's disease. , 1994, Journal of neurology, neurosurgery, and psychiatry.

[56]  P. Lansbury,et al.  Seeding “one-dimensional crystallization” of amyloid: A pathogenic mechanism in Alzheimer's disease and scrapie? , 1993, Cell.

[57]  D. Graham,et al.  βA4 amyloid protein deposition in brain after head trauma , 1991, The Lancet.

[58]  E. Peskind,et al.  Blast exposure causes early and persistent aberrant phospho- and cleaved-tau expression in a murine model of mild blast-induced traumatic brain injury. , 2013, Journal of Alzheimer's disease : JAD.

[59]  H. Braak,et al.  Neuropathological stageing of Alzheimer-related changes , 2004, Acta Neuropathologica.

[60]  Claudio Soto,et al.  Unfolding the role of protein misfolding in neurodegenerative diseases , 2003, Nature Reviews Neuroscience.

[61]  D. Graham,et al.  Altered β-APP Metabolism After Head Injury and its Relationship to the Aetiology of Alzheimer’s Disease , 1996 .